D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 43 Citations 8,478 73 World Ranking 4149 National Ranking 1861

Research.com Recognitions

Awards & Achievements

2011 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Apoptosis
  • Neuron

Bruce D. Carter spends much of his time researching Neurotrophin, Cell biology, Tropomyosin receptor kinase A, Low-affinity nerve growth factor receptor and Nerve growth factor. In his papers, he integrates diverse fields, such as Neurotrophin and Cognitive science. Bruce D. Carter has researched Tropomyosin receptor kinase A in several fields, including Neurotrophic factors, Tropomyosin receptor kinase B, Endocrinology and Receptor tyrosine kinase.

Receptor covers he research in Low-affinity nerve growth factor receptor. His Nerve growth factor research includes elements of Cancer research and Small hairpin RNA. In his study, Transcription factor is inextricably linked to Neuroscience, which falls within the broad field of Signal transduction.

His most cited work include:

  • Death of oligodendrocytes mediated by the interaction of nerve growth factor with its receptor p75 (714 citations)
  • Selective activation of NF-kappa B by nerve growth factor through the neurotrophin receptor p75. (619 citations)
  • Neuronal apoptosis linked to EglN3 prolyl hydroxylase and familial pheochromocytoma genes: developmental culling and cancer. (449 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Cell biology, Neurotrophin, Low-affinity nerve growth factor receptor, Receptor and Neuroscience. His work on Cell biology is being expanded to include thematically relevant topics such as Molecular biology. His Neurotrophin research is multidisciplinary, incorporating perspectives in Neurotrophic factors, Tropomyosin receptor kinase B and Nerve growth factor.

His Low-affinity nerve growth factor receptor research incorporates elements of Nuclear localization sequence, Ubiquitin, Growth factor receptor and Ceramide. His work deals with themes such as Cancer research, Apoptosis, Programmed cell death, Mutant and Signal transduction, which intersect with Receptor. Bruce D. Carter has included themes like Endocrinology and Receptor tyrosine kinase in his Tropomyosin receptor kinase A study.

He most often published in these fields:

  • Cell biology (61.18%)
  • Neurotrophin (35.29%)
  • Low-affinity nerve growth factor receptor (36.47%)

What were the highlights of his more recent work (between 2016-2021)?

  • Cell biology (61.18%)
  • Neurotrophin (35.29%)
  • Low-affinity nerve growth factor receptor (36.47%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Cell biology, Neurotrophin, Low-affinity nerve growth factor receptor, Endoplasmic reticulum and Peripheral myelin protein 22. His Cell biology study combines topics from a wide range of disciplines, such as Receptor, Knockout mouse and Somatosensory system. Bruce D. Carter interconnects Intestinal mucosa and Transgene in the investigation of issues within Receptor.

His work on Neurotrophin binding as part of general Neurotrophin research is frequently linked to Sympathetic nervous system, thereby connecting diverse disciplines of science. The various areas that Bruce D. Carter examines in his Low-affinity nerve growth factor receptor study include Axon and Trk receptor. His research integrates issues of Neurotrophic factors, Neural development and Signal transduction in his study of Neuroscience.

Between 2016 and 2021, his most popular works were:

  • A Chimeric Egfr Protein Reporter Mouse Reveals Egfr Localization and Trafficking In Vivo (19 citations)
  • Retrograde Degenerative Signaling Mediated by the p75 Neurotrophin Receptor Requires p150Glued Deacetylation by Axonal HDAC1 (16 citations)
  • Myelin-Associated Glycoprotein Inhibits Schwann Cell Migration and Induces Their Death. (15 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Apoptosis
  • Neuron

His primary areas of investigation include Cell biology, Axoplasmic transport, Axon, Low-affinity nerve growth factor receptor and Endocytic cycle. His Cell biology study incorporates themes from Internalization and Neuroscience. Internalization is the subject of his research, which falls under Receptor.

He has researched Receptor in several fields, including Epithelium and Cell. His studies in Neuroscience integrate themes in fields like Schwann cell, Schwann cell migration, Myelin-associated glycoprotein and Remyelination. His Endocytic cycle study integrates concerns from other disciplines, such as Neuronal Transmission, Neuron, Oncolytic virus and Pinocytosis.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Death of oligodendrocytes mediated by the interaction of nerve growth factor with its receptor p75

Patrizia Casaccia-Bonnefil;Bruce D. Carter;Rick T. Dobrowsky;Moses V. Chao.
Nature (1996)

984 Citations

Selective Activation of NF-κB by Nerve Growth Factor Through the Neurotrophin Receptor p75

Bruce D. Carter;Christian Kaltschmidt;Barbara Kaltschmidt;Nina Offenhäuser.
Science (1996)

870 Citations

Neuronal apoptosis linked to EglN3 prolyl hydroxylase and familial pheochromocytoma genes: developmental culling and cancer.

Sungwoo Lee;Eijiro Nakamura;Haifeng Yang;Wenyi Wei.
Cancer Cell (2005)

615 Citations

Competitive Signaling Between TrkA and p75 Nerve Growth Factor Receptors Determines Cell Survival

Sung Ok Yoon;Patrizia Casaccia-Bonnefil;Bruce Carter;Moses V. Chao.
The Journal of Neuroscience (1998)

613 Citations

The kinesin KIF1Bβ acts downstream from EglN3 to induce apoptosis and is a potential 1p36 tumor suppressor

Susanne Schlisio;Rajappa S. Kenchappa;Liesbeth C.W. Vredeveld;Rani E. George.
Genes & Development (2008)

351 Citations

Selective binding and internalisation by truncated receptors restrict the availability of BDNF during development

Stefano Biffo;Nina Offenhäuser;Bruce D. Carter;Yves-Alain Barde.
Development (1995)

338 Citations

Neurotrophins Live or Let Die: Does p75NTR Decide?

Bruce D. Carter;Gary R. Lewin.
Neuron (1997)

276 Citations

THE ZINC FINGER PROTEIN NRIF INTERACTS WITH THE NEUROTROPHIN RECEPTOR P75NTR AND PARTICIPATES IN PROGRAMMED CELL DEATH

Elisabeth Casademunt;Bruce D. Carter;Isabel Benzel;José M. Frade.
The EMBO Journal (1999)

235 Citations

Ligand-dependent cleavage of the P75 neurotrophin receptor is necessary for NRIF nuclear translocation and apoptosis in sympathetic neurons.

Rajappa S. Kenchappa;Niccolò Zampieri;Moses V. Chao;Philip A. Barker.
Neuron (2006)

223 Citations

Neurotrophin receptors: mediators of life and death.

Moses Chao;Patrizia Casaccia-Bonnefil;Bruce Carter;Alexandra Chittka.
Brain Research Reviews (1998)

220 Citations

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