World's Best Scientists 2026 revealed!
James B. Koprich

James B. Koprich

D-Index & Metrics

Neuroscience

D-Index
37
Citations
4613
World Ranking
8853
National Ranking
137

James B. Koprich publication distribution in Neuroscience in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Neuroscience in 2026. The highlighted bar marks where James B. Koprich sits on this spectrum.

38–47 publications: 18 scientists 48–57 publications: 79 scientists 58–67 publications: 193 scientists 68–77 publications: 323 scientists 78–87 publications: 406 scientists 88–97 publications: 452 scientists 98–107 publications: 539 scientists 108–117 publications: 505 scientists 118–127 publications: 522 scientists 128–137 publications: 469 scientists 138–147 publications: 456 scientists 148–157 publications: 459 scientists 158–167 publications: 397 scientists 168–177 publications: 383 scientists 178–187 publications: 350 scientists 188–197 publications: 302 scientists 198–207 publications: 306 scientists 208–217 publications: 262 scientists 218–227 publications: 242 scientists 228–237 publications: 220 scientists 238–247 publications: 203 scientists 248–257 publications: 174 scientists 258–267 publications: 176 scientists 268–277 publications: 175 scientists 278–287 publications: 125 scientists 288–297 publications: 116 scientists 298–307 publications: 127 scientists 308–317 publications: 128 scientists 318–327 publications: 99 scientists 328–337 publications: 89 scientists 338–347 publications: 78 scientists 348–357 publications: 96 scientists 358–367 publications: 66 scientists 368–377 publications: 59 scientists 378–387 publications: 65 scientists 388–397 publications: 54 scientists 398–407 publications: 48 scientists 408–417 publications: 49 scientists 418–427 publications: 34 scientists 428–437 publications: 31 scientists 438–447 publications: 30 scientists 448–457 publications: 31 scientists 458–467 publications: 36 scientists 468–477 publications: 40 scientists 478–487 publications: 35 scientists 488–497 publications: 30 scientists 498–507 publications: 23 scientists 508–517 publications: 26 scientists 518–527 publications: 20 scientists 528–537 publications: 23 scientists 538–547 publications: 20 scientists 548–557 publications: 20 scientists 558–567 publications: 17 scientists 568–577 publications: 14 scientists 578–587 publications: 20 scientists 588–597 publications: 20 scientists 598–607 publications: 19 scientists 608–617 publications: 18 scientists 618–627 publications: 17 scientists 628–637 publications: 11 scientists 638–647 publications: 11 scientists 648–657 publications: 11 scientists 658–667 publications: 8 scientists 668–677 publications: 7 scientists 678–687 publications: 11 scientists 688–697 publications: 10 scientists 698–707 publications: 4 scientists 708–717 publications: 6 scientists 718–727 publications: 5 scientists 728–737 publications: 5 scientists 738–747 publications: 9 scientists 748–757 publications: 9 scientists 758–767 publications: 3 scientists 768–777 publications: 7 scientists 778–787 publications: 7 scientists 788–797 publications: 6 scientists 798–807 publications: 2 scientists 808–817 publications: 2 scientists 818–827 publications: 7 scientists 828–837 publications: 0 scientists 838–847 publications: 9 scientists 848–857 publications: 3 scientists 858–867 publications: 1 scientists 868–877 publications: 3 scientists 878–886 publications: 6 scientists 887+ publications: 100 scientists
38 publications 887+

This scientist: 76 publications — 6th percentile

6% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 887 publications or more.

James B. Koprich D-index placement in Neuroscience in 2026

The chart shows the D-index (discipline H-index) distribution of Neuroscience scientists ranked by Research.com in 2026. The highlighted bar marks where James B. Koprich sits on this spectrum.

30–31 D-Index: 42 scientists 32–33 D-Index: 172 scientists 34–35 D-Index: 296 scientists 36–37 D-Index: 435 scientists 38–39 D-Index: 459 scientists 40–41 D-Index: 456 scientists 42–43 D-Index: 467 scientists 44–45 D-Index: 478 scientists 46–47 D-Index: 512 scientists 48–49 D-Index: 435 scientists 50–51 D-Index: 425 scientists 52–53 D-Index: 418 scientists 54–55 D-Index: 392 scientists 56–57 D-Index: 357 scientists 58–59 D-Index: 334 scientists 60–61 D-Index: 328 scientists 62–63 D-Index: 260 scientists 64–65 D-Index: 278 scientists 66–67 D-Index: 239 scientists 68–69 D-Index: 250 scientists 70–71 D-Index: 210 scientists 72–73 D-Index: 200 scientists 74–75 D-Index: 189 scientists 76–77 D-Index: 170 scientists 78–79 D-Index: 146 scientists 80–81 D-Index: 113 scientists 82–83 D-Index: 126 scientists 84–85 D-Index: 100 scientists 86–87 D-Index: 84 scientists 88–89 D-Index: 99 scientists 90–91 D-Index: 84 scientists 92–93 D-Index: 85 scientists 94–95 D-Index: 72 scientists 96–97 D-Index: 76 scientists 98–99 D-Index: 45 scientists 100–101 D-Index: 49 scientists 102–103 D-Index: 43 scientists 104–105 D-Index: 32 scientists 106–107 D-Index: 45 scientists 108–109 D-Index: 50 scientists 110–111 D-Index: 32 scientists 112–113 D-Index: 39 scientists 114–115 D-Index: 32 scientists 116–117 D-Index: 29 scientists 118–119 D-Index: 27 scientists 120–121 D-Index: 19 scientists 122–123 D-Index: 23 scientists 124–125 D-Index: 27 scientists 126–127 D-Index: 16 scientists 128–129 D-Index: 24 scientists 130–131 D-Index: 13 scientists 132–133 D-Index: 21 scientists 134–135 D-Index: 17 scientists 136–137 D-Index: 14 scientists 138–139 D-Index: 15 scientists 140–141 D-Index: 10 scientists 142–143 D-Index: 10 scientists 144–145 D-Index: 13 scientists 146–147 D-Index: 9 scientists 148–149 D-Index: 8 scientists 150–151 D-Index: 6 scientists 152–153 D-Index: 6 scientists 154–155 D-Index: 7 scientists 156–157 D-Index: 7 scientists 158–159 D-Index: 10 scientists 160–161 D-Index: 4 scientists 162 D-Index: 8 scientists 163+ D-Index: 100 scientists
30 D-Index 163+

This scientist: 37 D-Index — 10th percentile

10% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 163 D-Index or more.

Overview

James B. Koprich is affiliated with Fudan University in China and focuses their research primarily within the fields of Medicine and Neuroscience. Their work encompasses several subfields including Neurology, Cellular and Molecular Neuroscience, Molecular Biology, Epidemiology, and Cell Biology.

The main topics addressed in their research include:

  • Parkinson's Disease Mechanisms and Treatments
  • Neurological disorders and treatments
  • Nerve injury and regeneration
  • Neuroinflammation and Neurodegeneration Mechanisms
  • Nuclear Receptors and Signaling
  • Cellular transport and secretion
  • RNA regulation and disease

James B. Koprich has published extensively, with notable recent papers as follows:

  • "Early-onset impairment of the ubiquitin-proteasome system in dopaminergic neurons caused by α-synuclein," 2020, Acta Neuropathologica Communications
  • "Neurodegeneration by α-synuclein-specific T cells in AAV-A53T-α-synuclein Parkinson's disease mice," 2022, Brain Behavior and Immunity
  • "The Promise and Challenges of Developing miRNA-Based Therapeutics for Parkinson's Disease," 2020, Cells
  • "Propagated α-synucleinopathy recapitulates REM sleep behaviour disorder followed by parkinsonian phenotypes in mice," 2020, Brain
  • "Neuroprotection of Exendin-4 by Enhanced Autophagy in a Parkinsonian Rat Model of α-Synucleinopathy," 2021, Neurotherapeutics

Their frequent co-authors include:

  • Jonathan M. Brotchie
  • Chi Wang Ip
  • Jens Volkmann
  • Rhonda L. McFleder
  • Thomas Musacchio

James B. Koprich's research contributions have been published predominantly in the following venues:

  • Brain Behavior and Immunity
  • Clinical Neurophysiology
  • Brain
  • Journal of Neuroinflammation
  • npj Parkinson s Disease

Best Publications

  • Dynamic Changes in Presynaptic and Axonal Transport Proteins Combined with Striatal Neuroinflammation Precede Dopaminergic Neuronal Loss in a Rat Model of AAV α-Synucleinopathy

    Chee Yeun Chung;James B. Koprich;Hasan Siddiqi;Ole Isacson

  • Neuroinflammation mediated by IL-1β increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson's disease

    James B Koprich;Casper Reske-Nielsen;Prabhakar Mithal;Ole Stefan Isacson

  • The Pharmacology of l-DOPA-Induced Dyskinesia in Parkinson’s Disease

    Philippe Huot;Tom H. Johnston;James B. Koprich;Susan H. Fox

  • Animal models of α-synucleinopathy for Parkinson disease drug development

    James B. Koprich;Lorraine V. Kalia;Jonathan M. Brotchie

  • AAV1/2-induced overexpression of A53T-α-synuclein in the substantia nigra results in degeneration of the nigrostriatal system with Lewy-like pathology and motor impairment: a new mouse model for Parkinson’s disease

    Chi Wang Ip;Laura-Christin Klaus;Akua A. Karikari;Naomi P. Visanji

  • Expression of human A53T alpha-synuclein in the rat substantia nigra using a novel AAV1/2 vector produces a rapidly evolving pathology with protein aggregation, dystrophic neurite architecture and nigrostriatal degeneration with potential to model the pathology of Parkinson's disease

    James B Koprich;Tom H Johnston;M Gabriela Reyes;Xuan Sun

  • α-Synuclein-Based Animal Models of Parkinson's Disease: Challenges and Opportunities in a New Era

    Naomi P. Visanji;Jonathan M. Brotchie;Lorraine V. Kalia;James B. Koprich

  • Treatment with Trehalose Prevents Behavioral and Neurochemical Deficits Produced in an AAV α-Synuclein Rat Model of Parkinson’s Disease

    Qing He;Qing He;James B. Koprich;Ying Wang;Wen-bo Yu

  • Neurodegeneration by α-synuclein-specific T cells in AAV-A53T-α-synuclein Parkinson’s disease mice

    Unknown

  • Early-onset impairment of the ubiquitin-proteasome system in dopaminergic neurons caused by α-synuclein

    Chris McKinnon;Mitchell L. De Snoo;Elise Gondard;Clemens Neudorfer

  • The Toll-Like Receptor-3 Agonist Polyinosinic:Polycytidylic Acid Triggers Nigrostriatal Dopaminergic Degeneration

    Michela Deleidi;Penelope J. Hallett;James B. Koprich;Chee-Yeun Chung

  • Progressive neurodegeneration or endogenous compensation in an animal model of Parkinson's disease produced by decreasing doses of alpha-synuclein.

    James B. Koprich;Tom H. Johnston;Philippe Huot;M. Gabriela Reyes

  • Characterization of 3,4-methylenedioxymethamphetamine (MDMA) enantiomers in vitro and in the MPTP-lesioned primate: R-MDMA reduces severity of dyskinesia, whereas S-MDMA extends duration of ON-time.

    P. Huot;T.H. Johnston;Katie Lewis;J.B. Koprich

  • Subthalamic nucleus deep brain stimulation is neuroprotective in the A53T α‐synuclein Parkinson's disease rat model

    Thomas Musacchio;Maike Rebenstorff;Felix Fluri;Jonathan M. Brotchie

  • Nigrostriatal Dysfunction in Familial Alzheimer's Disease-Linked APPswe/PS1ΔE9 Transgenic Mice

    Sylvia E. Perez;Orly Lazarov;James B. Koprich;Er Yun Chen

  • The selective mu-opioid receptor antagonist ADL5510 reduces levodopa-induced dyskinesia without affecting antiparkinsonian action in MPTP-lesioned macaque model of Parkinson's disease.

    James B. Koprich;Susan H. Fox;Tom H. Johnston;Allan Goodman

  • Prenatal 3,4-methylenedioxymethamphetamine (ecstasy) alters exploratory behavior, reduces monoamine metabolism, and increases forebrain tyrosine hydroxylase fiber density of juvenile rats.

    James B. Koprich;Er Yun Chen;Nicholas M. Kanaan;Nicholas G. Campbell

  • Neonatal 3,4-methylenedioxymethamphetamine (ecstasy) alters dopamine and serotonin neurochemistry and increases brain-derived neurotrophic factor in the forebrain and brainstem of the rat.

    James B Koprich;Nicholas G Campbell;Jack W Lipton

  • Propagated α-synucleinopathy recapitulates REM sleep behaviour disorder followed by parkinsonian phenotypes in mice.

    Yan Shen;Wen-Bo Yu;Bo Shen;Hui Dong

  • The Promise and Challenges of Developing miRNA-Based Therapeutics for Parkinson's Disease.

    Simoneide S. Titze-de-Almeida;Cristina Soto-Sánchez;Eduardo Fernandez;James B. Koprich

  • UWA-121, a mixed dopamine and serotonin re-uptake inhibitor, enhances L-DOPA anti-parkinsonian action without worsening dyskinesia or psychosis-like behaviours in the MPTP-lesioned common marmoset.

    Philippe Huot;Tom H. Johnston;Katie D. Lewis;James B. Koprich

  • A53T-α-synuclein overexpression in murine locus coeruleus induces Parkinson’s disease-like pathology in neurons and glia

    Martin Timo Henrich;Fanni Fruzsina Geibl;Bolam Lee;Wei-Hua Chiu

  • Response of aged parkinsonian monkeys to in vivo gene transfer of GDNF.

    M.E. Emborg;J. Moirano;J. Raschke;V. Bondarenko

Frequent Co-Authors

Jonathan M. Brotchie
Jonathan M. Brotchie University Health Network
Jack W. Lipton
Jack W. Lipton Michigan State University
Ole Isacson
Ole Isacson Harvard University
Andrew B. West
Andrew B. West Duke University
Jeffrey H. Kordower
Jeffrey H. Kordower Arizona State University
Menachem Hanani
Menachem Hanani Hebrew University of Jerusalem
Elliott J. Mufson
Elliott J. Mufson Barrow Neurological Institute
Sylvia E. Perez
Sylvia E. Perez Barrow Neurological Institute
Anthony E. Lang
Anthony E. Lang University of Toronto
Jacqueline N. Crawley
Jacqueline N. Crawley University of California, Davis

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