His primary scientific interests are in Cell biology, Biochemistry, Molecular biology, Cyclin B and Centrosome. His Cell biology study combines topics from a wide range of disciplines, such as Ubiquitin ligase, Cyclin A and BBSome. His Biochemistry research is multidisciplinary, incorporating perspectives in Cancer cell and Cancer.
In his research, Proto-oncogene tyrosine-protein kinase Src, Myristoylation, Oncogene and Phosphotyrosine-binding domain is intimately related to ABL, which falls under the overarching field of Molecular biology. He works mostly in the field of Cyclin B, limiting it down to topics relating to Anaphase-promoting complex and, in certain cases, APC/C activator protein CDH1, Beta-Transducin Repeat-Containing Proteins and F-box protein. His Mitosis research incorporates elements of Cell cycle and Mitotic exit.
The scientist’s investigation covers issues in Cell biology, Cilium, Molecular biology, Mitosis and Ubiquitin ligase. His Cell biology research integrates issues from Cyclin B, Cell cycle, Cyclin A, Anaphase-promoting complex and Cyclin. His research in Cyclin B tackles topics such as Cyclin A2 which are related to areas like Cyclin D.
His Cilium research includes themes of Ciliopathies, Signal transduction and Intraflagellar transport. His Molecular biology research is multidisciplinary, incorporating elements of ABL and Phosphorylation. His Mitosis research incorporates themes from Mitotic exit, Spindle pole body, Centrosome and PLK1.
His primary areas of investigation include Cell biology, Cilium, Computational biology, KRAS and Stem cell. Peter K. Jackson works in the field of Cell biology, namely G protein-coupled receptor. The various areas that Peter K. Jackson examines in his Cilium study include Immunology, Subcellular localization, Organelle and Respiratory system.
His work deals with themes such as Plasma protein binding, Protein subunit and Ubiquitin ligase, which intersect with Subcellular localization. Peter K. Jackson has included themes like RHOA, CRISPR, Neuroblastoma RAS viral oncogene homolog and Effector in his Computational biology study. His biological study spans a wide range of topics, including Carcinogenesis, Cancer, Cancer research and Synthetic lethality.
Peter K. Jackson spends much of his time researching Cilium, Cell biology, Cancer cell, Gene and Cancer research. As part of one scientific family, Peter K. Jackson deals mainly with the area of Cilium, narrowing it down to issues related to the Respiratory system, and often Pathogenesis, Immunology, Lung and Organelle. His study of G protein-coupled receptor is a part of Cell biology.
His research on Cancer cell also deals with topics like
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Neonatal lethality and lymphopenia in mice with a homozygous disruption of the c-abl proto-oncogene
Victor L.J. Tybulewicz;Camila E. Crawford;Peter K. Jackson;Roderick T. Bronson.
A Core Complex of BBS Proteins Cooperates with the GTPase Rab8 to Promote Ciliary Membrane Biogenesis
Maxence V. Nachury;Alexander V. Loktev;Qihong Zhang;Christopher J. Westlake.
Mitosis in transition
Randall W. King;Peter K. Jackson;Marc W. Kirschner.
The lore of the RINGs: substrate recognition and catalysis by ubiquitin ligases.
Peter K. Jackson;Adam G. Eldridge;Ellen Freed;Laura Furstenthal.
Trends in Cell Biology (2000)
Sensitivity to antitubulin chemotherapeutics is regulated by MCL1 and FBW7
Ingrid E. Wertz;Saritha Kusam;Cynthia Lam;Toru Okamoto.
Separate domains of p21 involved in the inhibition of Cdk kinase and PCNA
Junjie Chen;Peter K. Jackson;Marc W. Kirschner;Anindya Dutta.
Small-molecule ligands bind to a distinct pocket in Ras and inhibit SOS-mediated nucleotide exchange activity
Till Maurer;Lindsay S. Garrenton;Angela Oh;Keith Pitts.
Proceedings of the National Academy of Sciences of the United States of America (2012)
Mapping the NPHP-JBTS-MKS Protein Network Reveals Ciliopathy Disease Genes and Pathways
Liyun Sang;Julie J. Miller;Kevin C. Corbit;Rachel H. Giles.
Emi1 Is a Mitotic Regulator that Interacts with Cdc20 and Inhibits the Anaphase Promoting Complex
Julie D.R. Reimann;Ellen Freed;Jerry Y. Hsu;Edgar R. Kramer.
The mouse type IV c-abl gene product is a nuclear protein, and activation of transforming ability is associated with cytoplasmic localization
Richard A. Van Etten;Peter Jackson;David Baltimore.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: