What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Internal medicine
His main research concerns Endocrinology, Internal medicine, Insulin, ATP-binding cassette transporter and Islet.
His Endocrinology research is multidisciplinary, incorporating perspectives in Peripheral blood mononuclear cell, Mitochondrion and Mitochondrial DNA.
His Mitochondrial Degradation research extends to Internal medicine, which is thematically connected.
His Insulin research integrates issues from Gymnema sylvestre, Secretion, Receptor, Endocannabinoid system and Cannabinoid receptor.
His study in ATP-binding cassette transporter is interdisciplinary in nature, drawing from both ATP hydrolysis, Protein structure, Transmembrane domain and Protein family.
His Islet study incorporates themes from Kidney and Transplantation.
His most cited work include:
- The ABC transporter structure and mechanism: perspectives on recent research (407 citations)
- Impaired glucose homeostasis and mitochondrial abnormalities in offspring of rats fed a fat-rich diet in pregnancy (277 citations)
- Predictors of success and failure in pulmonary rehabilitation (249 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are Internal medicine, Endocrinology, Islet, Insulin and Cell biology.
His study explores the link between Internal medicine and topics such as Diabetes mellitus that cross with problems in Pancreas.
His Endocrinology study combines topics in areas such as Receptor and Cell culture.
His research in Islet intersects with topics in Transplantation, Immunology, Type 1 diabetes, Mesenchymal stem cell and In vivo.
His Insulin study integrates concerns from other disciplines, such as Gymnema sylvestre, Secretion, Somatostatin and Kisspeptin.
His Cell biology research is multidisciplinary, incorporating perspectives in Endothelial stem cell and Cadherin.
He most often published in these fields:
- Internal medicine (44.59%)
- Endocrinology (41.44%)
- Islet (32.43%)
What were the highlights of his more recent work (between 2014-2021)?
- Islet (32.43%)
- Internal medicine (44.59%)
- Endocrinology (41.44%)
In recent papers he was focusing on the following fields of study:
Peter M. Jones mainly focuses on Islet, Internal medicine, Endocrinology, Cell biology and Insulin.
His Islet research includes themes of In vitro, Transplantation, Hepatic stellate cell, Apoptosis and In vivo.
His Internal medicine research includes elements of Diabetes mellitus and MEDLINE.
His work carried out in the field of Endocrinology brings together such families of science as Receptor and Cannabinoid receptor.
His Cell biology research is multidisciplinary, relying on both Biological activity, Protein subunit, Beta cell and Blot.
His Insulin study combines topics in areas such as Hormone, Type 2 Diabetes Mellitus, Secretion and WNT5A.
Between 2014 and 2021, his most popular works were:
- Altered Mitochondrial Function, Mitochondrial DNA and Reduced Metabolic Flexibility in Patients With Diabetic Nephropathy. (81 citations)
- Favorable outcome of experimental islet xenotransplantation without immunosuppression in a nonhuman primate model of diabetes. (44 citations)
- Annexin A1 Is a Key Modulator of Mesenchymal Stromal Cell–Mediated Improvements in Islet Function (43 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
Peter M. Jones mainly investigates Islet, Transplantation, Internal medicine, Endocrinology and Diabetes mellitus.
The study incorporates disciplines such as Molecular biology and Extracellular matrix, Stem cell, Cell biology in addition to Islet.
Peter M. Jones combines subjects such as Adipose tissue and Insulin with his study of Cell biology.
The concepts of his Transplantation study are interwoven with issues in Cancer research, Immunology, Type 1 diabetes, Mesenchymal stem cell and In vivo.
His biological study spans a wide range of topics, including Mitochondrion and RHOA.
His Endocrinology study incorporates themes from Mitochondrial DNA and Mitophagy, Mitochondrial Degradation.
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