What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Neuron
Gary R. Lewin focuses on Neuroscience, Sensory system, Nociceptor, Neurotrophin and Nerve growth factor.
His biological study spans a wide range of topics, including Synaptic plasticity and Trk receptor.
His work on Sensory neuron as part of general Sensory system research is frequently linked to Merkel nerve ending, thereby connecting diverse disciplines of science.
His Sensory neuron study incorporates themes from Anatomy and Cell biology.
His Nociceptor study results in a more complete grasp of Nociception.
His Nerve growth factor research is multidisciplinary, incorporating perspectives in Endocrinology, Hyperalgesia and Neurotrophin-3.
His most cited work include:
- PHYSIOLOGY OF THE NEUROTROPHINS (1767 citations)
- Severe neuropathies in mice with targeted mutations in the ErbB3 receptor (622 citations)
- Nerve growth factor-induced hyperalgesia in the neonatal and adult rat (522 citations)
What are the main themes of his work throughout his whole career to date?
Gary R. Lewin mainly investigates Neuroscience, Sensory system, Nociceptor, Nociception and Mechanotransduction.
He combines subjects such as Neurotrophic factors, Neurotrophin, Mechanosensitive channels and Nerve growth factor with his study of Neuroscience.
In his research, Mechanoreceptor and Spinal cord is intimately related to Anatomy, which falls under the overarching field of Sensory system.
His Nociceptor study combines topics from a wide range of disciplines, such as Anesthesia, Transduction, Hyperalgesia and TRPV1.
Gary R. Lewin works mostly in the field of Nociception, limiting it down to concerns involving Naked mole-rat and, occasionally, Biochemistry.
His work carried out in the field of Mechanotransduction brings together such families of science as Nerve injury and Ion channel.
He most often published in these fields:
- Neuroscience (53.85%)
- Sensory system (45.13%)
- Nociceptor (27.18%)
What were the highlights of his more recent work (between 2017-2021)?
- Sensory system (45.13%)
- Neuroscience (53.85%)
- Mechanotransduction (20.00%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Sensory system, Neuroscience, Mechanotransduction, Nociception and Mechanosensitive channels.
Gary R. Lewin does research in Sensory system, focusing on Mechanoreceptor specifically.
His research on Neuroscience frequently links to adjacent areas such as Transduction.
His work deals with themes such as Cell, Cell adhesion and Cartilage, which intersect with Mechanotransduction.
His studies deal with areas such as Neuropathic pain, Stimulation, Mechanosensation and Nerve growth factor as well as Nociception.
His Mechanosensitive channels research includes themes of Stimulus and Sural nerve.
Between 2017 and 2021, his most popular works were:
- The mechanosensitive ion channel Piezo2 mediates sensitivity to mechanical pain in mice (84 citations)
- The mechanosensitive ion channel Piezo2 mediates sensitivity to mechanical pain in mice (84 citations)
- Immune or Genetic-Mediated Disruption of CASPR2 Causes Pain Hypersensitivity Due to Enhanced Primary Afferent Excitability. (66 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Neuron
Gary R. Lewin mostly deals with Neuroscience, Sensory system, Nociception, Mechanotransduction and Transient receptor potential channel.
His Neuroscience research integrates issues from Mechanosensitive ion channel and Allodynia.
His research in Sensory system intersects with topics in Neuropathic pain and Perception.
His Nociception research incorporates themes from Mechanosensation, Sensory neuron, Immune system, Neuron and Dorsal root ganglion.
His research in Mechanotransduction intersects with topics in Ion transporter, PIEZO1, Membrane potential, Ion channel and Patch clamp.
His work deals with themes such as Transduction, Nociceptor, ANT, Threshold of pain and Pharmacology, which intersect with Transient receptor potential channel.
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