2013 - Fellow of the American Association for the Advancement of Science (AAAS)
1997 - Fellow of John Simon Guggenheim Memorial Foundation
Gordon L. Fain mainly focuses on Biophysics, Cell biology, Retina, Darkness and Rod. Gordon L. Fain is studying Membrane potential, which is a component of Biophysics. His biological study spans a wide range of topics, including Transduction, Vertebrate, Retinitis pigmentosa, Adaptation and Visual phototransduction.
His research integrates issues of Opsin, Retinal Rod Photoreceptor Cells and Transducin in his study of Visual phototransduction. The Retina study which covers Toad that intersects with Receptor coupling, Amplitude and Sensitivity. Gordon L. Fain has researched Darkness in several fields, including Mutation, Gene, Transgene and Calcium.
Biophysics, Cell biology, Anatomy, Rhodopsin and Retina are his primary areas of study. His Biophysics study combines topics in areas such as Darkness, Calcium, Retinal Rod Photoreceptor Cells and Transducin. Within one scientific family, he focuses on topics pertaining to Adaptation under Darkness, and may sometimes address concerns connected to Photopigment and Rod Photoreceptors.
He has included themes like Retinal degeneration, Retinitis pigmentosa, Adaptation, Opsin and Ciliary body in his Cell biology study. His studies examine the connections between Anatomy and genetics, as well as such issues in Transduction, with regards to Duplex retina, Lamprey and Vertebrate. Gordon L. Fain combines subjects such as Phosphodiesterase and Visual phototransduction with his study of Rhodopsin.
His main research concerns Biophysics, Retina, Vertebrate, Lamprey and Rhodopsin. In Biophysics, Gordon L. Fain works on issues like Transducin, which are connected to Patch clamp. His Retina research includes elements of Human physiology, Transduction, Retinal and Phosphodiesterase.
His Vertebrate study integrates concerns from other disciplines, such as Evolutionary biology and Anatomy. His studies deal with areas such as Retinal pigment epithelium and Visual phototransduction as well as Rhodopsin. His Visual phototransduction research incorporates elements of Mutant, Programmed cell death and Cell biology.
Gordon L. Fain mostly deals with Opsin, Rhodopsin, Retinal, Lamprey and Vertebrate. His Rhodopsin research incorporates themes from Retinal pigment epithelium, Biophysics, Retinal Cone Photoreceptor Cells and Anatomy. His Retinal research integrates issues from Retina and Cell biology.
The Retina study combines topics in areas such as Color vision, Cell specific and Rna expression. His work in Lamprey addresses issues such as Darkness, which are connected to fields such as Adaptation. His Retinal Rod Photoreceptor Cells research is multidisciplinary, incorporating perspectives in Photosensitivity and Transducin.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Adaptation in Vertebrate Photoreceptors
Gordon L. Fain;Hugh R. Matthews;M. Carter Cornwall;Yiannis Koutalos.
Physiological Reviews (2001)
Photoreceptor light adaptation is mediated by cytoplasmic calcium concentration.
H. R. Matthews;R. L. W. Murphy;G. L. Fain;T. D. Lamb.
ATP Consumption by Mammalian Rod Photoreceptors in Darkness and in Light
Haruhisa Okawa;Alapakkam P. Sampath;Simon B. Laughlin;Gordon L. Fain.
Current Biology (2008)
Sensitivity of toad rods: Dependence on wave‐length and background illumination.
G L Fain.
The Journal of Physiology (1976)
Bleached pigment activates transduction in isolated rods of the salamander retina.
M C Cornwall;G L Fain.
The Journal of Physiology (1994)
Phototransduction and the evolution of photoreceptors.
Gordon L. Fain;Roger Hardie;Simon B. Laughlin.
Current Biology (2010)
Measurement of cytoplasmic calcium concentration in the rods of wild-type and transducin knock-out mice
Michael L. Woodruff;A. P. Sampath;Hugh R. Matthews;N. V. Krasnoperova.
The Journal of Physiology (2002)
Photoreceptor degeneration in vitamin A deprivation and retinitis pigmentosa: the equivalent light hypothesis.
Gordon L. Fain;John E. Lisman.
Experimental Eye Research (1993)
Membrane conductances of photoreceptors.
G.L. Fain;G.L. Fain;J.E. Lisman;J.E. Lisman.
Progress in Biophysics & Molecular Biology (1981)
Spontaneous activity of opsin apoprotein is a cause of Leber congenital amaurosis.
Michael L Woodruff;Zhongyan Wang;Hae Yun Chung;T Michael Redmond.
Nature Genetics (2003)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: