D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 53 Citations 16,133 114 World Ranking 8349 National Ranking 585

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Enzyme

The scientist’s investigation covers issues in Molecular biology, Cell biology, Mutant, Gene targeting and Immunology. His work carried out in the field of Molecular biology brings together such families of science as Complementary DNA, Embryonic stem cell, Gene, IRF7 and Receptor complex. His biological study spans a wide range of topics, including Oxidative stress and dGTPase.

His Cell biology study incorporates themes from Genetics and Endothelium. His Mutant study combines topics in areas such as Gene expression, Mutation, Knockout mouse, Motor coordination and Genetically modified mouse. His Gene targeting study combines topics from a wide range of disciplines, such as Cytokine, DNA damage, Aryl hydrocarbon receptor, Transcription factor and Receptor.

His most cited work include:

  • Distinct and Essential Roles of Transcription Factors IRF-3 and IRF-7 in Response to Viruses for IFN-α/β Gene Induction (1163 citations)
  • STAT3 activation is sufficient to maintain an undifferentiated state of mouse embryonic stem cells (818 citations)
  • Severe osteopetrosis, defective interleukin-1 signalling and lymph node organogenesis in TRAF6-deficient mice. (562 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Molecular biology, Genetically modified mouse, Transgene, Cell biology and Gene. His study in Molecular biology is interdisciplinary in nature, drawing from both Gene expression, Carcinogenesis, Mutation, Mutant and Gene targeting. His research integrates issues of Knockout mouse and Anatomy in his study of Mutant.

His research in Genetically modified mouse intersects with topics in Endocrinology, In situ hybridization, Somatic cell, Internal medicine and Tyrosine hydroxylase. Motoya Katsuki has included themes like Embryonic stem cell, CYP3A7, Pathology, Myelin basic protein and In vivo in his Transgene study. His studies in Cell biology integrate themes in fields like Genetics and Metabotropic glutamate receptor.

He most often published in these fields:

  • Molecular biology (45.56%)
  • Genetically modified mouse (39.64%)
  • Transgene (33.14%)

What were the highlights of his more recent work (between 2001-2013)?

  • Cell biology (23.67%)
  • Neuroscience (6.51%)
  • Internal medicine (18.93%)

In recent papers he was focusing on the following fields of study:

Motoya Katsuki spends much of his time researching Cell biology, Neuroscience, Internal medicine, Endocrinology and Carcinogenesis. His study on Transducin is often connected to Vasculogenesis as part of broader study in Cell biology. His Internal medicine research is multidisciplinary, relying on both Megakaryocyte and Oncostatin M receptor.

The concepts of his Endocrinology study are interwoven with issues in Haematopoiesis, Hematopoietic Tissue, Stem cell and Epidermal growth factor receptor. His Carcinogenesis study combines topics in areas such as Molecular biology, Cancer research and Mutant. He combines subjects such as Genetically modified mouse, Transgene, In situ hybridization, Gene targeting and Null allele with his study of Knockout mouse.

Between 2001 and 2013, his most popular works were:

  • Loss of teratogenic response to 2,3,7,8-tetrachlorodibenzo- p-dioxin (TCDD) in mice lacking the Ah (dioxin) receptor (561 citations)
  • Crosstalk between neovessels and mural cells directs the site-specific expression of MT1-MMP to endothelial tip cells. (147 citations)
  • Targeted disruption of oncostatin M receptor results in altered hematopoiesis. (130 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Enzyme

Cell biology, Internal medicine, Endocrinology, Signal transduction and Beta-catenin are his primary areas of study. Motoya Katsuki has researched Cell biology in several fields, including Endothelium and Angiogenesis. His Internal medicine research is multidisciplinary, incorporating elements of Gene targeting, Embryo and Aryl hydrocarbon receptor repressor.

The study incorporates disciplines such as Megakaryocyte, Hematopoietic Tissue, Haematopoiesis, Stem cell and Oncostatin M receptor in addition to Endocrinology. Motoya Katsuki works mostly in the field of Signal transduction, limiting it down to topics relating to Fetal protein and, in certain cases, Molecular biology, as a part of the same area of interest. His Molecular biology research incorporates elements of Cadherin, Mutagenesis, Biochemistry, Nucleotide and Transversion.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Distinct and Essential Roles of Transcription Factors IRF-3 and IRF-7 in Response to Viruses for IFN-α/β Gene Induction

Mitsuharu Sato;Hirofumi Suemori;Naoki Hata;Masataka Asagiri.
Immunity (2000)

1884 Citations

STAT3 activation is sufficient to maintain an undifferentiated state of mouse embryonic stem cells

Takahiko Matsuda;Takanori Nakamura;Kazuki Nakao;Takao Arai.
The EMBO Journal (1999)

1281 Citations

Cardiac fibrosis in mice lacking brain natriuretic peptide

Naohisa Tamura;Yoshihiro Ogawa;Hideki Chusho;Kenji Nakamura.
Proceedings of the National Academy of Sciences of the United States of America (2000)

763 Citations

High incidence of spontaneous autoimmune encephalomyelitis in immunodeficient anti-myelin basic protein T cell receptor transgenic mice

Juan J. Lafaille;Kumiko Nagashima;Motoya Katsuki;Susumu Tonegawa.
Cell (1994)

759 Citations

Loss of teratogenic response to 2,3,7,8-tetrachlorodibenzo- p-dioxin (TCDD) in mice lacking the Ah (dioxin) receptor

Junsei Mimura;Keisuke Yamashita;Kenji Nakamura;Masanobu Morita.
Genes to Cells (2003)

740 Citations

Severe osteopetrosis, defective interleukin-1 signalling and lymph node organogenesis in TRAF6-deficient mice.

Asuka Naito;Sakura Azuma;Sakae Tanaka;Tsuyoshi Miyazaki.
Genes to Cells (1999)

713 Citations

Specific deficit of the ON response in visual transmission by targeted disruption of the mGIuR6 gene

Masayuki Masu;Hideki Iwakabe;Yoshiaki Tagawa;Tomomitsu Miyoshi.
Cell (1995)

645 Citations

Dwarfism and early death in mice lacking C-type natriuretic peptide.

Hideki Chusho;Naohisa Tamura;Yoshihiro Ogawa;Akihiro Yasoda.
Proceedings of the National Academy of Sciences of the United States of America (2001)

567 Citations

Murine homolog of SALL1 is essential for ureteric bud invasion in kidney development.

Ryuichi Nishinakamura;Yuko Matsumoto;Kazuki Nakao;Kenji Nakamura.
Development (2001)

454 Citations

K-Ras is essential for the development of the mouse embryo

Keiko Koera;Kenji Nakamura;Kazuki Nakao;Jun Miyoshi.
Oncogene (1997)

412 Citations

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