What is he best known for?
The fields of study he is best known for:
- Gene
- Alzheimer's disease
- Mitochondrion
His primary scientific interests are in Mitochondrion, Alzheimer's disease, Oxidative stress, Neuroscience and Disease.
His Mitochondrion study integrates concerns from other disciplines, such as Molecular biology and MFN2.
His work is dedicated to discovering how Alzheimer's disease, Pathogenesis are connected with Reactive oxygen species, Genetics and Endocrinology and other disciplines.
His Oxidative stress course of study focuses on Pathology and MitoQ and Microtubule-associated protein.
His Neuroscience research incorporates elements of BECN1, Bioinformatics and Autophagy-Related Protein 7.
The study incorporates disciplines such as mitochondrial fusion and FIS1 in addition to Mitochondrial fission.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
- Impaired Balance of Mitochondrial Fission and Fusion in Alzheimer's Disease (742 citations)
- Amyloid-β overproduction causes abnormal mitochondrial dynamics via differential modulation of mitochondrial fission/fusion proteins (590 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Mitochondrion, Neuroscience, Alzheimer's disease, Cell biology and Neurodegeneration.
Particularly relevant to Mitochondrial fission is his body of work in Mitochondrion.
Xinglong Wang interconnects Neuropathology, Disease, Parkinson's disease and Cell growth in the investigation of issues within Neuroscience.
His Alzheimer's disease research includes themes of Autophagy, Oxidative stress, Biochemistry, Signal transduction and Pathogenesis.
Xinglong Wang has researched Cell biology in several fields, including Genetically modified mouse, Programmed cell death, MFN2 and Neuroprotection.
His Neurodegeneration research focuses on Senile plaques and how it relates to Dementia.
He most often published in these fields:
- Mitochondrion (45.74%)
- Neuroscience (39.36%)
- Alzheimer's disease (37.23%)
What were the highlights of his more recent work (between 2016-2020)?
- Mitochondrion (45.74%)
- Neurodegeneration (25.53%)
- Neuroscience (39.36%)
In recent papers he was focusing on the following fields of study:
His primary areas of study are Mitochondrion, Neurodegeneration, Neuroscience, Cell biology and Amyotrophic lateral sclerosis.
His study looks at the relationship between Mitochondrion and topics such as mitochondrial fusion, which overlap with Central nervous system.
The various areas that Xinglong Wang examines in his Neurodegeneration study include Frontotemporal lobar degeneration and Senile plaques.
His Neuroscience study combines topics from a wide range of disciplines, such as Neurotoxicity and Disease.
The Cell biology study combines topics in areas such as Phenotype, Missense mutation, Genetically modified mouse and MFN2.
His Amyotrophic lateral sclerosis study combines topics in areas such as Cognitive deficit and Skeletal muscle.
Between 2016 and 2020, his most popular works were:
- Abnormalities of Mitochondrial Dynamics in Neurodegenerative Diseases (101 citations)
- Pathomechanisms of TDP-43 in neurodegeneration. (57 citations)
- Motor-Coordinative and Cognitive Dysfunction Caused by Mutant TDP-43 Could Be Reversed by Inhibiting Its Mitochondrial Localization (32 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Genetics
- Internal medicine
Neurodegeneration, Frontotemporal lobar degeneration, Mitochondrion, Amyotrophic lateral sclerosis and Cell biology are his primary areas of study.
His Frontotemporal lobar degeneration research integrates issues from Molecular biology, Protein aggregation, Alternative splicing and Intron.
His research combines Neuroscience and Amyotrophic lateral sclerosis.
His biological study spans a wide range of topics, including Neurotoxicity, Therapeutic approach, Pathological, Ubiquitin and Huntington's disease.
His work investigates the relationship between Cell biology and topics such as Genetically modified mouse that intersect with problems in Cognitive deficit, Phenotype and Missense mutation.
His Disease research is multidisciplinary, relying on both mitochondrial fusion and Central nervous system.
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