Hyperphosphorylation, Tau protein, Alzheimer's disease, Phosphorylation and Biochemistry are his primary areas of study. His Hyperphosphorylation research is multidisciplinary, incorporating perspectives in Protein phosphatase 2, DYRK1A, Human brain and Microtubule-associated protein. Fei Liu combines subjects such as Neuroscience and Gene isoform with his study of Tau protein.
His Alzheimer's disease study combines topics in areas such as Endocrinology, Glucose uptake, Neurodegeneration and Cell biology. Fei Liu has included themes like Calpain and Alternative splicing in his Neurodegeneration study. His Phosphorylation research integrates issues from Molecular biology, Glycosylation and Kinase.
The scientist’s investigation covers issues in Cell biology, Phosphorylation, Hyperphosphorylation, Exon and Alternative splicing. His research investigates the connection with Cell biology and areas like O glcnacylation which intersect with concerns in Ischemia. To a larger extent, he studies Biochemistry with the aim of understanding Phosphorylation.
The Hyperphosphorylation study combines topics in areas such as Alzheimer's disease, Tau protein, Neurodegeneration and Neuroscience, Human brain. His work in Alzheimer's disease addresses subjects such as Endocrinology, which are connected to disciplines such as Downregulation and upregulation. His research investigates the link between Exon and topics such as DYRK1A that cross with problems in Kinase activity.
His primary areas of study are Cell biology, Phosphorylation, Internal medicine, Hyperphosphorylation and Endocrinology. His work deals with themes such as Sirtuin, CREB and Alternative splicing, Exon, which intersect with Cell biology. A large part of his Phosphorylation studies is devoted to Protein kinase A.
His research integrates issues of Pathological, Disease, Pathology, Tau protein and Genetically modified mouse in his study of Hyperphosphorylation. His Endocrinology study incorporates themes from Endopeptidase, Neuroinflammation and Downregulation and upregulation. His work carried out in the field of Protein phosphatase 2 brings together such families of science as Dephosphorylation and Kinase.
His scientific interests lie mostly in Hyperphosphorylation, Internal medicine, Endocrinology, Tauopathy and Neuroscience. His Hyperphosphorylation study is concerned with the larger field of Phosphorylation. In his work, In vivo is strongly intertwined with Pathological, which is a subfield of Phosphorylation.
In his research, Glucose uptake is intimately related to Human brain, which falls under the overarching field of Endocrinology. Fei Liu combines subjects such as Frontotemporal lobar degeneration, Protein aggregation, Immunoglobulin G, Molecular biology and Intron with his study of Tauopathy. His Neuroscience research is multidisciplinary, incorporating elements of Disease and Drug discovery.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Tau pathology in Alzheimer disease and other tauopathies
Khalid Iqbal;Alejandra del C. Alonso;She Chen;M. Omar Chohan.
Biochimica et Biophysica Acta (2005)
Tau in Alzheimer disease and related tauopathies.
Khalid Iqbal;Fei Liu;Cheng-Xin Gong;Inge Grundke-Iqbal.
Current Alzheimer Research (2010)
Contributions of protein phosphatases PP1, PP2A, PP2B and PP5 to the regulation of tau phosphorylation
Fei Liu;Inge Grundke-Iqbal;Khalid Iqbal;Cheng-Xin Gong.
European Journal of Neuroscience (2005)
O-GlcNAcylation regulates phosphorylation of tau: A mechanism involved in Alzheimer's disease
Fei Liu;Khalid Iqbal;Inge Grundke-Iqbal;Gerald Warren Hart.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Mechanisms of tau-induced neurodegeneration
Khalid Iqbal;Fei Liu;Cheng-Xin Gong;Alejandra del C. Alonso.
Acta Neuropathologica (2009)
Tau and neurodegenerative disease: the story so far
Khalid Iqbal;Fei Liu;Cheng-Xin Gong.
Nature Reviews Neurology (2016)
Post-translational modifications of tau protein in Alzheimer's disease.
C.-X. Gong;F. Liu;I. Grundke-Iqbal;K. Iqbal.
Journal of Neural Transmission (2005)
Deficient brain insulin signalling pathway in Alzheimer's disease and diabetes.
Ying Liu;Fei Liu;Inge Grundke-Iqbal;Khalid Iqbal.
The Journal of Pathology (2011)
Microtubule-associated protein tau in development, degeneration and protection of neurons
Jian-Zhi Wang;Fei Liu.
Progress in Neurobiology (2008)
Reduced O-GlcNAcylation links lower brain glucose metabolism and tau pathology in Alzheimer's disease
Fei Liu;Fei Liu;Jianhua Shi;Hitoshi Tanimukai;Jinhua Gu.
Brain (2009)
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