His primary areas of investigation include Immunology, Gluten, Epitope, Biochemistry and Gliadin. His studies deal with areas such as Coeliac disease, Disease and Intestinal mucosa as well as Immunology. His studies in Gluten integrate themes in fields like Proteases, Innate immune system, MHC restriction and Interferon gamma.
His study with Epitope involves better knowledge in Antigen. The study incorporates disciplines such as T cell, Deamidation and HLA-DQ Antigen in addition to Gliadin. His Human leukocyte antigen research includes elements of Major histocompatibility complex and Allele.
Ludvig M. Sollid mainly focuses on Immunology, Epitope, Gluten, Antigen and Human leukocyte antigen. His Immunology research is multidisciplinary, incorporating elements of Coeliac disease and Disease. His Coeliac disease study combines topics in areas such as HLA-DQ and Pathogenesis.
Ludvig M. Sollid has researched Epitope in several fields, including T cell, Biochemistry, Tissue transglutaminase, Molecular biology and Gliadin. Ludvig M. Sollid combines subjects such as Inflammation, Lamina propria and Antigen presentation with his study of Antigen. Ludvig M. Sollid has included themes like Allele and Major histocompatibility complex in his Human leukocyte antigen study.
Immunology, T-cell receptor, Epitope, T cell and Gluten are his primary areas of study. Ludvig M. Sollid frequently studies issues relating to Disease and Immunology. His research in Epitope intersects with topics in HLA-DQ2, Human leukocyte antigen, Major histocompatibility complex, Immune system and Antigen presentation.
His T cell research integrates issues from Acquired immune system, Biopsy, B cell and Cell biology. The various areas that Ludvig M. Sollid examines in his Gluten study include Proteome, Inflammation, Coeliac disease, Quantitative proteomics and Ingestion. His Antibody study integrates concerns from other disciplines, such as Tissue transglutaminase and Molecular biology.
Ludvig M. Sollid mainly investigates Immunology, T cell, Antigen, Autoimmunity and Gluten. His Immunology study frequently draws connections to other fields, such as Disease. Many of his studies involve connections with topics such as Human leukocyte antigen and T cell.
His Antigen research is multidisciplinary, incorporating perspectives in Phenotype, Lupus erythematosus, B-cell receptor and Scleroderma. His Gluten research includes themes of Ingestion, Coeliac disease, Biopsy and Cytokine. His work deals with themes such as Gliadin and Pathogenesis, which intersect with Genetics.
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Structural Basis for Gluten Intolerance in Celiac Sprue
Lu Shan;Øyvind Molberg;Isabelle Parrot;Felix Hausch.
Tissue transglutaminase selectively modifies gliadin peptides that are recognized by gut-derived T cells in celiac disease
Øyvind Molberg;Stephen N. Mcadam;Roman Körner;Hanne Quarsten.
Nature Medicine (1998)
Coeliac disease: dissecting a complex inflammatory disorder
Ludvig M. Sollid.
Nature Reviews Immunology (2002)
Evidence for a primary association of celiac disease to a particular HLA-DQ alpha/beta heterodimer.
L M Sollid;G Markussen;J Ek;H Gjerde.
Journal of Experimental Medicine (1989)
Molecular Basis of Celiac Disease
Ludvig M. Sollid.
Annual Review of Immunology (2000)
The intestinal T cell response to alpha-gliadin in adult celiac disease is focused on a single deamidated glutamine targeted by tissue transglutaminase.
Helene Arentz-Hansen;Roman Körner;Øyvind Molberg;Hanne Quarsten.
Journal of Experimental Medicine (2000)
Immunobiology and immunopathology of human gut mucosa: Humoral immunity and intraepithelial lymphocytes
P. Brandtzaeg;T.S. Halstensen;K. Kett;P. Krajči.
Hla types in celiac disease patients not carrying the DQA1*05-DQB1*02 (DQ2) heterodimer: results from the european genetics cluster on celiac disease
Kati Karell;Andrew S Louka;Simon J Moodie;Henry Ascher.
Human Immunology (2003)
HLA susceptibility genes in celiac disease: Genetic mapping and role in pathogenesis
Ludvig M. Sollid;Erik Thorsby.
Gliadin-specific, HLA-DQ(alpha 1*0501,beta 1*0201) restricted T cells isolated from the small intestinal mucosa of celiac disease patients.
K. E. A. Lundin;H. Scott;T. Hansen;G. Paulsen.
Journal of Experimental Medicine (1993)
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