His primary areas of study are Cell biology, Growth factor, Cartilage oligomeric matrix protein, ADAMTS and Cartilage. His studies deal with areas such as Biochemistry and Arthritis as well as Cell biology. His Growth factor research is multidisciplinary, incorporating elements of Tumor necrosis factor alpha, Immunology, Fibroblast growth factor receptor 3, Chondrocyte and Fibroblast growth factor receptor.
His Chondrocyte research includes themes of Chondrogenesis, Cancer research and Cellular differentiation. Chuan-ju Liu has researched ADAMTS in several fields, including Plasma protein binding and Small interfering RNA. His research integrates issues of Epidermal growth factor and Granulin in his study of Cartilage.
His main research concerns Cell biology, Molecular biology, Immunology, Arthritis and Cancer research. His research in Cell biology intersects with topics in Chondrocyte, Endocrinology, Growth factor and Internal medicine. His biological study spans a wide range of topics, including MyoD, Reporter gene, Gene expression, Transcription factor and C2C12.
His studies in Arthritis integrate themes in fields like ADAMTS and Bioinformatics. His Cancer research study which covers Pathology that intersects with Bone morphogenetic protein. His Chondrogenesis study deals with Cellular differentiation intersecting with Bone morphogenetic protein 2.
Chuan-ju Liu mainly focuses on Cell biology, Tumor necrosis factor alpha, Chondrogenesis, Cartilage and In vivo. His Cell biology study combines topics from a wide range of disciplines, such as Chondrocyte, Transgene and Bone resorption. His Chondrocyte study combines topics in areas such as Elastin and Growth factor.
The concepts of his Tumor necrosis factor alpha study are interwoven with issues in TUNEL assay, Bioinformatics, Inflammation, Pathogenesis and Arthritis. Chuan-ju Liu interconnects Proinflammatory cytokine, Type 1 diabetes and Endocrinology in the investigation of issues within Chondrogenesis. His Cartilage study incorporates themes from Derivative and Regeneration.
His primary areas of investigation include Tumor necrosis factor alpha, Inflammation, Conditional gene knockout, Receptor and Pathology. The various areas that Chuan-ju Liu examines in his Tumor necrosis factor alpha study include TUNEL assay, Ankylosing spondylitis, Mediator, Pharmacology and In vivo. His study in Inflammation is interdisciplinary in nature, drawing from both Extracellular matrix, H&E stain and Intracellular.
His Conditional gene knockout research incorporates elements of Adipose tissue, Internal medicine and Endocrinology. His work carried out in the field of Receptor brings together such families of science as Clinical trial, Bioinformatics, Pathogenesis, Adverse effect and Immune system. His research on Arthritis focuses in particular on Inflammatory arthritis.
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The Growth Factor Progranulin Binds to TNF Receptors and Is Therapeutic Against Inflammatory Arthritis in Mice
Wei Tang;Yi Lu;Yi Lu;Qing-Yun Tian;Yan Zhang.
Science (2011)
Serum interleukin-6 as a marker of periprosthetic infection following total hip and knee arthroplasty.
Paul E. Di Cesare;Eric Chang;Charles F. Preston;Chuan-ju Liu.
Journal of Bone and Joint Surgery, American Volume (2005)
miR-199a, a bone morphogenic protein 2-responsive MicroRNA, regulates chondrogenesis via direct targeting to Smad1.
Edward A. Lin;Li Kong;Xiao-Hui Bai;Yi Luan.
Journal of Biological Chemistry (2009)
ADAMTS-7 Mediates Vascular Smooth Muscle Cell Migration and Neointima Formation in Balloon-Injured Rat Arteries
Li Wang;Jingang Zheng;Xue Bai;Bo Liu.
Circulation Research (2009)
Calmodulin Binds to the C Terminus of Sodium Channels Nav1.4 and Nav1.6 and Differentially Modulates Their Functional Properties
Raimund I. Herzog;Chuanju Liu;Stephen G. Waxman;Theodore R. Cummins.
The Journal of Neuroscience (2003)
Insights into the role of progranulin in immunity, infection, and inflammation
Jinlong Jian;Jessica Konopka;Chuanju Liu.
Journal of Leukocyte Biology (2013)
ADAMTS-7: a metalloproteinase that directly binds to and degrades cartilage oligomeric matrix protein
Chuan-ju Liu;Wei Kong;Kiril Ilalov;Shuang Yu.
The FASEB Journal (2006)
Modulation of the cardiac sodium channel Nav1.5 by fibroblast growth factor homologous factor 1B.
Chuan ju Liu;Chuan ju Liu;Sulayman D. Dib-Hajj;Sulayman D. Dib-Hajj;Muthukrishnan Renganathan;Muthukrishnan Renganathan;Theodore R. Cummins;Theodore R. Cummins.
Journal of Biological Chemistry (2003)
Fibroblast growth factor homologous factor 1B binds to the C terminus of the tetrodotoxin-resistant sodium channel rNav1.9a (NaN)
Chuan-ju Liu;Chuan-ju Liu;Sulayman D. Dib-Hajj;Sulayman D. Dib-Hajj;Stephen G. Waxman;Stephen G. Waxman.
Journal of Biological Chemistry (2001)
Inhibition of ADAMTS-7 and ADAMTS-12 degradation of cartilage oligomeric matrix protein by alpha-2-macroglobulin
Y. Luan;L. Kong;D.R. Howell;K. Ilalov.
Osteoarthritis and Cartilage (2008)
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