World's Best Scientists 2026 revealed!

D-Index & Metrics

Biology and Biochemistry

D-Index
65
Citations
18429
World Ranking
9029
National Ranking
4015

Overview

Juan Codina is affiliated with Wake Forest University in the United States. Their academic profile reflects an active engagement in the research community, although there are no recent papers, co-authors, or a list of publication venues directly associated with their name in the available data.

The absence of documented publications or collaborations suggests a profile that may either be emerging or focused on areas outside of widely indexed publication channels. Similarly, there is no recorded information about specific fields or subfields of study attributed to Juan Codina, which limits the ability to outline their precise research interests or specialized domains.

No book publications or awards have been listed, indicating no publicly noted major contributions in those categories. The lack of detailed data on topics worked on and recognized achievements leaves the profile concise and centered primarily on institutional affiliation.

This scientist's presence at Wake Forest University situates them within an academic environment known for diverse research disciplines, which may offer multiple avenues for engagement and scholarly work.

Best Publications

  • Beta-arrestin: a protein that regulates beta-adrenergic receptor function

    Martin J. Lohse;Jeffrey L. Benovic;Juan Codina;Marc G. Caron

  • A G protein directly regulates mammalian cardiac calcium channels.

    Atsuko Yatani;Juan Codina;Yutaka Imoto;John P. Reeves

  • Beta-arrestin2, a novel member of the arrestin/beta-arrestin gene family.

    Havard Attramadal;J. L. Arriza;C. Aoki;T. M. Dawson

  • Dopaminergic and ligand-independent activation of steroid hormone receptors.

    Ronan F. Power;Shaila K. Mani;Juan Codina;Orla M. Conneely

  • Functional desensitization of the isolated beta-adrenergic receptor by the beta-adrenergic receptor kinase: potential role of an analog of the retinal protein arrestin (48-kDa protein)

    J L Benovic;H Kühn;I Weyand;J Codina

  • Coupling of ATP-sensitive K+ channels to A1 receptors by G proteins in rat ventricular myocytes.

    G. E. Kirsch;J. Codina;L. Birnbaumer;A. M. Brown

  • Direct activation of mammalian atrial muscarinic potassium channels by GTP regulatory protein Gk

    Atsuko Yatani;Juan Codina;Arthur M. Brown;Lutz Birnbaumer

  • Gi2 mediates alpha 2-adrenergic inhibition of adenylyl cyclase in platelet membranes: in situ identification with G alpha C-terminal antibodies.

    William F. Simonds;Paul K. Goldsmith;Juan Codina;Cecilia G. Unson

  • Receptor-specific desensitization with purified proteins. Kinase dependence and receptor specificity of beta-arrestin and arrestin in the beta 2-adrenergic receptor and rhodopsin systems.

    M. J. Lohse;S. Andexinger;Ji. Pitcher;S. Trukawinski

  • Phosphorylation of the mammalian beta-adrenergic receptor by cyclic AMP-dependent protein kinase. Regulation of the rate of receptor phosphorylation and dephosphorylation by agonist occupancy and effects on coupling of the receptor to the stimulatory guanine nucleotide regulatory protein.

    J L Benovic;L J Pike;R A Cerione;C Staniszewski

  • The alpha subunit of the GTP binding protein Gk opens atrial potassium channels.

    Juan Codina;Atsuko Yatani;Dagoberto Grenet;Arthur M. Brown

  • The G protein-gated atrial K + channel is stimulated by three distinct G I α-subunits

    Atsuko Yatani;Rafael Mattera;Juan Codina;Rolf Graf

  • Stimulation and inhibition of adenylyl cyclases mediated by distinct regulatory proteins

    John D. Hildebrandt;Ronald D. Sekura;Juan Codina;Ravi Iyengar

  • The stimulatory G protein of adenylyl cyclase, Gs, also stimulates dihydropyridine-sensitive Ca2+ channels. Evidence for direct regulation independent of phosphorylation by cAMP-dependent protein kinase or stimulation by a dihydropyridine agonist.

    A Yatani;Y Imoto;J Codina;S L Hamilton

  • The mammalian beta 2-adrenergic receptor: reconstitution of functional interactions between pure receptor and pure stimulatory nucleotide binding protein of the adenylate cyclase system

    Richard A. Cerione;Juan Codina;Jeffrey L. Benovic;Robert J. Lefkowitz

  • Pertussis toxin substrate, the putative Ni component of adenylyl cyclases, is an alpha beta heterodimer regulated by guanine nucleotide and magnesium

    Juan Codina;John Hildebrandt;Ravi Iyengar;Lutz Birnbaumer

  • Ns and Ni, the stimulatory and inhibitory regulatory components of adenylyl cyclases. Purification of the human erythrocyte proteins without the use of activating regulatory ligands.

    J Codina;J D Hildebrandt;R D Sekura;M Birnbaumer

  • Splice variants of the alpha subunit of the G protein Gs activate both adenylyl cyclase and calcium channels

    Rafael Mattera;Michael P. Graziano;Atsuko Yatani;Zhimin Zhou

  • Newly identified brain potassium channels gated by the guanine nucleotide binding protein Go

    A. M. J. VanDongen;J. Codina;J. Olate;R. Mattera

  • Identification of a gamma subunit associated with the adenylyl cyclase regulatory proteins Ns and Ni.

    J D Hildebrandt;J Codina;R Risinger;L Birnbaumer

Frequent Co-Authors

Lutz Birnbaumer
Lutz Birnbaumer National Institutes of Health
Robert J. Lefkowitz
Robert J. Lefkowitz Duke University
Ravi Iyengar
Ravi Iyengar Icahn School of Medicine at Mount Sinai
Marc G. Caron
Marc G. Caron Duke University
Peter Gierschik
Peter Gierschik University of Ulm
Allen M. Spiegel
Allen M. Spiegel Albert Einstein College of Medicine
Jeffrey L. Benovic
Jeffrey L. Benovic Thomas Jefferson University
Martin J. Lohse
Martin J. Lohse ISAR Bioscience Institute
Susan L. Hamilton
Susan L. Hamilton Baylor College of Medicine
Walter Rosenthal
Walter Rosenthal Freie Universität Berlin

If you think any of the details on this page are incorrect, let us know.

Report an issue

We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:

Best Scientists Citing Juan Codina

Recently Published Articles