Member of the Association of American Physicians
Rafeul Alam mainly focuses on Immunology, Eosinophil, Molecular biology, Cytokine and Inflammation. All of his Immunology and Chemokine, Immunoglobulin E, Macrophage inflammatory protein, Monocyte and Basophil investigations are sub-components of the entire Immunology study. Rafeul Alam has researched Eosinophil in several fields, including Eosinophilia, Bronchoalveolar lavage, CCL5 and Interleukin 4.
His Molecular biology study combines topics in areas such as Endocrinology, JAK-STAT signaling pathway, MAPK14, Mitogen-activated protein kinase kinase and Internal medicine. His Cytokine research incorporates themes from Receptor, Glucocorticoid receptor, STAT protein and MAPK/ERK pathway. The study incorporates disciplines such as Reactive oxygen species, Immunopathology and Allergy in addition to Inflammation.
His main research concerns Immunology, Asthma, Cell biology, Cytokine and Histamine. His Immunology study often links to related topics such as Peripheral blood mononuclear cell. His Cytokine research is multidisciplinary, incorporating elements of Chemokine and Monocyte.
His Monocyte research is multidisciplinary, incorporating perspectives in Chemotaxis and Macrophage inflammatory protein. The concepts of his Histamine study are interwoven with issues in Lymphokine, Basophil and Antibody. Rafeul Alam combines subjects such as Cancer research, Receptor tyrosine kinase and Proto-oncogene tyrosine-protein kinase Src with his study of Tyrosine kinase.
His primary areas of study are Immunology, Asthma, Innate lymphoid cell, Bronchoalveolar lavage and Inflammation. Allergy, Interleukin 33, Interleukin 13, Specific immunotherapy and Antigen are among the areas of Immunology where the researcher is concentrating his efforts. His Asthma research entails a greater understanding of Internal medicine.
His Innate lymphoid cell research includes elements of Thymic stromal lymphopoietin, Cytokine milieu, Cytokine, Downregulation and upregulation and Adoptive cell transfer. He interconnects Eosinophil, Endocrinology, Neutrophilic inflammation, Severe asthma and Subclinical infection in the investigation of issues within Bronchoalveolar lavage. His biological study spans a wide range of topics, including T cell, Lung and Pathogenesis.
Rafeul Alam mostly deals with Immunology, Asthma, Bronchoalveolar lavage, Inflammation and Allergy. Rafeul Alam has researched Immunology in several fields, including Aspirin and Disease. In his research on the topic of Asthma, Airway obstruction and Ionomycin is strongly related with CD8.
His studies in Bronchoalveolar lavage integrate themes in fields like Eosinophil, Endocrinology, Subclinical infection and Pathogenesis. As part of one scientific family, he deals mainly with the area of Eosinophil, narrowing it down to issues related to the GATA3, and often Interleukin 17. His work in Allergy addresses issues such as Immunotherapy, which are connected to fields such as Peripheral blood mononuclear cell and ELISPOT.
A Tyrosine Kinase Created by Fusion of the PDGFRA and FIP1L1 Genes as a Therapeutic Target of Imatinib in Idiopathic Hypereosinophilic Syndrome
Jan Cools;Jan Cools;Daniel J. DeAngelo;Jason Gotlib;Elizabeth H. Stover.
The New England Journal of Medicine (2003)
Long term prevention of allergic lung inflammation in a mouse model of asthma by CpG oligodeoxynucleotides.
Sanjiv Sur;James S. Wild;Barun K. Choudhury;Nilanjana Sur.
Journal of Immunology (1999)
RANTES is a chemotactic and activating factor for human eosinophils.
R Alam;S Stafford;P Forsythe;R Harrison.
Journal of Immunology (1993)
Increased MCP-1, RANTES, and MIP-1alpha in bronchoalveolar lavage fluid of allergic asthmatic patients.
R Alam;J York;M Boyars;S Stafford.
American Journal of Respiratory and Critical Care Medicine (1996)
Eotaxin induces degranulation and chemotaxis of eosinophils through the activation of ERK2 and p38 mitogen-activated protein kinases
Gita T. Kampen;Susan Stafford;Tetsuya Adachi;Tan Jinquan.
ROS generated by pollen NADPH oxidase provide a signal that augments antigen-induced allergic airway inflammation
Istvan Boldogh;Attila Bacsi;Barun K. Choudhury;Nilesh Dharajiya.
Journal of Clinical Investigation (2005)
IFN-γ-Inducing Factor (IL-18) Increases Allergic Sensitization, Serum IgE, Th2 Cytokines, and Airway Eosinophilia in a Mouse Model of Allergic Asthma
James S. Wild;Anastasia Sigounas;Nilanjana Sur;Mohammed S. Siddiqui.
Journal of Immunology (2000)
Cell-Specific Expression of RANTES, MCP-1, and MIP-1α by Lower Airway Epithelial Cells and Eosinophils Infected with Respiratory Syncytial Virus
Barbara Olszewska-Pazdrak;Antonella Casola;Tadahito Saito;Rafeul Alam.
Journal of Virology (1998)
Increased frequency of dual-positive TH2/TH17 cells in bronchoalveolar lavage fluid characterizes a population of patients with severe asthma.
Chaoyu Irvin;Iram Zafar;James Good;Donald Rollins.
The Journal of Allergy and Clinical Immunology (2014)
The mechanism of IL-5 signal transduction
Tetsuya Adachi;Rafeul Alam.
American Journal of Physiology-cell Physiology (1998)
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