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Miyuki Nishi

Miyuki Nishi

D-Index & Metrics

Biology and Biochemistry

D-Index
58
Citations
12682
World Ranking
13134
National Ranking
906

Overview

Miyuki Nishi is a researcher affiliated with Kyoto University in Japan specializing in biochemistry, genetics, and molecular biology with a significant focus on medicine. Their work primarily addresses molecular biology and cardiac-related fields, contributing extensively to understanding ion channel regulation and function as well as cardiac electrophysiology and arrhythmias.

Their research contributions extend to subfields including cellular and molecular neuroscience, sensory systems, and nutrition and dietetics. Key topics explored in their publications include:

  • Ion channel regulation and function
  • Cardiac electrophysiology and arrhythmias
  • Muscle physiology and disorders
  • Ion channels and receptors
  • Trace elements in health
  • Nuclear structure and function
  • Connective tissue disorders research

Nishi has published in multiple scientific venues, with frequent appearances in:

  • bioRxiv (Cold Spring Harbor Laboratory)
  • Cell Calcium
  • The Journal of General Physiology
  • Cells
  • Cell Death and Disease

Recent papers authored or co-authored by Nishi include:

  • "Enhanced Ca2+ handling in thioglycolate-elicited peritoneal macrophages" (2021), Cell Calcium
  • "Atypical cell death and insufficient matrix organization in long-bone growth plates from Tric-b-knockout mice" (2023), Cell Death and Disease
  • "Ca2+ storage function is altered in the sarcoplasmic reticulum of skeletal muscle lacking mitsugumin 23" (2024), American Journal of Physiology-Cell Physiology
  • "The biophysical properties of TRIC-A and TRIC-B and their interactions with RyR2" (2023), The Journal of General Physiology
  • "Decreased cardiac pacemaking and attenuated β-adrenergic response in TRIC-A knockout mice" (2020), PLoS ONE

Throughout their career, Nishi has collaborated frequently with several researchers, with the most frequent co-authors being:

  • Hiroshi Takeshima
  • Atsuhiko Ichimura
  • Takaaki Kawabe
  • Yuu Miyazaki
  • Hiroki Nagatomo

Best Publications

  • Junctophilins: a novel family of junctional membrane complex proteins.

    Hiroshi Takeshima;Hiroshi Takeshima;Shinji Komazaki;Miyuki Nishi;Miyuki Nishi;Masamitsu Iino

  • TRPM2-mediated Ca2+ influx induces chemokine production in monocytes that aggravates inflammatory neutrophil infiltration

    Shinichiro Yamamoto;Shunichi Shimizu;Shigeki Kiyonaka;Nobuaki Takahashi

  • cDNA cloning and regional distribution of a novel member of the opioid receptor family

    Kazuhiko Fukuda;Shigehisa Kato;Kenjiro Mori;Miyuki Nishi

  • MG53 nucleates assembly of cell membrane repair machinery

    Chuanxi Cai;Haruko Masumiya;Noah Weisleder;Noriyuki Matsuda

  • Excitation-contraction uncoupling and muscular degeneration in mice lacking functional skeletal muscle ryanodine-receptor gene

    Takeshima H;Iino M;Takekura H;Nishi M

  • Primary structures and expression from cDNAs of rat opioid receptor δ-and μ-subtypes

    Kazuhiko Fukuda;Shigehisa Kato;Kenjiro Mori;Miyuki Nishi

  • Facilitation of long-term potentiation and memory in mice lacking nociceptin receptors

    Toshiya Manabe;Yukihiro Noda;Takayoshi Mamiya;Hiroyuki Katagiri

  • Unrestrained nociceptive response and disregulation of hearing ability in mice lacking the nociceptin/orphaninFQ receptor

    Miyuki Nishi;Takeshi Houtani;Yukihiro Noda;Takayoshi Mamiya

  • The POU domain transcription factor Brn-2 is required for the determination of specific neuronal lineages in the hypothalamus of the mouse.

    S Nakai;H Kawano;T Yudate;M Nishi

  • Brn-1 and Brn-2 share crucial roles in the production and positioning of mouse neocortical neurons.

    Yoshinobu Sugitani;Shigeyasu Nakai;Osamu Minowa;Miyuki Nishi

  • Membrane Repair Defects in Muscular Dystrophy Are Linked to Altered Interaction between MG53, Caveolin-3, and Dysferlin

    Chuanxi Cai;Noah Weisleder;Jae-Kyun Ko;Shinji Komazaki

  • Embryonic lethality and abnormal cardiac myocytes in mice lacking ryanodine receptor type 2.

    Hiroshi Takeshima;Shinji Komazaki;Kenzo Hirose;Miyuki Nishi

  • MG53 constitutes a primary determinant of cardiac ischemic preconditioning.

    Chun-Mei Cao;Yan Zhang;Noah Weisleder;Christopher Ferrante

  • Generation and Characterization of Mutant Mice Lacking Ryanodine Receptor Type 3

    Hiroshi Takeshima;Takaaki Ikemoto;Miyuki Nishi;Nobuyoshi Nishiyama

  • Deficiency of triad junction and contraction in mutant skeletal muscle lacking junctophilin type 1.

    Koichi Ito;Shinji Komazaki;Kazushige Sasamoto;Morikatsu Yoshida

  • Crucial roles of Brn1 in distal tubule formation and function in mouse kidney

    Shigeyasu Nakai;Yoshinobu Sugitani;Hiroshi Sato;Sadayoshi Ito

  • Structure and Regional Distribution of Nociceptin/Orphanin FQ Precursor

    Takeshi Houtani;Miyuki Nishi;Hiroshi Takeshima;Toshihide Nukada

  • Ca(2+)-induced Ca2+ release in myocytes from dyspedic mice lacking the type-1 ryanodine receptor.

    H Takeshima;T Yamazawa;T Ikemoto;H Takekura

  • Isolation and characterization of a gene for a ryanodine receptor/calcium release channel in Drosophila melanogaster.

    Hiroshi Takeshima;Miyuki Nishi;Naoyuki Iwabe;Takashi Miyata

  • TRIC channels are essential for Ca2+ handling in intracellular stores.

    Masayuki Yazawa;Christopher Ferrante;Jue Feng;Kazuhiro Mio

Frequent Co-Authors

Hiroshi Takeshima
Hiroshi Takeshima Kyoto University
Jianjie Ma
Jianjie Ma The Ohio State University
Tetsuo Noda
Tetsuo Noda Japanese Foundation For Cancer Research
Francesco Zorzato
Francesco Zorzato University of Ferrara
Masamitsu Iino
Masamitsu Iino Nihon University
Susan Treves
Susan Treves University Hospital of Basel
Tasuku Honjo
Tasuku Honjo Kyoto University
Yukihiro Noda
Yukihiro Noda Meijo University
Toshitaka Nabeshima
Toshitaka Nabeshima Meijo University
Kayo Inaba
Kayo Inaba Kyoto University

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