His primary areas of study are Genetics, Cell biology, Histone code, Histone and Histone methyltransferase. His work on Mitosis as part of general Cell biology research is often related to Genome instability, thus linking different fields of science. In his study, Bing Li carries out multidisciplinary Histone code and Histone methylation research.
The study incorporates disciplines such as Essential gene, Mutant and X chromosome, Dosage compensation in addition to Histone. His study in Histone methyltransferase is interdisciplinary in nature, drawing from both Histone H3, RNA polymerase II, Transcriptional regulation and Histone deacetylase complex. Bing Li combines subjects such as Chromatin remodeling and Histone octamer with his study of Histone H1.
Bing Li mainly focuses on Cell biology, Genetics, Chromatin, Histone and Nucleosome. His Cell biology research is multidisciplinary, relying on both Spindle checkpoint, Chromatin remodeling, Mad2 and Histone code. His Chromatin remodeling research is multidisciplinary, incorporating perspectives in Histone H2A and In vitro.
His Histone code research incorporates themes from Histone H3 and Histone H1. Bing Li carries out multidisciplinary research, doing studies in Histone H1 and Histone methylation. His research in Chromatin intersects with topics in Molecular biology and Epigenetics.
Bing Li mostly deals with Cell biology, Spindle checkpoint, Chromatin, Histone and Chromatin remodeling. His work deals with themes such as Anaphase-promoting complex, Histone H3, Genetics and Histone code, which intersect with Cell biology. Bing Li merges many fields, such as Histone code and Histone methylation, in his writings.
Bing Li usually deals with Spindle checkpoint and limits it to topics linked to Mad2 and Mitotic checkpoint complex and BUB3. His research on Chromatin focuses in particular on Nucleosome. His Histone H2A research is multidisciplinary, incorporating elements of Histone deacetylase complex and Histone H1.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The Role of Chromatin during Transcription
Bing Li;Michael Carey;Jerry L. Workman.
Histone H3 methylation by Set2 directs deacetylation of coding regions by Rpd3S to suppress spurious intragenic transcription.
Michael J. Carrozza;Bing Li;Laurence Florens;Tamaki Suganuma.
Acetyl-CoA Induces Cell Growth and Proliferation by Promoting the Acetylation of Histones at Growth Genes
Ling Cai;Benjamin M. Sutter;Bing Li;Benjamin P. Tu.
Molecular Cell (2011)
Readers of histone modifications
Miyong Yun;Jun Wu;Jerry L Workman;Bing Li.
Cell Research (2011)
Mad2-Independent Inhibition of APCCdc20 by the Mitotic Checkpoint Protein BubR1
Zhanyun Tang;Rajnish Bharadwaj;Bing Li;Hongtao Yu.
Developmental Cell (2001)
SIRT2 Maintains Genome Integrity and Suppresses Tumorigenesis through Regulating APC/C Activity
Hyun Seok Kim;Athanassios Vassilopoulos;Rui Hong Wang;Tyler Lahusen.
Cancer Cell (2011)
The Set2 histone methyltransferase functions through the phosphorylated carboxyl-terminal domain of RNA polymerase II.
Bing Li;LeAnn Howe;Scott Anderson;John R. Yates.
Journal of Biological Chemistry (2003)
Preferential occupancy of histone variant H2AZ at inactive promoters influences local histone modifications and chromatin remodeling
Bing Li;Samantha G. Pattenden;Daeyoup Lee;José Gutiérrez.
Proceedings of the National Academy of Sciences of the United States of America (2005)
Combined action of PHD and chromo domains directs the Rpd3S HDAC to transcribed chromatin.
Bing Li;Madelaine Gogol;Mike Carey;Mike Carey;Daeyoup Lee;Daeyoup Lee.
ZMYND11 links histone H3.3K36me3 to transcription elongation and tumour suppression
Hong Wen;Yuanyuan Li;Yuanxin Xi;Shiming Jiang.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: