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Neuroscience

D-Index
56
Citations
11725
World Ranking
4513
National Ranking
2040

Overview

Christine Konradi is affiliated with Vanderbilt University in the United States. Their research primarily spans the fields of Neuroscience and Biochemistry, Genetics and Molecular Biology, with notable focus on Cellular and Molecular Neuroscience, Cognitive Neuroscience, and Psychiatry and Mental Health.

The scientist has contributed to important topics such as Neuroscience and Neuropharmacology Research, Memory and Neural Mechanisms, Genetics and Neurodevelopmental Disorders, Neuroendocrine Regulation and Behavior, Neuroinflammation and Neurodegeneration Mechanisms, Neuroscience and Neural Engineering, as well as Nerve Injury and Regeneration.

Christine Konradi's recent papers include:

  • Parvalbumin interneuron vulnerability and brain disorders, 2020, Neuropsychopharmacology
  • Hippocampal volume and hippocampal neuron density, number and size in schizophrenia: a systematic review and meta-analysis of postmortem studies, 2020, Molecular Psychiatry
  • Neurotransmitter release progressively desynchronizes in induced human neurons during synapse maturation and aging, 2023, Cell Reports
  • Postmortem Studies of Hippocampal Subfields in Schizophrenia: Systematic Review and Meta-Analysis, 2020, Biological Psychiatry
  • Animal Model Reveals Mechanism of Hippocampal Hyperactivity in Psychosis, 2023, Schizophrenia Bulletin

Frequent co-authors in their work include Stephan Heckers, Maxwell J. Roeske, Alan S. Lewis, Jacob B. Ruden, and Laura L. Dugan.

Christine Konradi has published in venues such as Neuropsychopharmacology, Molecular Psychiatry, Cell Reports, Biological Psychiatry, and Schizophrenia Bulletin.

Best Publications

  • Altered Responsiveness to Cocaine and Increased Immobility in the Forced Swim Test Associated with Elevated cAMP Response Element-Binding Protein Expression in Nucleus Accumbens

    Andrea M. Pliakas;Richard R. Carlson;Rachael L. Neve;Christine Konradi

  • Molecular evidence for mitochondrial dysfunction in bipolar disorder.

    Christine Konradi;Molly Eaton;Matthew L. MacDonald;John Walsh

  • Mitochondrial dysfunction and pathology in bipolar disorder and schizophrenia

    Hayley B. Clay;Stephanie Sillivan;Christine Konradi

  • Molecular aspects of glutamate dysregulation: implications for schizophrenia and its treatment

    Christine Konradi;Stephan Heckers;Stephan Heckers

  • AMPHETAMINE REGULATES GENE EXPRESSION IN RAT STRIATUM VIA TRANSCRIPTION FACTOR CREB

    C Konradi;RL Cole;S Heckers;SE Hyman

  • Neuronal adaptation to amphetamine and dopamine: Molecular mechanisms of prodynorphin gene regulation in rat striatum

    Rebecca L Cole;Christine Konradi;James Douglass;Steven E Hyman

  • Amphetamine and Dopamine-Induced Immediate Early Gene Expression in Striatal Neurons Depends on Postsynaptic NMDA Receptors and Calcium

    Christine Konradi;Jean-Christophe Leveque;Steven E. Hyman

  • GABAergic mechanisms of hippocampal hyperactivity in schizophrenia

    Stephan Heckers;Christine Konradi

  • Spatiotemporal pattern of striatal ERK1/2 phosphorylation in a rat model of L-DOPA-induced dyskinesia and the role of dopamine D1 receptors.

    Jenny E. Westin;Linda Vercammen;Linda Vercammen;Elissa M. Strome;Christine Konradi

  • Hippocampal interneurons are abnormal in schizophrenia.

    Christine Konradi;C. Kevin Yang;Eric I. Zimmerman;Kathryn M. Lohmann

  • Hippocampal neurons in schizophrenia.

    S. Heckers;C. Konradi

  • Maladaptive striatal plasticity in l-DOPA-induced dyskinesia

    M. Angela Cenci;Christine Konradi

  • Hippocampal pathology in schizophrenia.

    Stephan Heckers;Christine Konradi

  • Antipsychotic drugs and neuroplasticity: insights into the treatment and neurobiology of schizophrenia.

    Christine Konradi;Christine Konradi;Stephan Heckers;Stephan Heckers

  • L-Type Ca2+ Channels Are Essential for Glutamate-Mediated CREB Phosphorylation and c-fos Gene Expression in Striatal Neurons

    Anjali Rajadhyaksha;Amy Barczak;Wendy Macı́as;Jean-Christophe Leveque

  • The cAMP-response-element-binding protein interacts, but Fos protein does not interact, with the proenkephalin enhancer in rat striatum.

    Christine Konradi;Linda A. Kobierski;Tuong V. Nguyen;Stephan Heckers

  • Dopamine D2‐like antagonists induce chromatin remodeling in striatal neurons through cyclic AMP‐protein kinase A and NMDA receptor signaling

    Jianhong Li;Yin Guo;Frederick Albert Schroeder;Rachael M. Youngs

  • Dopamine D1 receptors mediate CREB phosphorylation via phosphorylation of the NMDA receptor at Ser897-NR1.

    Joshua T. Dudman;Molly E. Eaton;Anjali Rajadhyaksha;Anjali Rajadhyaksha;Wendy Macías

  • Parvalbumin interneuron vulnerability and brain disorders

    Jacob B. Ruden;Laura L. Dugan;Laura L. Dugan;Christine Konradi

  • Drug-induced activation of dopamine D(1) receptor signaling and inhibition of class I/II histone deacetylase induce chromatin remodeling in reward circuitry and modulate cocaine-related behaviors

    Frederick Albert Schroeder;Krista L. Penta;Anouch Matevossian;Sara R. Jones

  • Intracellular modulation of NMDA receptor function by antipsychotic drugs.

    Jean Christophe Leveque;Wendy Macías;Anjali Rajadhyaksha;Richard R. Carlson

Frequent Co-Authors

Stephan Heckers
Stephan Heckers Vanderbilt University Medical Center
Steven E. Hyman
Steven E. Hyman Harvard University
William A. Carlezon
William A. Carlezon Harvard University
M. Angela Cenci
M. Angela Cenci Lund University
Sabina Berretta
Sabina Berretta Harvard University
Joseph T. Coyle
Joseph T. Coyle Harvard University
Joshua T. Dudman
Joshua T. Dudman Howard Hughes Medical Institute
Moussa B.H. Youdim
Moussa B.H. Youdim Technion – Israel Institute of Technology
Xandra O. Breakefield
Xandra O. Breakefield Harvard University
Laurie J. Ozelius
Laurie J. Ozelius Harvard University

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