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D-Index & Metrics

Biology and Biochemistry

D-Index
69
Citations
16593
World Ranking
7412
National Ranking
186

James S. Wiley publication distribution in Biology and Biochemistry in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Biology and Biochemistry in 2026. The highlighted bar marks where James S. Wiley sits on this spectrum.

47–56 publications: 8 scientists 57–66 publications: 35 scientists 67–76 publications: 106 scientists 77–86 publications: 231 scientists 87–96 publications: 414 scientists 97–106 publications: 546 scientists 107–116 publications: 704 scientists 117–126 publications: 849 scientists 127–136 publications: 980 scientists 137–146 publications: 942 scientists 147–156 publications: 969 scientists 157–166 publications: 950 scientists 167–176 publications: 951 scientists 177–186 publications: 915 scientists 187–196 publications: 787 scientists 197–206 publications: 841 scientists 207–216 publications: 735 scientists 217–226 publications: 709 scientists 227–236 publications: 651 scientists 237–246 publications: 605 scientists 247–256 publications: 510 scientists 257–266 publications: 524 scientists 267–276 publications: 434 scientists 277–286 publications: 418 scientists 287–296 publications: 350 scientists 297–306 publications: 363 scientists 307–316 publications: 315 scientists 317–326 publications: 296 scientists 327–336 publications: 261 scientists 337–346 publications: 240 scientists 347–356 publications: 219 scientists 357–366 publications: 197 scientists 367–376 publications: 154 scientists 377–386 publications: 161 scientists 387–396 publications: 155 scientists 397–406 publications: 145 scientists 407–416 publications: 124 scientists 417–426 publications: 112 scientists 427–436 publications: 132 scientists 437–446 publications: 116 scientists 447–456 publications: 99 scientists 457–466 publications: 81 scientists 467–476 publications: 91 scientists 477–486 publications: 80 scientists 487–496 publications: 80 scientists 497–506 publications: 60 scientists 507–516 publications: 36 scientists 517–526 publications: 46 scientists 527–536 publications: 54 scientists 537–546 publications: 44 scientists 547–556 publications: 43 scientists 557–566 publications: 43 scientists 567–576 publications: 42 scientists 577–586 publications: 25 scientists 587–596 publications: 34 scientists 597–606 publications: 23 scientists 607–616 publications: 33 scientists 617–626 publications: 31 scientists 627–636 publications: 27 scientists 637–646 publications: 25 scientists 647–656 publications: 28 scientists 657–666 publications: 34 scientists 667–676 publications: 18 scientists 677–686 publications: 16 scientists 687–696 publications: 10 scientists 697–706 publications: 12 scientists 707–716 publications: 21 scientists 717–726 publications: 12 scientists 727–736 publications: 12 scientists 737–746 publications: 10 scientists 747–756 publications: 7 scientists 757–766 publications: 13 scientists 767–776 publications: 15 scientists 777–786 publications: 13 scientists 787–796 publications: 9 scientists 797–806 publications: 9 scientists 807–816 publications: 7 scientists 817–826 publications: 4 scientists 827–836 publications: 9 scientists 837–846 publications: 7 scientists 847–856 publications: 3 scientists 857–866 publications: 5 scientists 867–876 publications: 5 scientists 877–886 publications: 11 scientists 887–896 publications: 3 scientists 897–906 publications: 4 scientists 907–916 publications: 7 scientists 917–926 publications: 5 scientists 927–936 publications: 6 scientists 937–946 publications: 6 scientists 947–956 publications: 3 scientists 957–966 publications: 7 scientists 967–976 publications: 2 scientists 977–986 publications: 2 scientists 987–996 publications: 1 scientists 997–1,006 publications: 5 scientists 1,007–1,016 publications: 2 scientists 1,017–1,026 publications: 2 scientists 1,027 publications: 1 scientists 1,028+ publications: 100 scientists
47 publications 1,028+

This scientist: 227 publications — 60th percentile

60% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 1,028 publications or more.

James S. Wiley D-index placement in Biology and Biochemistry in 2026

The chart shows the D-index (discipline H-index) distribution of Biology and Biochemistry scientists ranked by Research.com in 2026. The highlighted bar marks where James S. Wiley sits on this spectrum.

40–41 D-Index: 80 scientists 42–43 D-Index: 183 scientists 44–45 D-Index: 317 scientists 46–47 D-Index: 504 scientists 48–49 D-Index: 718 scientists 50–51 D-Index: 900 scientists 52–53 D-Index: 1,026 scientists 54–55 D-Index: 1,150 scientists 56–57 D-Index: 1,236 scientists 58–59 D-Index: 1,253 scientists 60–61 D-Index: 1,163 scientists 62–63 D-Index: 1,131 scientists 64–65 D-Index: 1,032 scientists 66–67 D-Index: 897 scientists 68–69 D-Index: 814 scientists 70–71 D-Index: 715 scientists 72–73 D-Index: 709 scientists 74–75 D-Index: 596 scientists 76–77 D-Index: 512 scientists 78–79 D-Index: 473 scientists 80–81 D-Index: 412 scientists 82–83 D-Index: 373 scientists 84–85 D-Index: 358 scientists 86–87 D-Index: 285 scientists 88–89 D-Index: 273 scientists 90–91 D-Index: 227 scientists 92–93 D-Index: 208 scientists 94–95 D-Index: 193 scientists 96–97 D-Index: 153 scientists 98–99 D-Index: 157 scientists 100–101 D-Index: 148 scientists 102–103 D-Index: 120 scientists 104–105 D-Index: 113 scientists 106–107 D-Index: 100 scientists 108–109 D-Index: 86 scientists 110–111 D-Index: 67 scientists 112–113 D-Index: 72 scientists 114–115 D-Index: 73 scientists 116–117 D-Index: 64 scientists 118–119 D-Index: 53 scientists 120–121 D-Index: 60 scientists 122–123 D-Index: 54 scientists 124–125 D-Index: 43 scientists 126–127 D-Index: 38 scientists 128–129 D-Index: 49 scientists 130–131 D-Index: 26 scientists 132–133 D-Index: 18 scientists 134–135 D-Index: 23 scientists 136–137 D-Index: 32 scientists 138–139 D-Index: 32 scientists 140–141 D-Index: 27 scientists 142–143 D-Index: 19 scientists 144–145 D-Index: 22 scientists 146–147 D-Index: 12 scientists 148–149 D-Index: 16 scientists 150–151 D-Index: 14 scientists 152–153 D-Index: 10 scientists 154–155 D-Index: 13 scientists 156–157 D-Index: 10 scientists 158–159 D-Index: 7 scientists 160–161 D-Index: 9 scientists 162–163 D-Index: 13 scientists 164–165 D-Index: 4 scientists 166 D-Index: 4 scientists 167+ D-Index: 98 scientists
40 D-Index 167+

This scientist: 69 D-Index — 63rd percentile

63% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 167 D-Index or more.

Overview

James S. Wiley is affiliated with the University of Melbourne in Australia. Their research spans significant areas within medicine and neuroscience, with a focus on neuroinflammation, neurodegeneration mechanisms, and Alzheimer's disease research and treatments. Their work also covers topics related to tryptophan and brain disorders, adenosine and purinergic signaling, and retinal diseases and treatments.

The primary fields of study for Wiley include:

  • Medicine
  • Neuroscience
  • Biochemistry, Genetics and Molecular Biology

Subfields of interest seen in their work are:

  • Physiology
  • Neurology
  • Biological Psychiatry
  • Ophthalmology
  • Genetics

Wiley's prominent research topics include:

  • Neuroinflammation and Neurodegeneration Mechanisms
  • Alzheimer's disease research and treatments
  • Tryptophan and brain disorders
  • Adenosine and Purinergic Signaling
  • Retinal Diseases and Treatments
  • Adolescent and Pediatric Healthcare
  • Retinal Imaging and Analysis

Recent publications attributable to James S. Wiley demonstrate active engagement with neurological and molecular science journals. Notable papers include:

  • "Genomics of Alzheimer's disease implicates the innate and adaptive immune systems," published in 2021 in Cellular and Molecular Life Sciences
  • "Deficits in Monocyte Function in Age Related Macular Degeneration: A Novel Systemic Change Associated With the Disease," published in 2021 in Frontiers in Medicine
  • "A P2RX7 single nucleotide polymorphism haplotype promotes exon 7 and 8 skipping and disrupts receptor function," published in 2020 in The FASEB Journal
  • "Identification of Leukocyte Surface P2X7 as a Biomarker Associated with Alzheimer's Disease," published in 2022 in International Journal of Molecular Sciences
  • "Flow cytometry identifies an early stage of platelet apoptosis produced by agonists of the P2X1 and P2X7 receptors," published in 2022 in Platelets

Among frequent co-authors collaborating with Wiley are:

  • Ben J. Gu
  • Xin Huang
  • Yihan Li
  • Colin L. Masters
  • Christopher Fowler

Wiley's research has been published in several journals with repeated contributions to:

  • Alzheimer s & Dementia
  • Cellular and Molecular Life Sciences
  • International Journal of Molecular Sciences
  • Frontiers in Medicine
  • The FASEB Journal

Best Publications

  • Analysis of immune-related loci identifies 48 new susceptibility variants for multiple sclerosis

    Ashley H. Beecham;Nikolaos A. Patsopoulos;Nikolaos A. Patsopoulos;Dionysia K. Xifara;Mary F. Davis

  • Genome-wide association study identifies new multiple sclerosis susceptibility loci on chromosomes 12 and 20

    Melanie Bahlo;David R Booth;Simon A Broadley;Matthew A Brown;Matthew A Brown

  • Modification of red cell membrane structure by cholesterol-rich lipid dispersions. A model for the primary spur cell defect.

    R A Cooper;E C Arner;J S Wiley;S J Shattil

  • A GLU (496) TO ALA POLYMORPHISM LEADS TO LOSS OF FUNCTION OF THE HUMAN P2X7 RECEPTOR

    Ben J. Gu;Weiyi Zhang;Rebecca A. Worthington;Ronald Sluyter

  • A Furosemide-Sensitive Cotransport of Sodium plus Potassium in the Human Red Cell

    James S. Wiley;Richard A. Cooper

  • Cytosine Arabinoside Influx and Nucleoside Transport Sites in Acute Leukemia

    J. S. Wiley;S. P. Jones;W. H. Sawyer;A. R. P. Paterson

  • A Polymorphism in the P2X7 Gene Increases Susceptibility to Extrapulmonary Tuberculosis

    Suran L. Fernando;Bernadette M. Saunders;Ronald Sluyter;Kristen K. Skarratt

  • Strikingly homologous immunoglobulin gene rearrangements and poor outcome in VH3-21-using chronic lymphocytic leukemia patients independent of geographic origin and mutational status.

    Mia Thorsélius;Alexander Kröber;Fiona Murray;Ulf Thunberg

  • Expression of P2X7 purinoceptors on human lymphocytes and monocytes: evidence for nonfunctional P2X7 receptors

    B. J. Gu;W. Y. Zhang;L. J. Bendall;I. P. Chessell

  • The isoquinoline derivative KN‐62 a potent antagonist of the P2Z‐receptor of human lymphocytes

    Caroline Eve Gargett;James S. Wiley

  • P2X7 Receptor Cell Surface Expression and Cytolytic Pore Formation Are Regulated by a Distal C-terminal Region

    Megan L. Smart;Ben Gu;Rekha G. Panchal;James Wiley

  • The human P2X7 receptor and its role in innate immunity

    J. S. Wiley;R. Sluyter;R. Sluyter;B. J. Gu;B. J. Gu;L. Stokes

  • An Ile-568 to Asn Polymorphism Prevents Normal Trafficking and Function of the Human P2X7 Receptor

    James S. Wiley;Lan-Phuong Dao-Ung;Changping Li;Anne N. Shemon

  • Adenosine Triphosphate–Induced Shedding of CD23 and L-Selectin (CD62L) From Lymphocytes Is Mediated by the Same Receptor but Different Metalloproteases

    B. Gu;L.J. Bendall;J.S. Wiley

  • A Thr357 to Ser Polymorphism in Homozygous and Compound Heterozygous Subjects Causes Absent or Reduced P2X7 Function and Impairs ATP-induced Mycobacterial Killing by Macrophages

    Anne N. Shemon;Ronald Sluyter;Suran L. Fernando;Alison L. Clarke

  • Filamin A mutations cause periventricular heterotopia with Ehlers-Danlos syndrome

    V. L. Sheen;A. Jansen;M. H. Chen;E. Parrini

  • Two haplotypes of the P2X7 receptor containing the Ala-348 to Thr polymorphism exhibit a gain-of-function effect and enhanced interleukin-1β secretion

    Leanne Stokes;Stephen J. Fuller;Ronald Sluyter;Ronald Sluyter;Kristen K. Skarratt

  • A loss-of-function polymorphic mutation in the cytolytic P2X7 receptor gene and chronic lymphocytic leukaemia: a molecular study

    James S Wiley;L Phuong Dao-Ung;Ben J Gu;Ronald Sluyter

  • An Arg307 To Gln Polymorphism Within The Atp-Binding Site Causes Loss Of Function Of The Human P2X7 Receptor.

    Ben J. Gu;Ronald Sluyter;Kristen K. Skarratt;Anne N. Shemon

  • Glu496 to Ala polymorphism in the P2X7 receptor impairs ATP-induced IL-1 beta release from human monocytes.

    Ronald Sluyter;Anne N Shemon;James S Wiley

Frequent Co-Authors

Matthew A. Brown
Matthew A. Brown Guy's and St Thomas' NHS Foundation Trust
Graeme J. Stewart
Graeme J. Stewart University of Sydney
Steven Petrou
Steven Petrou Florey Institute of Neuroscience and Mental Health
Helmut Butzkueven
Helmut Butzkueven Monash University
Pablo Moscato
Pablo Moscato University of Newcastle Australia
Melanie Bahlo
Melanie Bahlo Walter and Eliza Hall Institute of Medical Research
Lyn R. Griffiths
Lyn R. Griffiths Queensland University of Technology
Robyn H. Guymer
Robyn H. Guymer University of Melbourne
David R. Booth
David R. Booth University of Sydney
Warwick J. Britton
Warwick J. Britton University of Sydney

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