Tristram G. Parslow mainly investigates Molecular biology, Immunology, RNA, Cancer research and Cytokine. His Molecular biology research is multidisciplinary, relying on both Phenotype, Phosphatase, Glutamate receptor and Arginine. His work on T cell and Inflammation as part of general Immunology study is frequently connected to Trigeminal nerve and Lymphoid enhancer-binding factor 1, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
Tristram G. Parslow combines subjects such as Peptide sequence, Transcription, Sequence analysis and Binding site with his study of RNA. His research integrates issues of Endocrinology, T lymphocyte, Receptor, Internal medicine and Signal transduction in his study of Cancer research. The various areas that Tristram G. Parslow examines in his Cytokine study include Necrosis, Granuloma, Helminthiasis and Immune system.
The scientist’s investigation covers issues in RNA, Genetics, Molecular biology, Cell biology and Virology. His work in the fields of RNA, such as Telomerase RNA component, overlaps with other areas such as Dimer. His work deals with themes such as Cytoplasm, RNA-dependent RNA polymerase, Gene expression, Mutation and Sequence analysis, which intersect with Molecular biology.
His Cell biology research incorporates elements of Cell, Myeloma protein, Secretion and B cell. Tristram G. Parslow has included themes like Telomere and Function in his Telomerase study. Tristram G. Parslow works mostly in the field of Telomere, limiting it down to topics relating to Cancer research and, in certain cases, Immunology, Endocrinology, Internal medicine, T lymphocyte and Receptor.
The scientist’s investigation covers issues in RNA, Genetics, Telomere, Telomerase and Telomerase RNA component. The RNA-dependent RNA polymerase research Tristram G. Parslow does as part of his general RNA study is frequently linked to other disciplines of science, such as Dimer, therefore creating a link between diverse domains of science. In RNA-dependent RNA polymerase, Tristram G. Parslow works on issues like Molecular biology, which are connected to Clone, Dyskeratosis congenita and Point mutation.
His work on Telomerase rna as part of general Genetics study is frequently connected to Variant allele and Identification, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them. His Telomere research is multidisciplinary, incorporating perspectives in Chromosome and Cancer research. He works mostly in the field of Telomerase RNA component, limiting it down to topics relating to Cell biology and, in certain cases, Saccharomyces cerevisiae, Conserved sequence, Pseudoknot and Binding site, as a part of the same area of interest.
Tristram G. Parslow focuses on RNA, Genetics, Virology, Virus and Viral load. His RNA research incorporates themes from APOBEC3G, Conserved sequence, Saccharomyces cerevisiae and Cell biology. Pseudoknot, Telomere, Telomerase RNA component, Binding site and Telomerase are the primary areas of interest in his Genetics study.
The concepts of his Virology study are interwoven with issues in T cell, RNA-dependent RNA polymerase and Immunodeficiency. His research integrates issues of Three prime untranslated region, Genome and Coding region in his study of Virus. His Viral load study contributes to a more complete understanding of Immunology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Development of several organs that require inductive epithelial-mesenchymal interactions is impaired in LEF-1-deficient mice.
C van Genderen;R M Okamura;I Fariñas;R G Quo.
Genes & Development (1994)
Expression of the int-1 gene in transgenic mice is associated with mammary gland hyperplasia and adenocarcinomas in male and female mice.
Ann S. Tsukamoto;Rudolf Grosschedl;Raphael C. Guzman;Tristram Parslow.
Structure of the 5' ends of immunoglobulin genes: a novel conserved sequence
Tristram G. Parslow;Debra L. Blair;William J. Murphy;Daryl K. Granner.
Proceedings of the National Academy of Sciences of the United States of America (1984)
Human severe combined immunodeficiency due to a defect in ZAP-70, a T cell tyrosine kinase
Melissa E. Elder;Dong Lin;Jared Clever;Andrew C. Chan.
Tumour necrosis factor α restores granulomas and induces parasite egg-laying in schistosome-infected SCID mice
Payman Amiri;Richard M. Locksley;Tristram G. Parslow;Michael Sadickt.
ZAP-70 deficiency in an autosomal recessive form of severe combined immunodeficiency
Andrew C. Chan;Theresa A. Kadlecek;Melissa E. Elder;Alexandra H. Filipovich.
Loss of pancreatic islet tolerance induced by β-cell expression of interferon-γ
N Sarvetnick;J Shizuru;D Liggitt;D Liggitt;L Martin.
RNA secondary structure and binding sites for gag gene products in the 5' packaging signal of human immunodeficiency virus type 1.
J Clever;C Sassetti;T G Parslow.
Journal of Virology (1995)
An Inactivating Point Mutation in the Inhibitory Wedge of CD45 Causes Lymphoproliferation and Autoimmunity
Ravindra Majeti;Zheng Xu;Tristram G. Parslow;Jean L. Olson.
Oligomerization and RNA binding domains of the type 1 human immunodeficiency virus Rev protein: a dual function for an arginine-rich binding motif
Maria L. Zapp;Thomas J. Hope;Tristram G. Parslow;Michael R. Green.
Proceedings of the National Academy of Sciences of the United States of America (1991)
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