Ian P. Hall mainly focuses on Internal medicine, Immunology, Asthma, Genome-wide association study and Endocrinology. His Internal medicine study combines topics in areas such as Genotype, Haplotype and Placebo. His Immunology study incorporates themes from Idiopathic pulmonary fibrosis, Allele and Oncology.
His Asthma study combines topics from a wide range of disciplines, such as Cohort, Genetic association, Allergy and Genetic predisposition. His Genome-wide association study study incorporates themes from COPD, Lung, Pulmonary function testing and FEV1/FVC ratio. His research in Endocrinology intersects with topics in Agonist, Receptor, Ion channel and Bronchodilator.
The scientist’s investigation covers issues in Internal medicine, Asthma, Immunology, Endocrinology and Genome-wide association study. His Internal medicine research includes elements of Gastroenterology and Oncology. In his study, Corticosteroid and Anesthesia is strongly linked to Placebo, which falls under the umbrella field of Asthma.
He has researched Immunology in several fields, including Lung and Allele. His studies in Endocrinology integrate themes in fields like Agonist, Receptor, Inositol phosphate and Cell biology. His studies deal with areas such as COPD, Genetic association and FEV1/FVC ratio as well as Genome-wide association study.
His main research concerns Internal medicine, Genome-wide association study, COPD, Asthma and Immunology. His research integrates issues of Endocrinology and Oncology in his study of Internal medicine. His Genome-wide association study research is multidisciplinary, relying on both Lung, Genetic association and MAD1L1.
In his study, which falls under the umbrella issue of COPD, Bioinformatics and Pulmonary disease is strongly linked to Lung function. The various areas that Ian P. Hall examines in his Asthma study include Biobank and Locus. In general Immunology study, his work on Cytokine often relates to the realm of Expression, thereby connecting several areas of interest.
His scientific interests lie mostly in Genome-wide association study, COPD, Internal medicine, Asthma and Lung. His biological study spans a wide range of topics, including Respiratory system, FEV1/FVC ratio and Medical genetics. He combines subjects such as Genetic association and Bioinformatics with his study of COPD.
His Internal medicine research is multidisciplinary, incorporating perspectives in Gastroenterology and CpG site. His Asthma study is concerned with the field of Immunology as a whole. The concepts of his Immunology study are interwoven with issues in Phenotype, Pulmonary fibrosis, Genetic predisposition and Locus.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Sequence variants affecting eosinophil numbers associate with asthma and myocardial infarction.
Daniel F Gudbjartsson;Unnur S Bjornsdottir;Unnur S Bjornsdottir;Eva Halapi;Anna Helgadottir.
Nature Genetics (2009)
Genome-wide association study identifies five loci associated with lung function.
E Repapi;I Sayers;L V Wain;P R Burton.
Nature Genetics (2010)
Influence of beta 2-adrenergic receptor genotypes on signal transduction in human airway smooth muscle cells.
S A Green;J Turki;P Bejarano;I P Hall.
American Journal of Respiratory Cell and Molecular Biology (1995)
Association between β2-adrenoceptor polymorphism and susceptibility to bronchodilator desensitisation in moderately severe stable asthmatics
Soong Tan;Ian P Hall;Jane Dewar;Eleanor Dow.
The Lancet (1997)
Systematic Review and Meta-Analysis of the Association between β2-Adrenoceptor Polymorphisms and Asthma: A HuGE Review
Ammarin Thakkinstian;Mark McEvoy;Cosetta Minelli;Peter Gibson.
American Journal of Epidemiology (2005)
Genome-wide association and large-scale follow up identifies 16 new loci influencing lung function
María Soler Artigas;Daan W. Loth;Louise V. Wain;Sina A. Gharib.
Nature Genetics (2011)
Association of Glu 27 β2-adrenoceptor polymorphism with lower airway reactivity in asthmatic subjects
I.P Hall;A Wheatley;P Wilding;S.B Liggett.
The Lancet (1995)
IL-33 is more potent than IL-25 in provoking IL-13–producing nuocytes (type 2 innate lymphoid cells) and airway contraction
Jillian L. Barlow;Samantha Peel;Jane Fox;Veera Panova.
The Journal of Allergy and Clinical Immunology (2013)
Novel insights into the genetics of smoking behaviour, lung function, and chronic obstructive pulmonary disease (UK BiLEVE): a genetic association study in UK Biobank
Louise V Wain;Nick Shrine;Suzanne Miller;Victoria E Jackson.
The Lancet Respiratory Medicine (2015)
Beta-adrenergic agonists regulate KCa channels in airway smooth muscle by cAMP-dependent and -independent mechanisms.
H Kume;I P Hall;R J Washabau;K Takagi.
Journal of Clinical Investigation (1994)
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