What is he best known for?
The fields of study he is best known for:
His scientific interests lie mostly in Biochemistry, Recoverin, Retina, Retinal and Molecular biology.
His Biochemistry study combines topics in areas such as Biophysics, Calcium and Cell biology.
His Cell biology research is multidisciplinary, relying on both Mutation, Cyclase and Mutant.
His work on Visual phototransduction and Bipolar neuron as part of general Retina study is frequently linked to Immunostaining, bridging the gap between disciplines.
Alexander M. Dizhoor focuses mostly in the field of Retinal, narrowing it down to topics relating to Stimulation and, in certain cases, Extracellular, Wild type and Transmembrane domain.
His Molecular biology research integrates issues from HEK 293 cells, Fusion protein and Inner plexiform layer.
His most cited work include:
- Ectopic Expression of a Microbial-Type Rhodopsin Restores Visual Responses in Mice with Photoreceptor Degeneration (588 citations)
- Ectopic Expression of a Microbial-Type Rhodopsin Restores Visual Responses in Mice with Photoreceptor Degeneration (588 citations)
- The human photoreceptor membrane guanylyl cyclase, RetGC, is present in outer segments and is regulated by calcium and a soluble activator (297 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Cell biology, Retinal, Biochemistry, GUCY2D and Guanylate cyclase.
His Cell biology research incorporates elements of Calcium-binding protein, Calcium, Genetically modified mouse and Cyclase.
His Retinal research is multidisciplinary, incorporating elements of Retina, Membrane and Isozyme.
His study in Retina is interdisciplinary in nature, drawing from both Opsin, Anatomy and Darkness.
His research investigates the connection between Biochemistry and topics such as Biophysics that intersect with problems in Rhodopsin and Enzyme activator.
Alexander M. Dizhoor has researched GUCY2D in several fields, including HEK 293 cells, Molecular biology and Genetic enhancement.
He most often published in these fields:
- Cell biology (58.09%)
- Retinal (43.38%)
- Biochemistry (33.09%)
What were the highlights of his more recent work (between 2014-2021)?
- Retinal (43.38%)
- Cell biology (58.09%)
- GUCY2D (31.62%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Retinal, Cell biology, GUCY2D, Guanylate cyclase and Retinal degeneration.
His research integrates issues of Retina, Membrane and Cyclase in his study of Retinal.
His Retina research includes elements of Molecular biology and Channelrhodopsin.
His studies in Cell biology integrate themes in fields like Calcium, Visual phototransduction, Guanylyl Cyclase Activating Proteins and Retinal degeneration 3.
His work carried out in the field of Visual phototransduction brings together such families of science as Calcium-binding protein and Adaptation.
His work deals with themes such as Genetically modified mouse, Transgene and Genetic enhancement, which intersect with GUCY2D.
Between 2014 and 2021, his most popular works were:
- Optogenetic Approaches to Restoring Vision. (26 citations)
- Gene Therapy Fully Restores Vision to the All-Cone Nrl(-/-) Gucy2e(-/-) Mouse Model of Leber Congenital Amaurosis-1. (26 citations)
- Evaluating the role of retinal membrane guanylyl cyclase 1 (RetGC1) domains in binding guanylyl cyclase-activating proteins (GCAPs). (23 citations)
In his most recent research, the most cited papers focused on:
The scientist’s investigation covers issues in Cyclase, Retinal, Visual phototransduction, Cell biology and GUCY2D.
His Cyclase study integrates concerns from other disciplines, such as Biophysics and Intracellular.
His research ties Retina and Retinal together.
Retina is a subfield of Neuroscience that Alexander M. Dizhoor studies.
The Visual phototransduction study which covers Calcium-binding protein that intersects with Genetically modified mouse, Anatomy, Transgene and Activator.
The concepts of his Cell biology study are interwoven with issues in Biochemistry and Binding site.
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