José R. Penadés mainly investigates Microbiology, Staphylococcus aureus, Biofilm, Bacteria and Virulence. His study in Staphylococcus aureus is interdisciplinary in nature, drawing from both Mutant, Gene and Pathogenicity island. His study on Pathogenicity island is covered under Genetics.
His Biofilm study combines topics in areas such as Staphylococcal infections, Cell wall, Operon and Staphylococcus simulans. The various areas that José R. Penadés examines in his Bacteria study include Fibronectin and Protein A. His studies in Virulence integrate themes in fields like Phylogenetics and Mobile genetic elements.
José R. Penadés mainly focuses on Genetics, Staphylococcus aureus, Microbiology, Pathogenicity island and Biofilm. His works in Virulence, Genome, Gene, Bacteriophage and Pathogen are all subjects of inquiry into Genetics. His biological study spans a wide range of topics, including Human pathogen and Regulon.
His Microbiology research is multidisciplinary, relying on both Prophage, SOS response, Mutant, Complementation and Bacteria. His studies deal with areas such as Plasmid, DNA, Mobile genetic elements, Horizontal gene transfer and Staphylococcus Phages as well as Pathogenicity island. José R. Penadés interconnects Antibiotics and Cell wall in the investigation of issues within Biofilm.
José R. Penadés focuses on Genetics, Pathogenicity island, Staphylococcus aureus, Genome and Pathogen. His research in Virulence, Transduction, Gene and Prophage are components of Genetics. His Virulence research is multidisciplinary, incorporating elements of Bacteriophage, CRISPR and Bacteria.
He focuses mostly in the field of Pathogenicity island, narrowing it down to topics relating to DNA and, in certain cases, Structural protein, Repressor, Molecular mechanism and Horizontal gene transfer. His Staphylococcus aureus research integrates issues from Biofilm, Microbiology and Regulon. His Pathogen study integrates concerns from other disciplines, such as Fixation and Experimental evolution.
His primary areas of study are Genetics, Pathogenicity island, Transduction, Virulence and Prophage. His study in Staphylococcus aureus and Bacteria is carried out as part of his Genetics studies. The Pathogenicity island study combines topics in areas such as DNA and Mobile genetic elements.
His Virulence research is multidisciplinary, incorporating perspectives in Human pathogen, Biofilm, Regulator, Staphylococcal infections and Adaptation. Gene and Microbiology are frequently intertwined in his study. José R. Penadés combines subjects such as Bacteriophage and Escherichia coli with his study of Microbiology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Bap, a Staphylococcus aureus Surface Protein Involved in Biofilm Formation
Carme Cucarella;Cristina Solano;Jaione Valle;Beatriz Amorena.
Journal of Bacteriology (2001)
The Enterococcal Surface Protein, Esp, Is Involved in Enterococcus faecalis Biofilm Formation
Alejandro Toledo-Arana;Jaione Valle;Cristina Solano;Marı́a Jesús Arrizubieta.
Applied and Environmental Microbiology (2001)
SarA and not σB is essential for biofilm development by Staphylococcus aureus
Jaione Valle;Alejandro Toledo-Arana;Carmen Berasain;Jean-Marc Ghigo.
Molecular Microbiology (2003)
Role of Biofilm-Associated Protein Bap in the Pathogenesis of Bovine Staphylococcus aureus
Carme Cucarella;M. Ángeles Tormo;Carles Úbeda;M. Pilar Trotonda.
Infection and Immunity (2004)
Bap: a family of surface proteins involved in biofilm formation.
Iñigo Lasa;José R. Penadés.
Research in Microbiology (2006)
The phage-related chromosomal islands of Gram-positive bacteria.
Richard P. Novick;Gail E. Christie;Jose R. Penadés.
Nature Reviews Microbiology (2010)
BapA, a large secreted protein required for biofilm formation and host colonization of Salmonella enterica serovar Enteritidis
Cristina Latasa;Agnès Roux;Alejandro Toledo-Arana;Jean-Marc Ghigo.
Molecular Microbiology (2005)
Bap-dependent biofilm formation by pathogenic species of Staphylococcus: evidence of horizontal gene transfer?
M. Ángeles Tormo;Erwin Knecht;Friedrich Götz;Iñigo Lasa.
Protein A-mediated multicellular behavior in Staphylococcus aureus.
Nekane Merino;Alejandro Toledo-Arana;Marta Vergara-Irigaray;Jaione Valle.
Journal of Bacteriology (2009)
Antibiotic-induced SOS response promotes horizontal dissemination of pathogenicity island-encoded virulence factors in staphylococci.
Carles Úbeda;Elisa Maiques;Erwin Knecht;Íñigo Lasa.
Molecular Microbiology (2005)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: