His primary areas of study are Biochemistry, Reactive oxygen species, Cell biology, Myeloperoxidase and Hydrogen peroxide. As part of his studies on Biochemistry, he often connects relevant areas like Peroxide. His studies in Reactive oxygen species integrate themes in fields like Apoptosis and Caspase, Intrinsic apoptosis.
His biological study spans a wide range of topics, including Necrosis and Jurkat cells. His studies examine the connections between Cell biology and genetics, as well as such issues in Programmed cell death, with regards to Neutrophil clearance. His Myeloperoxidase research integrates issues from Phagosome, Phagocytosis, Microbiology, Superoxide and Hypochlorous acid.
Mark B. Hampton mainly investigates Biochemistry, Cell biology, Apoptosis, Peroxiredoxin and Hydrogen peroxide. As a member of one scientific family, Mark B. Hampton mostly works in the field of Biochemistry, focusing on Redox and, on occasion, Dithiothreitol. His Cell biology study integrates concerns from other disciplines, such as Thioredoxin and Programmed cell death.
The Caspase research Mark B. Hampton does as part of his general Apoptosis study is frequently linked to other disciplines of science, such as Phosphatidylserine, therefore creating a link between diverse domains of science. His Peroxiredoxin research includes themes of Thioredoxin reductase, Peroxide and Iodoacetamide. His biological study deals with issues like Hypochlorous acid, which deal with fields such as Chloramine.
His primary scientific interests are in Hypochlorous acid, Cell biology, Myeloperoxidase, Microbiology and Oxidative stress. His Cell biology research focuses on Mitochondrion in particular. His research in Myeloperoxidase focuses on subjects like Superoxide, which are connected to Glutathione disulfide, Azurophilic granule, Apoptosis, Hypoxia and Autophagy.
Phagosome and Phagocytosis are the subjects of his Microbiology studies. His study looks at the relationship between Oxidative stress and fields such as Immune system, as well as how they intersect with chemical problems. Mark B. Hampton has researched Respiratory burst in several fields, including Peroxiredoxin, Antioxidant, Reactive oxygen species and Thioredoxin reductase, Thioredoxin.
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Inside the Neutrophil Phagosome: Oxidants, Myeloperoxidase, and Bacterial Killing
Mark B. Hampton;Anthony J. Kettle;Christine C. Winterbourn.
Blood (1998)
Thiol chemistry and specificity in redox signaling.
Christine C. Winterbourn;Mark B. Hampton.
Free Radical Biology and Medicine (2008)
Dual regulation of caspase activity by hydrogen peroxide: implications for apoptosis
Mark B Hampton;Sten Orrenius.
FEBS Letters (1997)
Modeling the Reactions of Superoxide and Myeloperoxidase in the Neutrophil Phagosome: IMPLICATIONS FOR MICROBIAL KILLING*
Christine C. Winterbourn;Mark B. Hampton;John H Livesey;Anthony J. Kettle.
Journal of Biological Chemistry (2006)
Mitochondrial peroxiredoxin involvement in antioxidant defence and redox signalling.
Andrew G. Cox;Christine C. Winterbourn;Mark B. Hampton.
Biochemical Journal (2010)
Involvement of Caspases in Neutrophil Apoptosis: Regulation by Reactive Oxygen Species
Bengt Fadeel;Anders Åhlin;Anders Åhlin;Jan-Inge Henter;Jan-Inge Henter;Sten Orrenius;Sten Orrenius.
Blood (1998)
The High Reactivity of Peroxiredoxin 2 with H2O2 Is Not Reflected in Its Reaction with Other Oxidants and Thiol Reagents
Alexander V. Peskin;Felicia M. Low;Louise N. Paton;Ghassan J. Maghzal.
Journal of Biological Chemistry (2007)
Reactive Oxygen Species and Neutrophil Function
Christine C Winterbourn;Anthony J Kettle;Mark B Hampton.
Annual Review of Biochemistry (2016)
Redox Regulation of the Caspases during Apoptosisa
Mark B. Hampton;Bengt Fadeel;Sten Orrenius.
Annals of the New York Academy of Sciences (1998)
Requirements for NADPH oxidase and myeloperoxidase in neutrophil extracellular trap formation differ depending on the stimulus
Heather Parker;Mike Dragunow;Mark B. Hampton;Anthony J. Kettle.
Journal of Leukocyte Biology (2012)
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