Her main research concerns Alzheimer's disease, Neuroscience, Pathology, Atrophy and Central nervous system disease. Her Alzheimer's disease research includes elements of Neuroimaging and Dementia. Pathology connects with themes related to Positron emission tomography in her study.
Her studies deal with areas such as Braak staging and Human brain as well as Positron emission tomography. The subject of her Atrophy research is within the realm of Internal medicine. In her study, Early-onset Alzheimer's disease, Cardiology, Cerebral atrophy, Age of onset and Fluorodeoxyglucose is strongly linked to Precuneus, which falls under the umbrella field of Central nervous system disease.
Suzanne L. Baker focuses on Neuroscience, Pathology, Disease, Amyloid and Internal medicine. Many of her studies involve connections with topics such as Tau pathology and Neuroscience. Her works in Alzheimer's disease, Atrophy, Posterior cortical atrophy, Frontotemporal lobar degeneration and Frontotemporal dementia are all subjects of inquiry into Pathology.
Her Alzheimer's disease research incorporates themes from Fluorodeoxyglucose, Positron emission tomography, Standardized uptake value and Central nervous system disease. Her study in Internal medicine is interdisciplinary in nature, drawing from both Endocrinology, Oncology and Cardiology. Her Cardiology research is multidisciplinary, incorporating elements of Early-onset Alzheimer's disease, Cerebral atrophy and Age of onset.
Suzanne L. Baker mostly deals with Neuroscience, Neuroimaging, Disease, Pathology and Amyloid. Her Neuroscience research includes themes of Tau pathology, Normal aging and Cognitive decline. Her Disease research is multidisciplinary, incorporating perspectives in Clinical psychology and Cognition, Episodic memory.
In general Pathology, her work in Progressive supranuclear palsy is often linked to Neurofibrillary tangle linking many areas of study. Her Amyloid study also includes fields such as
Cognitive impairment, Standardized uptake value, Dementia, Pathology and Amyloid are her primary areas of study. She interconnects Progressive supranuclear palsy, Corticobasal degeneration, Frontotemporal lobar degeneration, Frontotemporal dementia and Grey matter in the investigation of issues within Standardized uptake value. Her Dementia research incorporates elements of Positron emission tomography and Neurodegeneration.
Suzanne L. Baker studied Positron emission tomography and Precision medicine that intersect with Internal medicine. Her work on Neuropathology, Alzheimer's disease and Posterior cortical atrophy as part of general Pathology study is frequently linked to Neurofibrillary tangle and Association, therefore connecting diverse disciplines of science. Her research in Amyloid intersects with topics in Executive functions, Asymptomatic, Cognitive test and Cognitive decline.
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PET Imaging of Tau Deposition in the Aging Human Brain
Michael Schöll;Michael Schöll;Samuel N. Lockhart;Daniel R. Schonhaut;James P. O’Neil.
Neuron (2016)
Tau PET patterns mirror clinical and neuroanatomical variability in Alzheimer's disease.
Rik Ossenkoppele;Rik Ossenkoppele;Rik Ossenkoppele;Daniel R. Schonhaut;Daniel R. Schonhaut;Michael Schöll;Michael Schöll;Samuel N. Lockhart.
Brain (2016)
Episodic memory loss is related to hippocampal-mediated -amyloid deposition in elderly subjects
E. C. Mormino;J. T. Kluth;C. M. Madison;G. D. Rabinovici;G. D. Rabinovici;G. D. Rabinovici.
Brain (2009)
Tau pathology and neurodegeneration contribute to cognitive impairment in Alzheimer's disease.
Alexandre Bejanin;Daniel R Schonhaut;Renaud La Joie;Joel H Kramer.
Brain (2017)
11C-PIB PET imaging in Alzheimer disease and frontotemporal lobar degeneration
G. D. Rabinovici;A. J. Furst;J. P. O'Neil;C. A. Racine.
Neurology (2007)
Striatal Dopamine and Working Memory
Susan M. Landau;Rayhan Lal;James P. O'Neil;Suzanne Baker.
Cerebral Cortex (2009)
Comparison of multiple tau-PET measures as biomarkers in aging and Alzheimer's disease
Anne Maass;Susan Landau;Suzanne L Baker;Andy Horng.
NeuroImage (2017)
Relationships between Beta-Amyloid and Functional Connectivity in Different Components of the Default Mode Network in Aging
Elizabeth C. Mormino;Andre Smiljic;Amynta O. Hayenga;Susan H. Onami.
Cerebral Cortex (2011)
Increased metabolic vulnerability in early-onset Alzheimer’s disease is not related to amyloid burden
Gil D. Rabinovici;Ansgar J. Furst;Ansgar J. Furst;Ansgar J. Furst;Adi Alkalay;Adi Alkalay;Caroline A. Racine.
Brain (2010)
Measurement of Longitudinal β-Amyloid Change with 18F-Florbetapir PET and Standardized Uptake Value Ratios
Susan Marie Landau;Allison Fero;Suzanne L Baker;Robert A. Koeppe.
The Journal of Nuclear Medicine (2015)
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