John D. Rioux focuses on Genetics, Genome-wide association study, Immunology, Genetic association and Disease. Locus, Linkage disequilibrium, Haplotype, Single-nucleotide polymorphism and Genotyping are the subjects of his Genetics studies. His studies in Locus integrate themes in fields like Genetic linkage and Allele.
His research integrates issues of ATG16L1, IRGM, Inflammatory bowel disease, Bioinformatics and Ulcerative colitis in his study of Genome-wide association study. Immunology is often connected to Genetic architecture in his work. His study explores the link between Genetic association and topics such as Case-control study that cross with problems in PTPN22 and Common disease-common variant.
John D. Rioux mostly deals with Genetics, Immunology, Inflammatory bowel disease, Disease and Genome-wide association study. His work on Genetics deals in particular with Single-nucleotide polymorphism, Locus, Haplotype, Linkage disequilibrium and Genetic association. His research in Immunology intersects with topics in Odds ratio and Allele.
His Inflammatory bowel disease research incorporates elements of Phenotype and Ulcerative colitis. The study incorporates disciplines such as Computational biology, Gene and Immune system in addition to Disease. His Genome-wide association study research is multidisciplinary, incorporating perspectives in Bioinformatics, Genotyping, IRGM, ATG16L1 and Genetic architecture.
John D. Rioux spends much of his time researching Inflammatory bowel disease, Internal medicine, Genetics, Genome-wide association study and Immunology. The Inflammatory bowel disease study combines topics in areas such as Phenotype, Patient perceptions, Single-nucleotide polymorphism and Ulcerative colitis. His Genetics study is mostly concerned with Genetic association, Linkage disequilibrium, Allele, Exome sequencing and Allele frequency.
In general Allele study, his work on Haplotype often relates to the realm of Offspring, thereby connecting several areas of interest. His Genome-wide association study research is multidisciplinary, incorporating elements of Medical genetics, Bioinformatics and Genetic architecture. His study connects LRRK2 and Immunology.
His main research concerns Genome-wide association study, Genetics, Inflammatory bowel disease, Immunology and Genetic association. His Genome-wide association study study is focused on Single-nucleotide polymorphism in general. His Linkage disequilibrium and Allele study in the realm of Genetics connects with subjects such as Coding and Low frequency.
His Inflammatory bowel disease research is multidisciplinary, relying on both Epithelium, Ubiquitin, Phenotype, Ulcerative colitis and Crohn's disease. The various areas that he examines in his Immunology study include LRRK2, Genotype, Interleukin-23 receptor, Cell surface receptor and STAT3. His Genetic association research incorporates themes from Acquired immune system, White blood cell, Minor allele frequency and Myeloid.
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Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease
Luke Jostins;Stephan Ripke;Rinse K Weersma;Richard H Duerr.
A Genome-Wide Association Study Identifies IL23R as an Inflammatory Bowel Disease Gene
Richard H. Duerr;Kent D. Taylor;Steven R. Brant;Steven R. Brant;John D. Rioux;John D. Rioux.
Large-Scale Identification, Mapping, and Genotyping of Single-Nucleotide Polymorphisms in the Human Genome
David G. Wang;Jian-Bing Fan;Jian-Bing Fan;Chia-Jen Siao;Chia-Jen Siao;Anthony Berno;Anthony Berno.
Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease
Jeffrey C. Barrett;Sarah Hansoul;Dan L. Nicolae;Judy H. Cho.
Nature Genetics (2008)
Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci
Andre Franke;Dermot P B McGovern;Jeffrey C. Barrett;Kai Wang.
Nature Genetics (2010)
Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis
Stephen Sawcer;Garrett Hellenthal;Matti Pirinen;Chris C. A. Spencer.
Risk alleles for multiple sclerosis identified by a genomewide study.
David A. Hafler;Alastair Compston;Stephen Sawcer;Mark J. Daly.
The New England Journal of Medicine (2007)
High-resolution haplotype structure in the human genome.
Mark J. Daly;John D. Rioux;Stephen F. Schaffner;Thomas J. Hudson;Thomas J. Hudson.
Nature Genetics (2001)
Hundreds of variants clustered in genomic loci and biological pathways affect human height
Hana Lango Allen;Karol Estrada;Guillaume Lettre;Sonja I. Berndt.
Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesis
John D. Rioux;John D. Rioux;Ramnik J. Xavier;Kent D. Taylor;Mark S. Silverberg.
Nature Genetics (2007)
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