What is he best known for?
The fields of study he is best known for:
His primary areas of investigation include Genetics, Alzheimer's disease, Apolipoprotein E, Genome-wide association study and Allele.
Many of his studies involve connections with topics such as Early-onset Alzheimer's disease and Genetics.
His Alzheimer's disease research is multidisciplinary, incorporating elements of Biological pathway, Linkage disequilibrium and Allele frequency.
María J. Bullido interconnects Odds ratio, Molecular biology and Genetic association in the investigation of issues within Apolipoprotein E.
The concepts of his Genome-wide association study study are interwoven with issues in Gene expression, Case-control study, Neurodegeneration and Genomics.
His Single-nucleotide polymorphism research includes elements of Disease susceptibility and Age of onset.
His most cited work include:
- Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease (2614 citations)
- Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease (2614 citations)
- Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease (1387 citations)
What are the main themes of his work throughout his whole career to date?
María J. Bullido mainly investigates Genetics, Disease, Apolipoprotein E, Alzheimer's disease and Allele.
Genetics is represented through his Single-nucleotide polymorphism, Genotype, Genome-wide association study, Allele frequency and Genetic association research.
His studies in Genome-wide association study integrate themes in fields like Case-control study and Genetic predisposition.
His Genetic association research is multidisciplinary, relying on both Odds ratio, PICALM and Age of onset.
His Apolipoprotein E research incorporates themes from Immunology and Polymorphism.
His Alzheimer's disease study integrates concerns from other disciplines, such as Endocrinology, Pathogenesis, Late onset, Exon and Degenerative disease.
He most often published in these fields:
- Genetics (58.18%)
- Disease (44.55%)
- Apolipoprotein E (30.91%)
What were the highlights of his more recent work (between 2016-2021)?
- Disease (44.55%)
- Genetics (58.18%)
- Genome-wide association study (21.82%)
In recent papers he was focusing on the following fields of study:
María J. Bullido mostly deals with Disease, Genetics, Genome-wide association study, Dementia and Cell biology.
His work deals with themes such as Psychiatry and Genetic association, which intersect with Disease.
Genetics is closely attributed to Inbreeding in his work.
His Genome-wide association study study combines topics from a wide range of disciplines, such as Alzheimer's disease and Allele frequency.
In his study, which falls under the umbrella issue of Dementia, Bioinformatics, Amyloid and Computational biology is strongly linked to Gene.
His biological study spans a wide range of topics, including Oxidative stress and Neurodegeneration.
Between 2016 and 2021, his most popular works were:
- Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease (429 citations)
- Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease (429 citations)
- Genome-wide association analysis of dementia and its clinical endophenotypes reveal novel loci associated with Alzheimer's disease and three causality networks: The GR@ACE project (29 citations)
In his most recent research, the most cited papers focused on:
His primary areas of study are Disease, Genome-wide association study, Dementia, Gene and Genetic association.
His Genome-wide association study research incorporates elements of Alzheimer's disease and Allele frequency.
The study incorporates disciplines such as Common disease-common variant, Genetic predisposition, Linkage disequilibrium, Case-control study and Minor allele frequency in addition to Alzheimer's disease.
In general Dementia study, his work on Cerebral amyloid angiopathy often relates to the realm of Mechanism, Endophenotype and Causality, thereby connecting several areas of interest.
The subject of his Genetic association research is within the realm of Single-nucleotide polymorphism.
This overview was generated by a machine learning system which analysed the scientist’s
body of work. If you have any feedback, you can
contact us
here.
