Internal medicine, Endocrinology, Adipose tissue, White adipose tissue and Adipocyte are his primary areas of study. His research in Internal medicine intersects with topics in Nuclear receptor and Cell biology. His studies deal with areas such as Estrogen-related receptor gamma and Beta-3 adrenergic receptor as well as Endocrinology.
His study in Adipose tissue focuses on Lipolysis in particular. His research in the fields of Thermogenin overlaps with other disciplines such as Cellular plasticity. James G. Granneman interconnects Steatosis, AMP-activated protein kinase, AMPK, Insulin and Fatty liver in the investigation of issues within Adipocyte.
James G. Granneman focuses on Internal medicine, Endocrinology, Adipose tissue, Cell biology and Adipocyte. His Internal medicine study frequently links to other fields, such as Gene expression. James G. Granneman works mostly in the field of Endocrinology, limiting it down to concerns involving Messenger RNA and, occasionally, Molecular biology and Gs alpha subunit.
The Adipose tissue study combines topics in areas such as Downregulation and upregulation, Mitochondrion and Denervation. In general Cell biology study, his work on Lipid droplet and Kinase often relates to the realm of Population, thereby connecting several areas of interest. James G. Granneman works mostly in the field of Adipocyte, limiting it down to topics relating to Lipolysis and, in certain cases, Intracellular and Organelle.
James G. Granneman mainly investigates Adipose tissue, Cell biology, Adipocyte, Lipolysis and Lipid droplet. His Brown adipose tissue study in the realm of Adipose tissue interacts with subjects such as Metabolic syndrome, Research areas and Group discussion. His study looks at the relationship between Cell biology and topics such as White adipose tissue, which overlap with Adipogenesis and Stromal cell.
His work in Adipocyte tackles topics such as Proinflammatory cytokine which are related to areas like Cancer research, Downregulation and upregulation, Tumor progression, Bone marrow and Adipose tissue macrophages. His Lipid droplet study incorporates themes from Lipid metabolism, Lung, Perilipin and Bioinformatics. In the field of Endocrinology and Internal medicine James G. Granneman studies Carbohydrate metabolism.
His primary areas of investigation include Adipose tissue, Cell biology, Lipolysis, Adipogenesis and White adipose tissue. His study in Adipose tissue is interdisciplinary in nature, drawing from both Lung, Lipid droplet and Disease. His research on Cell biology often connects related topics like Adipose triglyceride lipase.
His Lipolysis research integrates issues from Inflammation, Triglyceride lipase, Fatty acid and Fatty liver. His Adipogenesis research integrates issues from Cell, Cell type, Stromal cell, Adipocyte and Stem cell. His White adipose tissue study combines topics in areas such as Receptor and STAT3.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
In Vivo Identification of Bipotential Adipocyte Progenitors Recruited by β3-Adrenoceptor Activation and High-Fat Feeding
Yun Hee Lee;Anelia P. Petkova;Emilio P. Mottillo;James G. Granneman.
Cell Metabolism (2012)
PER2 controls lipid metabolism by direct regulation of PPARγ
Benedetto Grimaldi;Marina Maria Bellet;Sayako Katada;Giuseppe Astarita.
Cell Metabolism (2010)
Molecular cloning and expression of the rat beta 3-adrenergic receptor.
James G. Granneman;Kristine N. Lahners;Archana Chaudhry.
Molecular Pharmacology (1991)
Perilipin Controls Lipolysis by Regulating the Interactions of AB-hydrolase Containing 5 (Abhd5) and Adipose Triglyceride Lipase (Atgl)
James G. Granneman;Hsiao Ping H Moore;Rukmani Krishnamoorthy;Miloni Rathod.
Journal of Biological Chemistry (2009)
Analysis of Lipolytic Protein Trafficking and Interactions in Adipocytes
James G. Granneman;Hsiao Ping H Moore;Rachel L. Granneman;Andrew S. Greenberg.
Journal of Biological Chemistry (2007)
Metabolic and cellular plasticity in white adipose tissue I: effects of β3-adrenergic receptor activation
James G. Granneman;Pipeng Li;Zhengxian Zhu;Yuyan Lu.
American Journal of Physiology-endocrinology and Metabolism (2005)
Nurr1 enhances transcription of the human dopamine transporter gene through a novel mechanism.
Paola Sacchetti;Todd R. Mitchell;James G. Granneman;Michael J. Bannon.
Journal of Neurochemistry (2001)
PPAR δ agonists stimulate oligodendrocyte differentiation in tissue culture
Inderjeet Saluja;James G. Granneman;Robert P. Skoff.
Glia (2001)
15O PET Measurement of Blood Flow and Oxygen Consumption in Cold-Activated Human Brown Fat
Otto Muzik;Thomas J. Mangner;William R. Leonard;Ajay Kumar.
The Journal of Nuclear Medicine (2013)
Cellular origins of cold-induced brown adipocytes in adult mice
Yun Hee Lee;Anelia P. Petkova;Anish A. Konkar;James G. Granneman.
The FASEB Journal (2015)
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