His primary scientific interests are in Inflammatory bowel disease, Genetics, Immunology, Genome-wide association study and Ulcerative colitis. His Inflammatory bowel disease study is concerned with Disease in general. His studies deal with areas such as Gene expression profiling, Gene and NOD2 as well as Immunology.
The various areas that he examines in his Genome-wide association study study include Case-control study, ATG16L1, Diarrhea and Interleukin-23 receptor. As a part of the same scientific family, Steven R. Brant mostly works in the field of Ulcerative colitis, focusing on Cohort and, on occasion, Missense mutation and Pharmacogenetics. The study incorporates disciplines such as Medical genetics and Genetic architecture in addition to IRGM.
His scientific interests lie mostly in Inflammatory bowel disease, Internal medicine, Genetics, Crohn's disease and Ulcerative colitis. His research integrates issues of Odds ratio, Colitis, Immunology and Single-nucleotide polymorphism, Genotype in his study of Inflammatory bowel disease. His Internal medicine research integrates issues from Gastroenterology, Surgery and Colonoscopy.
The subject of his Crohn's disease research is within the realm of Disease. His Ulcerative colitis research includes themes of Incidence, Retrospective cohort study, Cohort and Confidence interval. In his study, Interleukin-23 receptor is strongly linked to ATG16L1, which falls under the umbrella field of Genome-wide association study.
Steven R. Brant focuses on Internal medicine, Inflammatory bowel disease, Gastroenterology, Crohn's disease and Ulcerative colitis. His work on Chemokine as part of general Internal medicine research is often related to Clostridium difficile, thus linking different fields of science. Inflammatory bowel disease is the subject of his research, which falls under Disease.
His Disease research is multidisciplinary, incorporating elements of Genetics, Incidence, Family history and Cohort. As a member of one scientific family, Steven R. Brant mostly works in the field of Ulcerative colitis, focusing on Aminosalicylic acid and, on occasion, Drug metabolism. His Immunology research is multidisciplinary, relying on both Exome sequencing and Gene.
His primary areas of study are Inflammatory bowel disease, Internal medicine, Human leukocyte antigen, Crohn's disease and Gastroenterology. His work in Inflammatory bowel disease tackles topics such as Colitis which are related to areas like Neutrophilia, Systemic inflammation and FOXP3. His work on Ulcerative colitis and Logistic regression as part of his general Internal medicine study is frequently connected to Research design, thereby bridging the divide between different branches of science.
Ulcerative colitis is a subfield of Disease that Steven R. Brant studies. His biological study spans a wide range of topics, including Nurses' Health Study and Calcitriol receptor, Vitamin D and neurology, Vitamin D-binding protein, Vitamin. His Gastroenterology research incorporates elements of Odds ratio, Receptor, microRNA, Biomarker and Western blot.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
A frameshift mutation in NOD2 associated with susceptibility to Crohn's disease
Yasunori Ogura;Denise K. Bonen;Naohiro Inohara;Dan L. Nicolae.
Nature (2001)
Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease
Luke Jostins;Stephan Ripke;Rinse K Weersma;Richard H Duerr.
Nature (2012)
Toward an Integrated Clinical, Molecular and Serological Classification of Inflammatory Bowel Disease: Report of a Working Party of the 2005 Montreal World Congress of Gastroenterology
Mark S. Silverberg;Jack Satsangi;Tariq Ahmad;Ian D. R. Arnott.
Canadian Journal of Gastroenterology & Hepatology (2005)
A Genome-Wide Association Study Identifies IL23R as an Inflammatory Bowel Disease Gene
Richard H. Duerr;Kent D. Taylor;Steven R. Brant;Steven R. Brant;John D. Rioux;John D. Rioux.
Science (2006)
Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease
Jeffrey C. Barrett;Sarah Hansoul;Dan L. Nicolae;Judy H. Cho.
Nature Genetics (2008)
Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci
Andre Franke;Dermot P B McGovern;Jeffrey C. Barrett;Kai Wang.
Nature Genetics (2010)
Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesis
John D. Rioux;John D. Rioux;Ramnik J. Xavier;Kent D. Taylor;Mark S. Silverberg.
Nature Genetics (2007)
Meta-analysis identifies 29 additional ulcerative colitis risk loci, increasing the number of confirmed associations to 47.
Carl A. Anderson;Gabrielle Boucher;Charlie W. Lees;Andre Franke.
Nature Genetics (2011)
Deep resequencing of GWAS loci identifies independent rare variants associated with inflammatory bowel disease.
Manuel A. Rivas;Manuel A. Rivas;Manuel A. Rivas;Mélissa Beaudoin;Agnes Gardet;Christine Stevens.
Nature Genetics (2011)
Deletion polymorphism upstream of IRGM associated with altered IRGM expression and Crohn's disease.
Steven A McCarroll;Alan Huett;Petric Kuballa;Shannon D Chilewski.
Nature Genetics (2008)
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