D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 117 Citations 66,727 260 World Ranking 2337 National Ranking 1355

Overview

What is he best known for?

The fields of study William L. Gerald is best known for:

  • Gene
  • Cancer
  • Prostate cancer

The Vimentin portion of his research involves studies in Desmin and CD99. His study in Vimentin extends to CD99 with its themes. Internal medicine connects with themes related to Hormone in his study. He performs integrative study on Hormone and Estrogen receptor in his works. His work blends Estrogen receptor and Gene studies together. By researching both Gene and Gene duplication, he produces research that crosses academic boundaries. He integrates Gene duplication and Genetics in his studies. As part of his studies on Genetics, William L. Gerald often connects relevant areas like Prostate cancer. His work blends Prostate cancer and Breast cancer studies together.

His most cited work include:

  • An estrogen receptor-negative breast cancer subset characterized by a hormonally regulated transcriptional program and response to androgen (487 citations)
  • EWS-FLI1 fusion transcript structure is an independent determinant of prognosis in Ewing's sarcoma. (464 citations)
  • Duplication of the fusion of TMPRSS2 to ERG sequences identifies fatal human prostate cancer (406 citations)

What are the main themes of his work throughout his whole career to date

William L. Gerald integrates Genetics and Polymerase chain reaction in his research. He merges many fields, such as Polymerase chain reaction and Genetics, in his writings. His study ties his expertise on Carcinoma together with the subject of Pathology. Carcinoma is often connected to Pathology in his work. Internal medicine is closely attributed to Lymph node in his work. He integrates Lymph node with Chemotherapy in his research. William L. Gerald brings together Chemotherapy and Regimen to produce work in his papers. His study brings together the fields of Surgery and Regimen. Borrowing concepts from Disease, William L. Gerald weaves in ideas under Surgery.

William L. Gerald most often published in these fields:

  • Genetics (70.00%)
  • Pathology (55.00%)
  • Internal medicine (55.00%)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Comprehensive genomic characterization defines human glioblastoma genes and core pathways

Roger McLendon;Allan Friedman;Darrell Bigner;Erwin G. Van Meir.
Nature (2008)

6113 Citations

Integrative genomic profiling of human prostate cancer

Barry S. Taylor;Nikolaus Schultz;Haley Hieronymus;Anuradha Gopalan.
Cancer Cell (2010)

3644 Citations

Genes that mediate breast cancer metastasis to lung

Andy J. Minn;Gaorav P. Gupta;Peter M. Siegel;Peter M. Siegel;Paula D. Bos.
Nature (2005)

3246 Citations

Multiclass cancer diagnosis using tumor gene expression signatures

Sridhar Ramaswamy;Pablo Tamayo;Ryan Rifkin;Sayan Mukherjee.
Proceedings of the National Academy of Sciences of the United States of America (2001)

2520 Citations

Endogenous human microRNAs that suppress breast cancer metastasis

Sohail F. Tavazoie;Claudio Alarcón;Thordur Oskarsson;David Padua.
Nature (2008)

2338 Citations

Crucial role of p53-dependent cellular senescence in suppression of Pten-deficient tumorigenesis

Zhenbang Chen;Lloyd C. Trotman;David Shaffer;Hui Kuan Lin.
Nature (2005)

2202 Citations

Genes that mediate breast cancer metastasis to the brain

Paula D. Bos;Xiang H.-F. Zhang;Cristina Nadal;Weiping Shu.
Nature (2009)

1829 Citations

MET amplification occurs with or without T790M mutations in EGFR mutant lung tumors with acquired resistance to gefitinib or erlotinib

James Bean;Cameron Brennan;Jin-Yuan Shih;Gregory Riely.
Proceedings of the National Academy of Sciences of the United States of America (2007)

1745 Citations

Inactivation of the apoptosis effector Apaf-1 in malignant melanoma.

María S. Soengas;Paola Capodieci;David Polsky;Jaume Mora.
Nature (2001)

1238 Citations

Id1 and Id3 are required for neurogenesis, angiogenesis and vascularization of tumour xenografts

David Lyden;Alison Z. Young;David Zagzag;Wei Yan.
Nature (1999)

1138 Citations

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