The scientist’s investigation covers issues in Kaposi's sarcoma, Angiogenesis, Immunology, Molecular biology and Proinflammatory cytokine. He interconnects In situ hybridization, Virus, Virology, Sarcoma and Monocyte in the investigation of issues within Kaposi's sarcoma. His work in Virology tackles topics such as Gene which are related to areas like Inhibitor of apoptosis.
His Angiogenesis study is associated with Cancer research. His research in Cancer research focuses on subjects like Vascular permeability, which are connected to Epidermis. The various areas that he examines in his Molecular biology study include Carcinogenesis, Cytosol, Matrix metalloproteinase, Second messenger system and In vivo.
His primary scientific interests are in Cancer research, Kaposi's sarcoma, Cell biology, Angiogenesis and Pathology. The concepts of his Cancer research study are interwoven with issues in Cell culture, Immunology, Cytokine, Carcinogenesis and Colorectal cancer. Michael Stürzl combines subjects such as Gene expression, Virus, Virology, Sarcoma and Proinflammatory cytokine with his study of Kaposi's sarcoma.
His work is dedicated to discovering how Virology, In situ hybridization are connected with Molecular biology and other disciplines. In his study, which falls under the umbrella issue of Angiogenesis, CD31 is strongly linked to Endothelial stem cell. He focuses mostly in the field of Pathology, narrowing it down to matters related to In vivo and, in some cases, In vitro.
His primary areas of study are Cancer research, Cell biology, Colorectal cancer, Pathology and Angiogenesis. His biological study spans a wide range of topics, including Carcinogenesis, Colitis, Mural cell and Vascular permeability. His studies deal with areas such as Immunology and Inflammatory bowel disease as well as Vascular permeability.
His work on Extracellular as part of general Cell biology study is frequently connected to Guanylate-binding protein, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them. The study incorporates disciplines such as Metastasis, Oncology and Immune escape in addition to Colorectal cancer. His Angiogenesis research includes elements of Cytokine and Paracrine signalling.
His scientific interests lie mostly in Cell biology, Cancer research, Colorectal cancer, Pathology and Inflammation. His study in the fields of GTPase under the domain of Cell biology overlaps with other disciplines such as Guanylate-binding protein. His Cancer research research is multidisciplinary, incorporating perspectives in Colitis, Inflammatory bowel disease, Carcinogenesis, Kaposi's sarcoma-associated herpesvirus and Vascular permeability.
His Colorectal cancer research includes themes of Apoptosis, Angiogenesis and Immunology. He has included themes like Cancer, Protein kinase B, Senescence, Vascular endothelial growth factor and PI3K/AKT/mTOR pathway in his Angiogenesis study. His research integrates issues of Programmed cell death and Mouse model of colorectal and intestinal cancer in his study of Immunology.
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Monocytes in Kaposi's sarcoma lesions are productively infected by human herpesvirus 8.
C Blasig;C Zietz;B Haar;F Neipel.
Journal of Virology (1997)
Biology of Kaposi's sarcoma.
B. Ensoli;C. Sgadari;G. Barillari;M.C. Sirianni.
European Journal of Cancer (2001)
Expression of vascular endothelial growth factor receptor-3 and podoplanin suggests a lymphatic endothelial cell origin of Kaposi's sarcoma tumor cells.
Weninger W;Partanen Ta;Breiteneder-Geleff S;Mayer C.
Laboratory Investigation (1999)
Endothelial CCR2 Signaling Induced by Colon Carcinoma Cells Enables Extravasation via the JAK2-Stat5 and p38MAPK Pathway
Monika Julia Wolf;Alexandra Hoos;Judith Bauer;Steffen Boettcher.
Cancer Cell (2012)
Reactivation and persistence of human herpesvirus-8 infection in B cells and monocytes by Th-1 cytokines increased in Kaposi's sarcoma.
Paolo Monini;Sandra Colombini;Michael Stürzl;Delia Goletti.
Vascular endothelial growth factor regulates angiogenesis and vascular permeability in Kaposi's sarcoma.
Emmanuelle Cornali;Christian Zietz;Roberto Benelli;Wolfgang Weninger.
American Journal of Pathology (1996)
Kaposi’s sarcoma: a result of the interplay among inflammatory cytokines, angiogenic factors and viral agents
Barbara Ensoli;Michael Stürzl.
Cytokine & Growth Factor Reviews (1998)
The Tat protein of human immunodeficiency virus type-1 promotes vascular cell growth and locomotion by engaging the α5β1 and αvβ3 integrins and by mobilizing sequestered basic fibroblast growth factor
Giovanni Barillari;Cecilia Sgadari;Valeria Fiorelli;Felipe Samaniego.
The helical domain of GBP-1 mediates the inhibition of endothelial cell proliferation by inflammatory cytokines.
Eric Guenzi;Kristin Töpolt;Emmanuelle Cornali;Emmanuelle Cornali;Clara Lubeseder‐Martellato.
The EMBO Journal (2001)
Expression of K13/v-FLIP Gene of Human Herpesvirus 8 and Apoptosis in Kaposi's Sarcoma Spindle Cells
Michael Stürzl;Christine Hohenadl;Christian Zietz;Esmeralda Castanos-Velez.
Journal of the National Cancer Institute (1999)
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