D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 64 Citations 11,580 234 World Ranking 6394 National Ranking 463

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • DNA

The scientist’s investigation covers issues in Kaposi's sarcoma, Angiogenesis, Immunology, Molecular biology and Proinflammatory cytokine. He interconnects In situ hybridization, Virus, Virology, Sarcoma and Monocyte in the investigation of issues within Kaposi's sarcoma. His work in Virology tackles topics such as Gene which are related to areas like Inhibitor of apoptosis.

His Angiogenesis study is associated with Cancer research. His research in Cancer research focuses on subjects like Vascular permeability, which are connected to Epidermis. The various areas that he examines in his Molecular biology study include Carcinogenesis, Cytosol, Matrix metalloproteinase, Second messenger system and In vivo.

His most cited work include:

  • Monocytes in Kaposi's sarcoma lesions are productively infected by human herpesvirus 8. (264 citations)
  • Biology of Kaposi's sarcoma. (230 citations)
  • Expression of vascular endothelial growth factor receptor-3 and podoplanin suggests a lymphatic endothelial cell origin of Kaposi's sarcoma tumor cells. (226 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Cancer research, Kaposi's sarcoma, Cell biology, Angiogenesis and Pathology. The concepts of his Cancer research study are interwoven with issues in Cell culture, Immunology, Cytokine, Carcinogenesis and Colorectal cancer. Michael Stürzl combines subjects such as Gene expression, Virus, Virology, Sarcoma and Proinflammatory cytokine with his study of Kaposi's sarcoma.

His work is dedicated to discovering how Virology, In situ hybridization are connected with Molecular biology and other disciplines. In his study, which falls under the umbrella issue of Angiogenesis, CD31 is strongly linked to Endothelial stem cell. He focuses mostly in the field of Pathology, narrowing it down to matters related to In vivo and, in some cases, In vitro.

He most often published in these fields:

  • Cancer research (29.79%)
  • Kaposi's sarcoma (19.57%)
  • Cell biology (19.15%)

What were the highlights of his more recent work (between 2013-2021)?

  • Cancer research (29.79%)
  • Cell biology (19.15%)
  • Colorectal cancer (14.04%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Cancer research, Cell biology, Colorectal cancer, Pathology and Angiogenesis. His biological study spans a wide range of topics, including Carcinogenesis, Colitis, Mural cell and Vascular permeability. His studies deal with areas such as Immunology and Inflammatory bowel disease as well as Vascular permeability.

His work on Extracellular as part of general Cell biology study is frequently connected to Guanylate-binding protein, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them. The study incorporates disciplines such as Metastasis, Oncology and Immune escape in addition to Colorectal cancer. His Angiogenesis research includes elements of Cytokine and Paracrine signalling.

Between 2013 and 2021, his most popular works were:

  • Inhibition of cGAS DNA Sensing by a Herpesvirus Virion Protein (139 citations)
  • Vascular occlusion by neutrophil extracellular traps in COVID-19. (81 citations)
  • Gamma Interferon-Induced Guanylate Binding Protein 1 Is a Novel Actin Cytoskeleton Remodeling Factor (46 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • DNA

His scientific interests lie mostly in Cell biology, Cancer research, Colorectal cancer, Pathology and Inflammation. His study in the fields of GTPase under the domain of Cell biology overlaps with other disciplines such as Guanylate-binding protein. His Cancer research research is multidisciplinary, incorporating perspectives in Colitis, Inflammatory bowel disease, Carcinogenesis, Kaposi's sarcoma-associated herpesvirus and Vascular permeability.

His Colorectal cancer research includes themes of Apoptosis, Angiogenesis and Immunology. He has included themes like Cancer, Protein kinase B, Senescence, Vascular endothelial growth factor and PI3K/AKT/mTOR pathway in his Angiogenesis study. His research integrates issues of Programmed cell death and Mouse model of colorectal and intestinal cancer in his study of Immunology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Monocytes in Kaposi's sarcoma lesions are productively infected by human herpesvirus 8.

C Blasig;C Zietz;B Haar;F Neipel.
Journal of Virology (1997)

339 Citations

Biology of Kaposi's sarcoma.

B. Ensoli;C. Sgadari;G. Barillari;M.C. Sirianni.
European Journal of Cancer (2001)

310 Citations

Expression of vascular endothelial growth factor receptor-3 and podoplanin suggests a lymphatic endothelial cell origin of Kaposi's sarcoma tumor cells.

Weninger W;Partanen Ta;Breiteneder-Geleff S;Mayer C.
Laboratory Investigation (1999)

295 Citations

Endothelial CCR2 Signaling Induced by Colon Carcinoma Cells Enables Extravasation via the JAK2-Stat5 and p38MAPK Pathway

Monika Julia Wolf;Alexandra Hoos;Judith Bauer;Steffen Boettcher.
Cancer Cell (2012)

270 Citations

Reactivation and persistence of human herpesvirus-8 infection in B cells and monocytes by Th-1 cytokines increased in Kaposi's sarcoma.

Paolo Monini;Sandra Colombini;Michael Stürzl;Delia Goletti.
Blood (1999)

235 Citations

Vascular endothelial growth factor regulates angiogenesis and vascular permeability in Kaposi's sarcoma.

Emmanuelle Cornali;Christian Zietz;Roberto Benelli;Wolfgang Weninger.
American Journal of Pathology (1996)

232 Citations

Kaposi’s sarcoma: a result of the interplay among inflammatory cytokines, angiogenic factors and viral agents

Barbara Ensoli;Michael Stürzl.
Cytokine & Growth Factor Reviews (1998)

227 Citations

The Tat protein of human immunodeficiency virus type-1 promotes vascular cell growth and locomotion by engaging the α5β1 and αvβ3 integrins and by mobilizing sequestered basic fibroblast growth factor

Giovanni Barillari;Cecilia Sgadari;Valeria Fiorelli;Felipe Samaniego.
Blood (1999)

224 Citations

The helical domain of GBP-1 mediates the inhibition of endothelial cell proliferation by inflammatory cytokines.

Eric Guenzi;Kristin Töpolt;Emmanuelle Cornali;Emmanuelle Cornali;Clara Lubeseder‐Martellato.
The EMBO Journal (2001)

220 Citations

Expression of K13/v-FLIP Gene of Human Herpesvirus 8 and Apoptosis in Kaposi's Sarcoma Spindle Cells

Michael Stürzl;Christine Hohenadl;Christian Zietz;Esmeralda Castanos-Velez.
Journal of the National Cancer Institute (1999)

219 Citations

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