D-Index & Metrics Best Publications

Didier Hannequin

Grenoble Alpes University
France

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name Citations Publications World Ranking National Ranking

Medicine D-index 80 Citations 30,716 361 World Ranking 11708 National Ranking 350

Genetics D-index 75 Citations 28,404 208 World Ranking 1225 National Ranking 50

Overview

What is he best known for?

The fields of study Didier Hannequin is best known for:

Gene
Alzheimer's disease
Genetics

In his study, Didier Hannequin carries out multidisciplinary Genetics and Locus (genetics) research. He integrates Locus (genetics) and Gene in his research. His work on PDGFRB expands to the thematically related Gene. His research on PDGFRB frequently links to adjacent areas such as Genetics. His study brings together the fields of Dementia and Disease. Didier Hannequin undertakes interdisciplinary study in the fields of Dementia and Frontotemporal dementia through his research. Frontotemporal dementia and Disease are frequently intertwined in his study. His multidisciplinary approach integrates Internal medicine and Psychiatry in his work. He connects Psychiatry with Internal medicine in his study.

His most cited work include:

Genome-wide association study identifies variants at CLU and CR1 associated with Alzheimer's disease (1794 citations)
Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease (1614 citations)
APP locus duplication causes autosomal dominant early-onset Alzheimer disease with cerebral amyloid angiopathy (1069 citations)

What are the main themes of his work throughout his whole career to date

His work on Alzheimer's disease expands to the thematically related Disease. He undertakes multidisciplinary investigations into Alzheimer's disease and Dementia in his work. His Dementia study frequently links to adjacent areas such as Pathology. As part of his studies on Pathology, Didier Hannequin often connects relevant areas like Frontotemporal dementia. His Frontotemporal dementia study frequently intersects with other fields, such as Disease. Didier Hannequin performs integrative study on Internal medicine and Psychiatry in his works. Didier Hannequin combines Psychiatry and Internal medicine in his studies. Didier Hannequin performs multidisciplinary study on Genetics and Allele in his works. His multidisciplinary approach integrates Allele and Genetics in his work.

Didier Hannequin most often published in these fields:

Disease (70.00%)
Internal medicine (57.37%)
Genetics (54.21%)

What were the highlights of his more recent work (between 2018-2022)?

Disease (91.67%)
Dementia (83.33%)
Internal medicine (75.00%)

In recent works Didier Hannequin was focusing on the following fields of study:

In the field of C9orf72 and Primary progressive aphasia Didier Hannequin studies Frontotemporal dementia. He connects Primary progressive aphasia with Frontotemporal dementia in his research. Voxel-based morphometry and Grey matter are the subject areas of his White matter study. The Parkinson's disease portion of his research involves studies in Deep brain stimulation and Lewy body. Didier Hannequin conducted interdisciplinary study in his works that combined Lewy body and Parkinson's disease. He integrates many fields, such as Disease and Medical diagnosis, in his works. Didier Hannequin undertakes interdisciplinary study in the fields of Dementia and Neurocognitive through his research. While working in this field, he studies both Internal medicine and Physical medicine and rehabilitation. In his research, he performs multidisciplinary study on Physical medicine and rehabilitation and Internal medicine.

Between 2018 and 2022, his most popular works were:

A trial of gantenerumab or solanezumab in dominantly inherited Alzheimer’s disease (122 citations)
Neurite density is reduced in the presymptomatic phase of C9orf72 disease (40 citations)
Nucleus Basalis of Meynert Stimulation for Lewy Body Dementia (18 citations)

In his most recent research, the most cited works focused on:

Parkinson's disease
Dementia
Amyotrophic lateral sclerosis

His study connects Cognitive decline and Dementia. Cognitive decline is closely attributed to Dementia in his research. His work on Disease is being expanded to include thematically relevant topics such as Lewy body. His Lewy body study frequently links to other fields, such as Disease. He combines Internal medicine and Physical medicine and rehabilitation in his research. Didier Hannequin combines Physical medicine and rehabilitation and Internal medicine in his studies. His study connects Asymptomatic and Pathology. He regularly ties together related areas like Pathology in his Asymptomatic studies. He integrates Oncology with Gastroenterology in his study.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease

Jean-Charles Lambert;Jean-Charles Lambert;Jean-Charles Lambert;Carla A Ibrahim-Verbaas;Denise Harold;Adam C Naj.
Nature Genetics (2013)

3630 Citations

Genome-wide association study indentifies variants at CLU and CR1 associated with Alzheimer’s disease

J Lambert;S Heath;G Even;D Campion.
Nature Genetics (2009)

2641 Citations

Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease.

Paul Hollingworth;Denise Harold;Rebecca Sims;Amy Gerrish.
Nature Genetics (2011)

1904 Citations

APP locus duplication causes autosomal dominant early-onset Alzheimer disease with cerebral amyloid angiopathy

Anne Rovelet-Lecrux;Didier Hannequin;Gregory Raux;Nathalie Le Meur.
Nature Genetics (2006)

1410 Citations

Gene-wide analysis detects two new susceptibility genes for Alzheimer's disease.

Valentina Escott-Price;Céline Bellenguez;Li-San Wang;Seung-Hoan Choi.
PLOS ONE (2014)

1380 Citations

Analysis of shared heritability in common disorders of the brain

Verneri Anttila;Verneri Anttila;Brendan Bulik-Sullivan;Brendan Bulik-Sullivan;Hilary K. Finucane;Raymond K. Walters;Raymond K. Walters.
Science (2018)

1200 Citations

Early-Onset Autosomal Dominant Alzheimer Disease: Prevalence, Genetic Heterogeneity, and Mutation Spectrum

Dominique Campion;Cécile Dumanchin;Didier Hannequin;Bruno Dubois.
American Journal of Human Genetics (1999)

986 Citations

Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease

Rebecca Sims;Sven J. Van Der Lee;Adam C. Naj;Céline Bellenguez;Céline Bellenguez.
Nature Genetics (2017)

683 Citations

APOE and Alzheimer disease: a major gene with semi-dominant inheritance

E Genin;D Hannequin;D Wallon;K Sleegers.
Molecular Psychiatry (2011)

614 Citations

A Polymorphism in CALHM1 Influences Ca2+ Homeostasis, Aβ Levels, and Alzheimer's Disease Risk

Ute Dreses-Werringloer;Jean Charles Lambert;Valérie Vingtdeux;Haitian Zhao.
Cell (2008)

359 Citations

If you think any of the details on this page are incorrect, let us know.