His primary scientific interests are in Autism, Neuroscience, Developmental psychology, Social behavior and Social identity approach. His study in Autism is interdisciplinary in nature, drawing from both Habituation, Dopamine receptor D2 and Cortex. His Neuroscience research includes elements of Neurodevelopmental disorder, Genetics, SHANK2 and Candidate gene.
His studies in Neurodevelopmental disorder integrate themes in fields like Causes of autism, Pervasive developmental disorder, Developmental disorder and Single-nucleotide polymorphism. His study focuses on the intersection of SHANK2 and fields such as Long-term potentiation with connections in the field of NMDA receptor, Glutamate receptor, Neurotransmission, Postsynaptic density and Excitatory postsynaptic potential. His Developmental psychology study incorporates themes from Stimulus, Sensory system and Sniffing.
His primary areas of study are Autism, Neuroscience, Developmental psychology, Social behavior and Social identity approach. His Autism study combines topics in areas such as Phenotype, Genetics, Corpus callosum and Immunology. He has included themes like Elevated plus maze, SHANK2 and Neurodevelopmental disorder in his Neuroscience study.
His SHANK2 study combines topics from a wide range of disciplines, such as Long-term potentiation, Glutamatergic, 22q13 deletion syndrome and SHANK3 Gene. His work in Neurodevelopmental disorder addresses issues such as Candidate gene, which are connected to fields such as Single-nucleotide polymorphism, Causes of autism, SNP and Pervasive developmental disorder. The various areas that Mu Yang examines in his Developmental psychology study include Stimulus, Inbred strain, Audiology and Odor.
Mu Yang focuses on Autism, Autism spectrum disorder, Neuroscience, Immunology and Social identity approach. His Autism study is concerned with the larger field of Developmental psychology. In the field of Developmental psychology, his study on Social cue overlaps with subjects such as Chromosomal region.
His Autism spectrum disorder research integrates issues from Pharmacological interventions and Speech delay. Mu Yang works mostly in the field of Neuroscience, limiting it down to topics relating to Phenotype and, in certain cases, Copy-number variation, Knockout rat, Clinical trial and Single gene. His work on Antigen and Epitope as part of general Immunology research is frequently linked to Autoantibody and Maternal autoantibodies, bridging the gap between disciplines.
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Behavioural phenotyping assays for mouse models of autism
Jill L. Silverman;Mu Yang;Catherine Lord;Jacqueline N. Crawley.
Nature Reviews Neuroscience (2010)
Clinical manifestations in 105 persons with nevoid basal cell carcinoma syndrome.
V. E. Kimonis;A. M. Goldstein;B. Pastakia;M. L. Yang.
American Journal of Medical Genetics (1997)
Autism-like behavioral phenotypes in BTBR T+tf/J mice
H. G. McFarlane;G. K. Kusek;M. Yang;J. L. Phoenix.
Genes, Brain and Behavior (2008)
Haploinsufficiency of the autism-associated Shank3 gene leads to deficits in synaptic function, social interaction, and social communication.
Ozlem Bozdagi;Takeshi Sakurai;Danae Papapetrou;Xiaobin Wang.
Molecular Autism (2010)
Simple behavioral assessment of mouse olfaction.
Mu Yang;Jacqueline N. Crawley.
Current protocols in protein science (2009)
Automated Three-Chambered Social Approach Task for Mice
Mu Yang;Jill L. Silverman;Jacqueline N. Crawley.
Current protocols in protein science (2011)
Reduced Excitatory Neurotransmission and Mild Autism-Relevant Phenotypes in Adolescent Shank3 Null Mutant Mice
Mu Yang;Ozlem Bozdagi;Maria Luisa Scattoni;Maria Luisa Scattoni;Markus Wöhr;Markus Wöhr.
The Journal of Neuroscience (2012)
Cue and context conditioning of defensive behaviors to cat odor stimuli
Robert J. Blanchard;Mu Yang;Chun I. Li;Alan Gervacio.
Neuroscience & Biobehavioral Reviews (2001)
Behavioral Abnormalities and Circuit Defects in the Basal Ganglia of a Mouse Model of 16p11.2 Deletion Syndrome
Thomas Portmann;Mu Yang;Rong Mao;Georgia Panagiotakos.
Cell Reports (2014)
Social approach behaviors are similar on conventional versus reverse lighting cycles, and in replications across cohorts, in BTBR T+ tf/J, C57BL/6J, and vasopressin receptor 1B mutant mice.
Mu Yang;Maria Luisa Scattoni;Vladimir Zhodzishsky;Thomas Chen.
Frontiers in Behavioral Neuroscience (2007)
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