H-Index & Metrics Best Publications

H-Index & Metrics

Discipline name H-index Citations Publications World Ranking National Ranking
Microbiology D-index 49 Citations 10,346 127 World Ranking 2220 National Ranking 916

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Virus
  • DNA

His primary scientific interests are in Molecular biology, Epstein–Barr virus, Virology, Virus and LYN. His Molecular biology research includes elements of SH2 domain, Protein tyrosine phosphatase and Mutation, Gene, Mutant. His work deals with themes such as Cell culture, Gene expression, Antibody, B cell and Oncovirus, which intersect with Epstein–Barr virus.

His studies examine the connections between Virology and genetics, as well as such issues in Cell surface receptor, with regards to Heparan sulfate and Cell type. His study in Virus is interdisciplinary in nature, drawing from both Transfection and Transmembrane domain. His studies in LYN integrate themes in fields like B-cell receptor and Syk.

His most cited work include:

  • Induction of bcl-2 expression by Epstein-Barr virus latent membrane protein 1 protects infected B cells from programmed cell death. (1040 citations)
  • Epstein-Barr Virus LMP2A Drives B Cell Development and Survival in the Absence of Normal B Cell Receptor Signals (517 citations)
  • CDC42 and CDC43, two additional genes involved in budding and the establishment of cell polarity in the yeast Saccharomyces cerevisiae. (508 citations)

What are the main themes of his work throughout his whole career to date?

His main research concerns Epstein–Barr virus, Virology, Virus, Molecular biology and Cell biology. His Epstein–Barr virus study incorporates themes from Cancer research, Lipid bilayer fusion, B cell, Herpesvirus glycoprotein B and Lymphoma. His studies link Fusion protein with Virology.

In his research on the topic of Virus, Cell type is strongly related with Receptor. His work focuses on many connections between Molecular biology and other disciplines, such as Mutant, that overlap with his field of interest in Mutation. His study looks at the relationship between Cell biology and topics such as B-cell receptor, which overlap with Transgene.

He most often published in these fields:

  • Epstein–Barr virus (45.98%)
  • Virology (37.95%)
  • Virus (36.61%)

What were the highlights of his more recent work (between 2013-2021)?

  • Virus (36.61%)
  • Virology (37.95%)
  • Viral entry (14.73%)

In recent papers he was focusing on the following fields of study:

His primary areas of investigation include Virus, Virology, Viral entry, Herpes simplex virus and Epstein–Barr virus. His Virus research is multidisciplinary, relying on both HEK 293 cells, Erythropoietin-producing hepatocellular receptor, Cell type and Lymphoma. His Virology research includes themes of Phenotype and Fusion protein.

Richard Longnecker has included themes like Viral envelope, Kaposi's sarcoma-associated herpesvirus, Receptor, Entry into host and Viral protein in his Viral entry study. His Herpes simplex virus research integrates issues from Interferon, Viral encephalitis and Viral replication. The study incorporates disciplines such as Tropism, Cancer research, B cell, Cell biology and breakpoint cluster region in addition to Epstein–Barr virus.

Between 2013 and 2021, his most popular works were:

  • Ephrin receptor A2 is a functional entry receptor for Epstein-Barr virus. (79 citations)
  • The COMPLEXity in herpesvirus entry. (50 citations)
  • Latent Membrane Protein 2 (LMP2). (46 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Virus
  • DNA

His scientific interests lie mostly in Virology, Cell biology, Virus, Lipid bilayer fusion and Epstein–Barr virus. His work in Cell biology addresses issues such as Cell fusion, which are connected to fields such as Tyrosine kinase, Oncovirus and Erythropoietin-producing hepatocellular receptor. His study explores the link between Virus and topics such as HEK 293 cells that cross with problems in EPH receptor A2, Kaposi's sarcoma-associated herpesvirus, Cell type and Ephrin A2.

The various areas that Richard Longnecker examines in his Lipid bilayer fusion study include Viral envelope and Membrane glycoproteins. His studies deal with areas such as Cancer research, Cell growth, Cancer cell, Apoptosis and breakpoint cluster region as well as Epstein–Barr virus. His research in Herpesvirus glycoprotein B intersects with topics in Fusion protein and Glycoprotein.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Induction of bcl-2 expression by Epstein-Barr virus latent membrane protein 1 protects infected B cells from programmed cell death.

Sheila Henderson;Martin Rowe;Chris Gregory;Debbie Croom-Carter.
Cell (1991)

1667 Citations

Epstein-Barr Virus LMP2A Drives B Cell Development and Survival in the Absence of Normal B Cell Receptor Signals

Robert G Caldwell;Joanna B Wilson;Steven J Anderson;Richard Longnecker.
Immunity (1998)

793 Citations

Herpesvirus entry: an update.

Patricia G. Spear;Richard Longnecker.
Journal of Virology (2003)

774 Citations

CDC42 and CDC43, two additional genes involved in budding and the establishment of cell polarity in the yeast Saccharomyces cerevisiae.

Alison E.M. Adams;Douglas I. Johnson;Douglas I. Johnson;Richard M. Longnecker;Barbara F. Sloat.
Journal of Cell Biology (1990)

730 Citations

BTK/TEC KINASES REGULATE SUSTAINED INCREASES IN INTRACELLULAR CA2+ FOLLOWING B-CELL RECEPTOR ACTIVATION

Anne Catherine Fluckiger;Zuomei Li;Roberta M. Kato;Matthew I. Wahl.
The EMBO Journal (1998)

528 Citations

Fusing structure and function: a structural view of the herpesvirus entry machinery

Sarah A. Connolly;Julia O. Jackson;Theodore S. Jardetzky;Richard Longnecker.
Nature Reviews Microbiology (2011)

450 Citations

Integral membrane protein 2 of Epstein-Barr virus regulates reactivation from latency through dominant negative effects on protein-tyrosine kinases.

Cheryl L. Miller;Anne L. Burkhardt;Jennifer H. Lee;Becky Stealey.
Immunity (1995)

433 Citations

An integral membrane protein (LMP2) blocks reactivation of Epstein-Barr virus from latency following surface immunoglobulin crosslinking.

Cheryl L. Miller;Jennifer H. Lee;Elliott Kieff;Richard Longnecker.
Proceedings of the National Academy of Sciences of the United States of America (1994)

351 Citations

In Vivo Behavior of Genetically Engineered Herpes Simplex Viruses R7017 and R7020: Construction and Evaluation in Rodents

Bernard Meignier;Richard Longnecker;Bernard Roizman.
The Journal of Infectious Diseases (1988)

342 Citations

Epstein-barr virus

Richard M Longnecker;Elliott Kieff;Jeffrey I. Cohen.
(2013)

328 Citations

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