What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Internal medicine
His main research concerns Cell biology, Ceramide, Apoptosis, Sphingomyelin and Signal transduction.
His study in Cell biology focuses on Sphingolipid in particular.
His studies in Ceramide integrate themes in fields like Oxidative stress, Cell, Protein kinase A, Second messenger system and Programmed cell death.
His Apoptosis research is multidisciplinary, relying on both Cell culture, Cancer research, Endocrinology, Cancer cell and Internal medicine.
His Cancer research study combines topics from a wide range of disciplines, such as Tumor necrosis factor alpha, Blockade, Protein kinase B and Cell growth.
Thierry Levade has researched Signal transduction in several fields, including Phospholipase, Enzyme, Distribution and Sphingomyelin phosphodiesterase.
His most cited work include:
- Daunorubicin-induced apoptosis: triggering of ceramide generation through sphingomyelin hydrolysis. (334 citations)
- Glutathione Regulation of Neutral Sphingomyelinase in Tumor Necrosis Factor-α-induced Cell Death (333 citations)
- Signalling sphingomyelinases: which, where, how and why? (299 citations)
What are the main themes of his work throughout his whole career to date?
Thierry Levade mostly deals with Ceramide, Cell biology, Biochemistry, Sphingolipid and Sphingomyelin.
His Ceramide study integrates concerns from other disciplines, such as Sphingosine, Cancer research and Lipid signaling.
His research integrates issues of Cancer cell, Caspase, Programmed cell death and Autophagy in his study of Cell biology.
His studies deal with areas such as Sphingomyelin synthase and Melanoma as well as Sphingolipid.
His Sphingomyelin research is multidisciplinary, incorporating perspectives in Lysosome and Niemann–Pick disease.
His Apoptosis research includes elements of Molecular biology and Cell growth.
He most often published in these fields:
- Ceramide (42.95%)
- Cell biology (33.33%)
- Biochemistry (27.24%)
What were the highlights of his more recent work (between 2013-2021)?
- Ceramide (42.95%)
- Sphingolipid (26.28%)
- Cancer research (19.23%)
In recent papers he was focusing on the following fields of study:
His primary areas of study are Ceramide, Sphingolipid, Cancer research, Melanoma and Cell biology.
His study in Ceramide is interdisciplinary in nature, drawing from both Cancer cell, Endocrinology and Sphingomyelin.
His biological study spans a wide range of topics, including Ataxia, Downregulation and upregulation, Mitochondrion and Ichthyosis.
His Cancer research research is multidisciplinary, incorporating elements of Tumor necrosis factor alpha, Immune system and Immunology.
He interconnects Tumor microenvironment, Cancer, Sphingosine, Sphingosine-1-phosphate and Microphthalmia-associated transcription factor in the investigation of issues within Melanoma.
He combines subjects such as Lysosomal storage disease, Cell, Mucopolysaccharidosis and Genetic enhancement with his study of Cell biology.
Between 2013 and 2021, his most popular works were:
- A Review of Gaucher Disease Pathophysiology, Clinical Presentation and Treatments (190 citations)
- TNFα blockade overcomes resistance to anti-PD-1 in experimental melanoma. (130 citations)
- Legionella pneumophila S1P-lyase targets host sphingolipid metabolism and restrains autophagy. (69 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
Thierry Levade spends much of his time researching Cancer research, Ceramide, Cell biology, Melanoma and Immunology.
In his research on the topic of Cancer research, Cytokine, Immune system, Cancer, Arthritis and Lymphocytosis is strongly related with Tumor necrosis factor alpha.
His Ceramide research is under the purview of Biochemistry.
His work in the fields of Sphingomyelin and Toxin overlaps with other areas such as Fumonisin.
His Cell biology study incorporates themes from Cancer cell, Cell, Membrane fluidity and Transduction.
The study incorporates disciplines such as Sphingosine, HEK 293 cells, RNA interference, Sphingosine kinase 1 and Immunotherapy in addition to Melanoma.
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