D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 42 Citations 14,269 85 World Ranking 16971 National Ranking 429

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Enzyme

His primary areas of study are Genetics, Cell biology, Histone code, Histone and Histone methyltransferase. His work on Mitosis as part of general Cell biology research is often related to Genome instability, thus linking different fields of science. In his study, Bing Li carries out multidisciplinary Histone code and Histone methylation research.

The study incorporates disciplines such as Essential gene, Mutant and X chromosome, Dosage compensation in addition to Histone. His study in Histone methyltransferase is interdisciplinary in nature, drawing from both Histone H3, RNA polymerase II, Transcriptional regulation and Histone deacetylase complex. Bing Li combines subjects such as Chromatin remodeling and Histone octamer with his study of Histone H1.

His most cited work include:

  • The Role of Chromatin during Transcription (2711 citations)
  • Histone H3 methylation by Set2 directs deacetylation of coding regions by Rpd3S to suppress spurious intragenic transcription. (1060 citations)
  • Acetyl-CoA Induces Cell Growth and Proliferation by Promoting the Acetylation of Histones at Growth Genes (434 citations)

What are the main themes of his work throughout his whole career to date?

Bing Li mainly focuses on Cell biology, Genetics, Chromatin, Histone and Nucleosome. His Cell biology research is multidisciplinary, relying on both Spindle checkpoint, Chromatin remodeling, Mad2 and Histone code. His Chromatin remodeling research is multidisciplinary, incorporating perspectives in Histone H2A and In vitro.

His Histone code research incorporates themes from Histone H3 and Histone H1. Bing Li carries out multidisciplinary research, doing studies in Histone H1 and Histone methylation. His research in Chromatin intersects with topics in Molecular biology and Epigenetics.

He most often published in these fields:

  • Cell biology (64.86%)
  • Genetics (32.43%)
  • Chromatin (32.43%)

What were the highlights of his more recent work (between 2014-2020)?

  • Cell biology (64.86%)
  • Spindle checkpoint (17.57%)
  • Chromatin (32.43%)

In recent papers he was focusing on the following fields of study:

Bing Li mostly deals with Cell biology, Spindle checkpoint, Chromatin, Histone and Chromatin remodeling. His work deals with themes such as Anaphase-promoting complex, Histone H3, Genetics and Histone code, which intersect with Cell biology. Bing Li merges many fields, such as Histone code and Histone methylation, in his writings.

Bing Li usually deals with Spindle checkpoint and limits it to topics linked to Mad2 and Mitotic checkpoint complex and BUB3. His research on Chromatin focuses in particular on Nucleosome. His Histone H2A research is multidisciplinary, incorporating elements of Histone deacetylase complex and Histone H1.

Between 2014 and 2020, his most popular works were:

  • A sequential multi-target Mps1 phosphorylation cascade promotes spindle checkpoint signaling (85 citations)
  • The Bub1-Plk1 kinase complex promotes spindle checkpoint signalling through Cdc20 phosphorylation. (64 citations)
  • The Cdc20-binding Phe Box of the Spindle Checkpoint Protein BubR1 Maintains the Mitotic Checkpoint Complex During Mitosis (42 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The Role of Chromatin during Transcription

Bing Li;Michael Carey;Jerry L. Workman.
Cell (2007)

3967 Citations

Histone H3 methylation by Set2 directs deacetylation of coding regions by Rpd3S to suppress spurious intragenic transcription.

Michael J. Carrozza;Bing Li;Laurence Florens;Tamaki Suganuma.
Cell (2005)

1414 Citations

Acetyl-CoA Induces Cell Growth and Proliferation by Promoting the Acetylation of Histones at Growth Genes

Ling Cai;Benjamin M. Sutter;Bing Li;Benjamin P. Tu.
Molecular Cell (2011)

630 Citations

Readers of histone modifications

Miyong Yun;Jun Wu;Jerry L Workman;Bing Li.
Cell Research (2011)

619 Citations

Mad2-Independent Inhibition of APCCdc20 by the Mitotic Checkpoint Protein BubR1

Zhanyun Tang;Rajnish Bharadwaj;Bing Li;Hongtao Yu.
Developmental Cell (2001)

514 Citations

SIRT2 Maintains Genome Integrity and Suppresses Tumorigenesis through Regulating APC/C Activity

Hyun Seok Kim;Athanassios Vassilopoulos;Rui Hong Wang;Tyler Lahusen.
Cancer Cell (2011)

511 Citations

The Set2 histone methyltransferase functions through the phosphorylated carboxyl-terminal domain of RNA polymerase II.

Bing Li;LeAnn Howe;Scott Anderson;John R. Yates.
Journal of Biological Chemistry (2003)

412 Citations

Preferential occupancy of histone variant H2AZ at inactive promoters influences local histone modifications and chromatin remodeling

Bing Li;Samantha G. Pattenden;Daeyoup Lee;José Gutiérrez.
Proceedings of the National Academy of Sciences of the United States of America (2005)

381 Citations

Combined action of PHD and chromo domains directs the Rpd3S HDAC to transcribed chromatin.

Bing Li;Madelaine Gogol;Mike Carey;Mike Carey;Daeyoup Lee;Daeyoup Lee.
Science (2007)

369 Citations

ZMYND11 links histone H3.3K36me3 to transcription elongation and tumour suppression

Hong Wen;Yuanyuan Li;Yuanxin Xi;Shiming Jiang.
Nature (2014)

280 Citations

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