Jian Q. Feng spends much of his time researching Cell biology, Internal medicine, Endocrinology, DMP1 and Osteoblast. The various areas that he examines in his Cell biology study include Dental alveolus, Anatomy and Pathology. In Internal medicine, he works on issues like Bone morphogenetic protein, which are connected to Wnt signaling pathway.
His biological study spans a wide range of topics, including Regulation of gene expression and Fibroblast growth factor. His DMP1 study integrates concerns from other disciplines, such as Mineralized tissues, Pulp, Dentin, Odontoblast and Gene targeting. His studies deal with areas such as Immunology, Bone marrow, Osteoprotegerin and Cellular differentiation as well as Osteoblast.
Jian Q. Feng mainly investigates Cell biology, DMP1, Endocrinology, Internal medicine and Osteocyte. He combines subjects such as Osteoblast, Anatomy and Dentin, Odontoblast with his study of Cell biology. His Osteoblast study incorporates themes from Cellular differentiation and Bone marrow.
His DMP1 research is multidisciplinary, incorporating perspectives in Transgene, Fibroblast growth factor 23, Hypophosphatemic Rickets, Pathology and Molecular biology. His Internal medicine research includes themes of In vivo, Bone morphogenetic protein and Conditional gene knockout. His work in Osteocyte covers topics such as Bone remodeling which are related to areas like Osteoclast.
Jian Q. Feng mainly focuses on Cell biology, Wnt signaling pathway, Bone cell, DMP1 and Osteocyte. His Cell biology research incorporates elements of Cementum and Cell fate determination. His Wnt signaling pathway research includes elements of Stem cell and Odontoblast.
The study incorporates disciplines such as Chondrogenesis, Chondrocyte and Cartilage in addition to Bone cell. His research in DMP1 intersects with topics in NFIC, Dentin sialoprotein, Odontoblast differentiation and Fibroblast growth factor 23. His studies in Osteocyte integrate themes in fields like Endoplasmic reticulum, Internal medicine, GTPase and Endocrinology.
His primary scientific interests are in Cell biology, Osteocyte, Sclerostin, RANKL and Osteoclast. His Cell biology research integrates issues from Cementum and Periodontal fiber. His study in Cementum is interdisciplinary in nature, drawing from both Dental alveolus, Periodontium, Stem cell, AXIN2 and GLI1.
His Osteocyte research incorporates themes from Endoplasmic reticulum, Tissue homeostasis, GTPase and Homeostasis. The concepts of his RANKL study are interwoven with issues in Focal adhesion, Bone marrow, Bone resorption and Osteoblast. His Wnt signaling pathway research is multidisciplinary, relying on both Dental cementum, Genetically modified mouse, Mesenchymal stem cell and Cellular origin.
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Identification of the haematopoietic stem cell niche and control of the niche size
Jiwang Zhang;Chao Niu;Ling Ye;Haiyang Huang.
Nature (2003)
Evidence for osteocyte regulation of bone homeostasis through RANKL expression
Tomoki Nakashima;Mikihito Hayashi;Takanobu Fukunaga;Kosaku Kurata.
Nature Medicine (2011)
Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolism
Jian Q Feng;Leanne M Ward;Shiguang Liu;Yongbo Lu.
Nature Genetics (2006)
The Growth Factor Progranulin Binds to TNF Receptors and Is Therapeutic Against Inflammatory Arthritis in Mice
Wei Tang;Yi Lu;Yi Lu;Qing-Yun Tian;Yan Zhang.
Science (2011)
Osteocyte Wnt/β-Catenin Signaling Is Required for Normal Bone Homeostasis
Ina Kramer;Christine Halleux;Hansjoerg Keller;Marco Pegurri.
Molecular and Cellular Biology (2010)
Implant-derived magnesium induces local neuronal production of CGRP to improve bone-fracture healing in rats
Yifeng Zhang;Jiankun Xu;Ye Chun Ruan;Mei Kuen Yu.
Nature Medicine (2016)
Extracellular matrix made by bone marrow cells facilitates expansion of marrow-derived mesenchymal progenitor cells and prevents their differentiation into osteoblasts
Xiao Dong Chen;Vladimir Dusevich;Jian Q. Feng;Stavros C. Manolagas.
Journal of Bone and Mineral Research (2007)
Deletion of dentin matrix protein-1 leads to a partial failure of maturation of predentin into dentin, hypomineralization, and expanded cavities of pulp and root canal during postnatal tooth development.
Ling Ye;Mary MacDougall;Shubin Zhang;Yixia Xie.
Journal of Biological Chemistry (2004)
periostin Null Mice Exhibit Dwarfism, Incisor Enamel Defects, and an Early-Onset Periodontal Disease-Like Phenotype
Hector Rios;Shrinagesh V. Koushik;Haiyan Wang;Jian Wang.
Molecular and Cellular Biology (2005)
Multiple functions of Osterix are required for bone growth and homeostasis in postnatal mice
Xin Zhou;Zhaoping Zhang;Jian Q. Feng;Vladmir M. Dusevich.
Proceedings of the National Academy of Sciences of the United States of America (2010)
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