H-Index & Metrics Best Publications

H-Index & Metrics

Discipline name H-index Citations Publications World Ranking National Ranking
Immunology D-index 80 Citations 25,117 275 World Ranking 651 National Ranking 2
Medicine D-index 82 Citations 26,089 282 World Ranking 8598 National Ranking 39

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Enzyme

His primary areas of investigation include Immunology, Gluten, Epitope, Biochemistry and Gliadin. His studies deal with areas such as Coeliac disease, Disease and Intestinal mucosa as well as Immunology. His studies in Gluten integrate themes in fields like Proteases, Innate immune system, MHC restriction and Interferon gamma.

His study with Epitope involves better knowledge in Antigen. The study incorporates disciplines such as T cell, Deamidation and HLA-DQ Antigen in addition to Gliadin. His Human leukocyte antigen research includes elements of Major histocompatibility complex and Allele.

His most cited work include:

  • Structural Basis for Gluten Intolerance in Celiac Sprue (1232 citations)
  • Tissue transglutaminase selectively modifies gliadin peptides that are recognized by gut-derived T cells in celiac disease (953 citations)
  • Coeliac disease: dissecting a complex inflammatory disorder (802 citations)

What are the main themes of his work throughout his whole career to date?

Ludvig M. Sollid mainly focuses on Immunology, Epitope, Gluten, Antigen and Human leukocyte antigen. His Immunology research is multidisciplinary, incorporating elements of Coeliac disease and Disease. His Coeliac disease study combines topics in areas such as HLA-DQ and Pathogenesis.

Ludvig M. Sollid has researched Epitope in several fields, including T cell, Biochemistry, Tissue transglutaminase, Molecular biology and Gliadin. Ludvig M. Sollid combines subjects such as Inflammation, Lamina propria and Antigen presentation with his study of Antigen. Ludvig M. Sollid has included themes like Allele and Major histocompatibility complex in his Human leukocyte antigen study.

He most often published in these fields:

  • Immunology (63.99%)
  • Epitope (36.50%)
  • Gluten (32.12%)

What were the highlights of his more recent work (between 2017-2021)?

  • Immunology (63.99%)
  • T-cell receptor (17.27%)
  • Epitope (36.50%)

In recent papers he was focusing on the following fields of study:

Immunology, T-cell receptor, Epitope, T cell and Gluten are his primary areas of study. Ludvig M. Sollid frequently studies issues relating to Disease and Immunology. His research in Epitope intersects with topics in HLA-DQ2, Human leukocyte antigen, Major histocompatibility complex, Immune system and Antigen presentation.

His T cell research integrates issues from Acquired immune system, Biopsy, B cell and Cell biology. The various areas that Ludvig M. Sollid examines in his Gluten study include Proteome, Inflammation, Coeliac disease, Quantitative proteomics and Ingestion. His Antibody study integrates concerns from other disciplines, such as Tissue transglutaminase and Molecular biology.

Between 2017 and 2021, his most popular works were:

  • The SysteMHC Atlas project. (75 citations)
  • Disease-driving CD4+ T cell clonotypes persist for decades in celiac disease (50 citations)
  • Distinct phenotype of CD4 + T cells driving celiac disease identified in multiple autoimmune conditions (46 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Immune system

Ludvig M. Sollid mainly investigates Immunology, T cell, Antigen, Autoimmunity and Gluten. His Immunology study frequently draws connections to other fields, such as Disease. Many of his studies involve connections with topics such as Human leukocyte antigen and T cell.

His Antigen research is multidisciplinary, incorporating perspectives in Phenotype, Lupus erythematosus, B-cell receptor and Scleroderma. His Gluten research includes themes of Ingestion, Coeliac disease, Biopsy and Cytokine. His work deals with themes such as Gliadin and Pathogenesis, which intersect with Genetics.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Structural Basis for Gluten Intolerance in Celiac Sprue

Lu Shan;Øyvind Molberg;Isabelle Parrot;Felix Hausch.
Science (2002)

1782 Citations

Tissue transglutaminase selectively modifies gliadin peptides that are recognized by gut-derived T cells in celiac disease

Øyvind Molberg;Stephen N. Mcadam;Roman Körner;Hanne Quarsten.
Nature Medicine (1998)

1281 Citations

Coeliac disease: dissecting a complex inflammatory disorder

Ludvig M. Sollid.
Nature Reviews Immunology (2002)

1188 Citations

Evidence for a primary association of celiac disease to a particular HLA-DQ alpha/beta heterodimer.

L M Sollid;G Markussen;J Ek;H Gjerde.
Journal of Experimental Medicine (1989)

1116 Citations

Molecular Basis of Celiac Disease

Ludvig M. Sollid.
Annual Review of Immunology (2000)

810 Citations

The intestinal T cell response to alpha-gliadin in adult celiac disease is focused on a single deamidated glutamine targeted by tissue transglutaminase.

Helene Arentz-Hansen;Roman Körner;Øyvind Molberg;Hanne Quarsten.
Journal of Experimental Medicine (2000)

760 Citations

Immunobiology and immunopathology of human gut mucosa: Humoral immunity and intraepithelial lymphocytes

P. Brandtzaeg;T.S. Halstensen;K. Kett;P. Krajči.
Gastroenterology (1989)

739 Citations

Hla types in celiac disease patients not carrying the DQA1*05-DQB1*02 (DQ2) heterodimer: results from the european genetics cluster on celiac disease

Kati Karell;Andrew S Louka;Simon J Moodie;Henry Ascher.
Human Immunology (2003)

726 Citations

HLA susceptibility genes in celiac disease: Genetic mapping and role in pathogenesis

Ludvig M. Sollid;Erik Thorsby.
Gastroenterology (1993)

724 Citations

Gliadin-specific, HLA-DQ(alpha 1*0501,beta 1*0201) restricted T cells isolated from the small intestinal mucosa of celiac disease patients.

K. E. A. Lundin;H. Scott;T. Hansen;G. Paulsen.
Journal of Experimental Medicine (1993)

661 Citations

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