What is he best known for?
The fields of study he is best known for:
Kay Huebner mostly deals with Molecular biology, Cancer research, FHIT, Tumor suppressor gene and Genetics.
His work deals with themes such as Complementary DNA, Peptide sequence, Gene, Genomic library and DNA, which intersect with Molecular biology.
He interconnects Carcinogenesis, Cancer, Gene expression, WWOX and Lung cancer in the investigation of issues within Cancer research.
His FHIT research is multidisciplinary, incorporating perspectives in Acid anhydride hydrolases, Locus, Exon, Chromosomal fragile site and Histidine.
His Tumor suppressor gene research incorporates themes from Cell growth, Carcinogen, Apoptosis, Loss of heterozygosity and In vivo.
In his study, which falls under the umbrella issue of Genetics, CDX2, DLX3, HNF1B, homeobox A9 and Homeobox A1 is strongly linked to Myeloid leukemia.
His most cited work include:
- MicroRNA-29 family reverts aberrant methylation in lung cancer by targeting DNA methyltransferases 3A and 3B. (1363 citations)
- The FHIT Gene, Spanning the Chromosome 3p14.2 Fragile Site and Renal Carcinoma–Associated t(3;8) Breakpoint, Is Abnormal in Digestive Tract Cancers (914 citations)
- Molecular cloning of a new transforming gene from a chemically transformed human cell line (825 citations)
What are the main themes of his work throughout his whole career to date?
Kay Huebner spends much of his time researching Molecular biology, Cancer research, FHIT, Gene and Genetics.
His Molecular biology research integrates issues from Cell culture, Chromosomal translocation, Complementary DNA, Chromosome and Locus.
His Complementary DNA research incorporates elements of Peptide sequence and X chromosome.
His study looks at the intersection of Cancer research and topics like Carcinogenesis with Pathology.
Kay Huebner combines subjects such as DNA damage, Genome instability, Cancer cell, Loss of heterozygosity and Chromosomal fragile site with his study of FHIT.
His studies deal with areas such as Chromosome 21 and Chromosome 7 as well as Chromosome 22.
He most often published in these fields:
- Molecular biology (49.03%)
- Cancer research (36.77%)
- FHIT (35.81%)
What were the highlights of his more recent work (between 2007-2020)?
- FHIT (35.81%)
- Cancer research (36.77%)
- DNA damage (8.39%)
In recent papers he was focusing on the following fields of study:
Kay Huebner focuses on FHIT, Cancer research, DNA damage, Cancer and Gene.
His FHIT study incorporates themes from Chromosomal fragile site, Molecular biology, Genome instability and Cell biology.
His Molecular biology study combines topics in areas such as Mitomycin C, Histone, EZH2 and Loss of heterozygosity.
The Cancer research study combines topics in areas such as Gene expression, Carcinogenesis, WWOX, Suppressor and microRNA.
His Cancer research includes elements of Immunology and Pathology.
His work on Messenger RNA, Human genome, RNA and Bone morphogenetic protein as part of general Gene study is frequently linked to Text mining, therefore connecting diverse disciplines of science.
Between 2007 and 2020, his most popular works were:
- Reprogramming of miRNA networks in cancer and leukemia (321 citations)
- Breast cancer signatures for invasiveness and prognosis defined by deep sequencing of microRNA (293 citations)
- Integrated MicroRNA and mRNA Signatures Associated with Survival in Triple Negative Breast Cancer (129 citations)
In his most recent research, the most cited papers focused on:
His primary scientific interests are in Cancer research, FHIT, Cancer, DNA damage and Breast cancer.
His Cancer research study frequently draws connections between related disciplines such as Suppressor.
His research integrates issues of Molecular biology, Locus, Chromosomal fragile site, Chromosome Fragile Site and Cell cycle in his study of FHIT.
The concepts of his Molecular biology study are interwoven with issues in Chromosome and X chromosome.
His Locus study results in a more complete grasp of Genetics.
His Chromosomal fragile site research is included under the broader classification of Gene.
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