What is he best known for?
The fields of study he is best known for:
Kunio Nagashima mainly investigates Cell biology, Molecular biology, Virus, Mutant and Biochemistry.
His research on Cell biology focuses in particular on Endosome.
His Molecular biology study incorporates themes from Lamin, Cytoplasm, Cell nucleus, Multipolar spindles and Gene isoform.
His study on Virus is covered under Virology.
His Virology research incorporates themes from Glycome, Galectin-1, Immune system and Glycoprotein.
His Mutant research integrates issues from WW domain, Peptide sequence, Ubiquitin and Nedd4 Ubiquitin Protein Ligases.
His most cited work include:
- Loss of a-Type Lamin Expression Compromises Nuclear Envelope Integrity Leading to Muscular Dystrophy (1007 citations)
- Phosphatidylinositol (4,5) bisphosphate regulates HIV-1 Gag targeting to the plasma membrane. (405 citations)
- Proteomic and Biochemical Analysis of Purified Human Immunodeficiency Virus Type 1 Produced from Infected Monocyte-Derived Macrophages (365 citations)
What are the main themes of his work throughout his whole career to date?
Cell biology, Molecular biology, Virology, Virus and Mutant are his primary areas of study.
His study in Cell biology is interdisciplinary in nature, drawing from both Biochemistry and Cell membrane.
His work investigates the relationship between Molecular biology and topics such as Capsid that intersect with problems in Biophysics.
In general Virology, his work in Viral replication and Virus Release is often linked to TSG101 linking many areas of study.
His study in Retrovirus and Equine infectious anemia falls within the category of Virus.
His Mutant research is multidisciplinary, incorporating perspectives in Mutation, Plasma protein binding and Peptide sequence.
He most often published in these fields:
- Cell biology (35.05%)
- Molecular biology (32.47%)
- Virology (22.68%)
What were the highlights of his more recent work (between 2013-2021)?
- Cell biology (35.05%)
- Cancer research (5.15%)
- Stem cell (3.09%)
In recent papers he was focusing on the following fields of study:
His main research concerns Cell biology, Cancer research, Stem cell, Molecular biology and Retinal.
Kunio Nagashima has researched Cell biology in several fields, including Transcriptome, Induced pluripotent stem cell and Capsid.
His studies in Cancer research integrate themes in fields like Cell culture, Carcinogenesis, Cancer cell, Phenotype and Metastasis.
His Stem cell research is multidisciplinary, relying on both Apoptosis, Kinase and Mitochondrion.
In the subject of general Molecular biology, his work in Colocalization is often linked to Homology region, thereby combining diverse domains of study.
When carried out as part of a general Retinal research project, his work on Visual phototransduction and Retinitis pigmentosa is frequently linked to work in Congenital blindness, therefore connecting diverse disciplines of study.
Between 2013 and 2021, his most popular works were:
- In Vitro Modeling Using Ciliopathy-Patient-Derived Cells Reveals Distinct Cilia Dysfunctions Caused by CEP290 Mutations (66 citations)
- Chromatin decompaction by the nucleosomal binding protein HMGN5 impairs nuclear sturdiness (59 citations)
- HIV-1 uncoats in the nucleus near sites of integration (57 citations)
In his most recent research, the most cited papers focused on:
Kunio Nagashima mostly deals with Cell biology, Cancer research, Transcription, Cancer cell and Cell culture.
His Cell biology research includes themes of Molecular biology, Ciliopathy, Fibroblast growth factor and Induced pluripotent stem cell.
He conducted interdisciplinary study in his works that combined Molecular biology and Nucleosome binding.
The Cancer research study combines topics in areas such as Pathology, Kidney, Polycystic kidney disease and Kidney cancer.
He combines subjects such as EEF1A2, In vitro, Metastasis and Perinucleolar compartment with his study of Transcription.
The study incorporates disciplines such as Mitochondrial DNA, Stem cell, Kinase, Mitochondrion and Transplantation in addition to Cancer cell.
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