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Microbiology

D-Index
54
Citations
9168
World Ranking
3997
National Ranking
1553

Overview

Mahtab Moayeri is affiliated with the National Institutes of Health in the United States. Their research primarily falls within the field of Biochemistry, Genetics and Molecular Biology, with a notable emphasis on Molecular Biology, Genetics, Immunology, Ecology, and Biotechnology as subfields.

The scientist's main research topics include:

  • Bacillus and Francisella bacterial research
  • Bacterial Genetics and Biotechnology
  • Bacteriophages and microbial interactions
  • Microbial Inactivation Methods
  • Microbial infections and disease research
  • Herpesvirus Infections and Treatments
  • Genetically Modified Organisms Research

Among their recent scholarly contributions are the following papers:

  • Mycobacterium tuberculosis Induces Irg1 in Murine Macrophages by a Pathway Involving Both TLR-2 and STING/IFNAR Signaling and Requiring Bacterial Phagocytosis, 2022, Frontiers in Cellular and Infection Microbiology
  • Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons, 2021, Nature Neuroscience
  • Frontline Science: Anthrax lethal toxin-induced, NLRP1-mediated IL-1β release is a neutrophil and PAD4-dependent event, 2020, Journal of Leukocyte Biology
  • Anthrax lethal factor cleaves regulatory subunits of phosphoinositide-3 kinase to contribute to toxin lethality, 2020, Nature Microbiology
  • S9.6-based hybrid capture immunoassay for pathogen detection, 2023, Scientific Reports

Moayeri has frequently published in several scientific journals, with the most common venues including:

  • bioRxiv (Cold Spring Harbor Laboratory)
  • Journal of Biological Chemistry
  • Frontiers in Cellular and Infection Microbiology
  • Nature Neuroscience
  • Nature Microbiology

Collaboration features prominently in their research. Frequent coauthors include Stephen H. Leppla, Rasem Fattah, Ankur Bothra, Ma Qian, and Danielle O'Mard.

Best Publications

  • Rapid induction of inflammatory lipid mediators by the inflammasome in vivo

    Jakob von Moltke;Norver J. Trinidad;Mahtab Moayeri;Alexander F. Kintzer

  • Bacillus anthracis lethal toxin induces TNF-α–independent hypoxia-mediated toxicity in mice

    Mahtab Moayeri;Diana Haines;Howard A. Young;Stephen H. Leppla

  • The roles of anthrax toxin in pathogenesis.

    Mahtab Moayeri;Stephen H Leppla

  • Anthrax lethal factor cleavage of Nlrp1 is required for activation of the inflammasome.

    Jonathan L. Levinsohn;Zachary L. Newman;Kristina A. Hellmich;Rasem Fattah

  • Cellular and systemic effects of anthrax lethal toxin and edema toxin.

    Mahtab Moayeri;Stephen H. Leppla

  • Plant-Based Vaccine: Mice Immunized with Chloroplast-Derived Anthrax Protective Antigen Survive Anthrax Lethal Toxin Challenge

    Vijay Koya;Mahtab Moayeri;Stephen H. Leppla;Henry Daniell

  • Dual Role for Inflammasome Sensors NLRP1 and NLRP3 in Murine Resistance to Toxoplasma gondii

    Gezahegn Gorfu;Kimberly Cirelli;Mariane Bandeira Melo;Katrin Mayer-Barber

  • Anthrax lethal and edema toxins in anthrax pathogenesis

    Shihui Liu;Mahtab Moayeri;Stephen H. Leppla

  • Hijacking Multivesicular Bodies Enables Long-Term and Exosome-Mediated Long-Distance Action of Anthrax Toxin

    Laurence Abrami;Lucia Brandi;Mahtab Moayeri;Michael J. Brown

  • Bacillus anthracis Edema Toxin Causes Extensive Tissue Lesions and Rapid Lethality in Mice

    Aaron M. Firoved;Georgina F. Miller;Mahtab Moayeri;Rahul Kakkar

  • Capillary morphogenesis protein-2 is the major receptor mediating lethality of anthrax toxin in vivo.

    Shihui Liu;Devorah Crown;Sharmina Miller-Randolph;Mahtab Moayeri

  • Lethality during continuous anthrax lethal toxin infusion is associated with circulatory shock but not inflammatory cytokine or nitric oxide release in rats.

    Xizhong Cui;Mahtab Moayeri;Yan Li;Xuemei Li

  • Sulforaphane inhibits multiple inflammasomes through an Nrf2-independent mechanism

    Allison J. Greaney;Nolan K. Maier;Stephen H. Leppla;Mahtab Moayeri

  • Anthrax lethal toxin‐induced inflammasome formation and caspase‐1 activation are late events dependent on ion fluxes and the proteasome

    Katherine E. Wickliffe;Stephen H. Leppla;Mahtab Moayeri

  • Inflammasome Sensor NLRP1 Controls Rat Macrophage Susceptibility to Toxoplasma gondii

    Kimberly M. Cirelli;Gezahegn Gorfu;Musa A. Hassan;Morton Printz

  • Detection of anthrax toxin by an ultrasensitive immunoassay using europium nanoparticles.

    Shixing Tang;Mahtab Moayeri;Zhaochun Chen;Harri Harma

  • Key tissue targets responsible for anthrax-toxin-induced lethality

    Shihui Liu;Yi Zhang;Mahtab Moayeri;Jie Liu

  • Blocking anthrax lethal toxin at the protective antigen channel by using structure-inspired drug design

    Vladimir A. Karginov;Ekaterina M. Nestorovich;Mahtab Moayeri;Stephen H. Leppla

  • Inflammasome sensor Nlrp1b-dependent resistance to anthrax is mediated by caspase-1, IL-1 signaling and neutrophil recruitment.

    Mahtab Moayeri;Devorah Crown;Zachary L. Newman;Shu Okugawa

  • Anthrax Lethal Factor Cleaves Mouse Nlrp1b in Both Toxin-Sensitive and Toxin-Resistant Macrophages

    Kristina A. Hellmich;Jonathan L. Levinsohn;Rasem Fattah;Zachary L. Newman

Frequent Co-Authors

Stephen H. Leppla
Stephen H. Leppla National Institutes of Health
Robert H. Purcell
Robert H. Purcell National Institute of Allergy and Infectious Diseases
Rodney A. Welch
Rodney A. Welch University of Wisconsin–Madison
Charles B. Shoemaker
Charles B. Shoemaker Tufts University
Seth L. Masters
Seth L. Masters Hudson Institute of Medical Research
Michael E. Grigg
Michael E. Grigg National Institutes of Health
Thomas H. Bugge
Thomas H. Bugge National Institutes of Health
Michael Eckhaus
Michael Eckhaus National Institutes of Health
Bruno B. Andrade
Bruno B. Andrade Oswaldo Cruz Foundation
Jeroen P. J. Saeij
Jeroen P. J. Saeij University of California, Davis

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