Jianjie Ma mainly focuses on Cell biology, Ryanodine receptor, Skeletal muscle, Biochemistry and Endoplasmic reticulum. His studies deal with areas such as Apoptosis, Muscular dystrophy, Membrane protein and Cell membrane as well as Cell biology. His Ryanodine receptor study incorporates themes from Heptanol, Molecular biology and Biophysics.
His Skeletal muscle research is multidisciplinary, incorporating elements of Mitochondrial ROS, Signal transduction, Muscle contraction, Mitochondrial permeability transition pore and Myocyte. His work focuses on many connections between Biochemistry and other disciplines, such as Voltage-dependent calcium channel, that overlap with his field of interest in Ruthenium red. Jianjie Ma works mostly in the field of Endoplasmic reticulum, limiting it down to concerns involving Calcium signaling and, occasionally, Calcium channel.
His primary scientific interests are in Cell biology, Skeletal muscle, Biochemistry, Ryanodine receptor and Endoplasmic reticulum. His Cell biology research is multidisciplinary, relying on both Membrane protein and Cell membrane. The various areas that Jianjie Ma examines in his Skeletal muscle study include Myocyte and Cardiac muscle.
His Biochemistry study combines topics from a wide range of disciplines, such as Biophysics, Antibody and Voltage-dependent calcium channel. His Ryanodine receptor study integrates concerns from other disciplines, such as Molecular biology, Binding site and Muscle contraction. His Endoplasmic reticulum course of study focuses on Homeostasis and STIM1.
Jianjie Ma mostly deals with Cell biology, Internal medicine, Skeletal muscle, Wound healing and Pharmacology. Many of his studies on Cell biology apply to Cell membrane as well. His study in Internal medicine is interdisciplinary in nature, drawing from both Endocrinology and Cardiology.
His Skeletal muscle research integrates issues from Ryanodine receptor, PI3K/AKT/mTOR pathway and Evans Blue. His Ryanodine receptor study results in a more complete grasp of Endoplasmic reticulum. Jianjie Ma has researched Calcium signaling in several fields, including Biophysics and Lysosome.
Jianjie Ma focuses on Cell biology, Wound healing, Skeletal muscle, Internal medicine and Fluorescence microscope. His studies in Cell biology integrate themes in fields like Muscle atrophy and Cell growth. The concepts of his Wound healing study are interwoven with issues in Angiogenesis, Nanotechnology and Wound care.
Jianjie Ma interconnects Autophagy, Lysosome, Protein degradation, Tissue homeostasis and PI3K/AKT/mTOR pathway in the investigation of issues within Skeletal muscle. His Internal medicine research is multidisciplinary, incorporating perspectives in Endocrinology and Cytoskeleton. The study incorporates disciplines such as Spectrin, Ankyrin and Edema in addition to Endocrinology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The genome of the social amoeba Dictyostelium discoideum
L. Eichinger;J. A. Pachebat;J. A. Pachebat;G. Glöckner;M.-A. Rajandream.
NAADP mobilizes calcium from acidic organelles through two-pore channels
Peter J. Calcraft;Margarida Ruas;Zui Pan;Xiaotong Cheng.
Purified ryanodine receptor from rabbit skeletal muscle is the calcium-release channel of sarcoplasmic reticulum.
Jeffrey S. Smith;Toshiaki Imagawa;Jianjie Ma;Michael Fill.
The Journal of General Physiology (1988)
MG53 nucleates assembly of cell membrane repair machinery
Chuanxi Cai;Haruko Masumiya;Noah Weisleder;Noriyuki Matsuda.
Nature Cell Biology (2009)
Mitochondrial depolarization accompanies cytochrome c release during apoptosis in PC6 cells
Kaisa M. Heiskanen;Manjunatha B. Bhat;Hsing-Wen Wang;Jianjie Ma.
Journal of Biological Chemistry (1999)
Mechanism of action of isothiocyanates: the induction of ARE-regulated genes is associated with activation of ERK and JNK and the phosphorylation and nuclear translocation of Nrf2.
Changjiang Xu;Xiaoling Yuan;Zui Pan;Guoxiang Shen.
Molecular Cancer Therapeutics (2006)
Abnormal ryanodine receptor channels in malignant hyperthermia
M. Fill;R. Coronado;James R Mickelson;J. Vilven.
Biophysical Journal (1990)
Membrane Repair Defects in Muscular Dystrophy Are Linked to Altered Interaction between MG53, Caveolin-3, and Dysferlin
Chuanxi Cai;Noah Weisleder;Jae-Kyun Ko;Shinji Komazaki.
Journal of Biological Chemistry (2009)
Inhibition of Intestinal Tumorigenesis in Apcmin/+ Mice by (−)-Epigallocatechin-3-Gallate, the Major Catechin in Green Tea
Jihyeung Ju;Jungil Hong;Jian Nian Zhou;Zui Pan.
Cancer Research (2005)
Zinc in Wound Healing Modulation.
Pei-Hui Lin;Matthew Sermersheim;Haichang Li;Peter H U Lee.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: