What is he best known for?
The fields of study he is best known for:
Cell biology, Molecular biology, Embryonic stem cell, Gene targeting and Internal medicine are his primary areas of study.
The study incorporates disciplines such as Genetics, Cell adhesion, Immunology and Mesoderm in addition to Cell biology.
His work carried out in the field of Molecular biology brings together such families of science as IRF7, Sp3 transcription factor and Mutation, Gene, DNA repair.
The Gene study combines topics in areas such as Oxidative stress, dGTPase and DNA damage.
His work deals with themes such as Cancer research, Inner cell mass, Cellular differentiation, Trophoblast and Signal transduction, which intersect with Embryonic stem cell.
His work is dedicated to discovering how Internal medicine, Endocrinology are connected with Myocardial infarction, Heart failure and Brain natriuretic peptide and other disciplines.
His most cited work include:
- Distinct and Essential Roles of Transcription Factors IRF-3 and IRF-7 in Response to Viruses for IFN-α/β Gene Induction (1163 citations)
- STAT3 activation is sufficient to maintain an undifferentiated state of mouse embryonic stem cells (818 citations)
- Targeted disruption of the Rad51 gene leads to lethality in embryonic mice. (713 citations)
What are the main themes of his work throughout his whole career to date?
Kazuki Nakao mostly deals with Cell biology, Molecular biology, Gene, Neuroscience and Embryonic stem cell.
His work in Cell biology addresses issues such as Metabotropic glutamate receptor, which are connected to fields such as Neurotransmission.
His research in Molecular biology intersects with topics in Cell culture, Transgene, Mutation, Carcinogenesis and Gene targeting.
His Gene research incorporates elements of DNA damage and DNA.
His Neuroscience research includes themes of Synaptic plasticity, Metabotropic glutamate receptor 1 and Knockout mouse.
His Embryonic stem cell research is multidisciplinary, incorporating elements of Stem cell and Cellular differentiation.
He most often published in these fields:
- Cell biology (43.42%)
- Molecular biology (24.34%)
- Gene (17.11%)
What were the highlights of his more recent work (between 2015-2021)?
- Cell biology (43.42%)
- Embryo (9.21%)
- Mutant (13.82%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Cell biology, Embryo, Mutant, Gene and Internal medicine.
His Cell biology research is multidisciplinary, incorporating perspectives in Hormone, Muscle hypertrophy and Programmed cell death.
His research on Embryo also deals with topics like
- Cell together with Endoderm,
- Electroporation which is related to area like Cas9.
His Mutant research integrates issues from Phenotype, Prepulse inhibition, Excitatory synapse and Circadian rhythm.
His study in the field of SGK1 and Transporter also crosses realms of Genome-wide association study and Hyperuricemia.
As part of the same scientific family, Kazuki Nakao usually focuses on Internal medicine, concentrating on Endocrinology and intersecting with Downregulation and upregulation and Protein kinase B.
Between 2015 and 2021, his most popular works were:
- Apolipoprotein M Protects Lipopolysaccharide-Treated Mice from Death and Organ Injury. (26 citations)
- A possible aid in targeted insertion of large DNA elements by CRISPR/Cas in mouse zygotes. (24 citations)
- Dephosphorylated parafibromin is a transcriptional coactivator of the Wnt/Hedgehog/Notch pathways. (22 citations)
In his most recent research, the most cited papers focused on:
His scientific interests lie mostly in APOM, Internal medicine, Endocrinology, Gene and Sepsis.
There are a combination of areas like Type 2 diabetes, Insulin resistance, Downregulation and upregulation, Diabetes mellitus and Insulin receptor integrated together with his APOM study.
Internal medicine is often connected to Protein kinase B in his work.
Kazuki Nakao works on Gene which deals in particular with Locus.
Kazuki Nakao has included themes like Disseminated intravascular coagulation, Lipopolysaccharide, Kidney and Plasminogen activator in his Sepsis study.
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