What is he best known for?
The fields of study he is best known for:
Marc Piechaczyk spends much of his time researching Molecular biology, Biochemistry, Cell biology, Messenger RNA and Transcription.
His Molecular biology research includes elements of Epidermal growth factor, Mrna level, Cell cycle, Gene and Epitope.
His Biochemistry study frequently draws connections to other fields, such as Jurkat cells.
His biological study spans a wide range of topics, including SUMO protein and Oncogene.
Marc Piechaczyk is interested in Glyceraldehyde 3-phosphate dehydrogenase, which is a field of Messenger RNA.
His Glyceraldehyde 3-phosphate dehydrogenase research integrates issues from RNA, Primer extension, Nucleotide, DNA and Peptide sequence.
His most cited work include:
- Various rat adult tissues express only one major mRNA species from the glyceraldehyde-3-phosphate-dehydrogenase multigenic family (1851 citations)
- Post-transcriptional regulation of glyceraldehyde-3-phosphate-dehydrogenase gene expression in rat tissues (389 citations)
- Extreme instability of myc mRNA in normal and transformed human cells. (344 citations)
What are the main themes of his work throughout his whole career to date?
Marc Piechaczyk mainly investigates Molecular biology, Biochemistry, Cell biology, Antibody and Virology.
The study incorporates disciplines such as Gene expression, RNA, Messenger RNA, Gene and Transcription in addition to Molecular biology.
His work in Messenger RNA addresses subjects such as Regulation of gene expression, which are connected to disciplines such as Protein biosynthesis.
His work on Proteasome as part of general Cell biology study is frequently connected to Degradation, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
The Antibody study combines topics in areas such as Genetic enhancement and In vivo.
His Virology research includes themes of Major histocompatibility complex and Recombinant DNA.
He most often published in these fields:
- Molecular biology (39.29%)
- Biochemistry (24.11%)
- Cell biology (23.21%)
What were the highlights of his more recent work (between 2001-2014)?
- Cell biology (23.21%)
- Proteasome (15.18%)
- Virology (16.96%)
In recent papers he was focusing on the following fields of study:
His primary areas of study are Cell biology, Proteasome, Virology, Molecular biology and Transcription factor.
His Cell biology research incorporates themes from SUMO protein, Biochemistry and Cytosol.
The concepts of his Proteasome study are interwoven with issues in Protein subunit, Ubiquitin, Protein degradation and Proteolysis.
His studies in Virology integrate themes in fields like Immune system, Antibody, Immunology and G protein.
Marc Piechaczyk specializes in Molecular biology, namely Flow cytometry.
His Transcription factor research is multidisciplinary, incorporating elements of Cytoplasm and Kinase.
Between 2001 and 2014, his most popular works were:
- CNF1 exploits the ubiquitin-proteasome machinery to restrict Rho GTPase activation for bacterial host cell invasion. (220 citations)
- The HBZ Factor of Human T-cell Leukemia Virus Type I Dimerizes with Transcription Factors JunB and c-Jun and Modulates Their Transcriptional Activity (177 citations)
- Ubiquitin-independent degradation of proteins by the proteasome. (163 citations)
In his most recent research, the most cited papers focused on:
Cell biology, Proteasome, Biochemistry, Transcription factor and Ubiquitin are his primary areas of study.
His work carried out in the field of Cell biology brings together such families of science as Protein subunit, Proteolysis, RNA splicing, SR protein and SUMO protein.
Marc Piechaczyk interconnects Kinase and Mutant in the investigation of issues within Transcription factor.
His Ubiquitin research is multidisciplinary, relying on both Cell cycle, Oncogene, Cell division, MHC class I and Hypoxia-Inducible Factor 1.
His research in c-jun intersects with topics in Molecular biology and Transactivation.
Marc Piechaczyk integrates Molecular biology and Clostridium difficile toxin A in his studies.
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