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Genetics

D-Index
77
Citations
25939
World Ranking
1758
National Ranking
805

Research.com Recognitions

  • 2018 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

Thomas W. Glover is affiliated with the University of Michigan-Ann Arbor in the United States. Their research primarily spans the field of Biochemistry, Genetics, and Molecular Biology, with a focus on Genetics and Molecular Biology as the predominant subfields.

Their scientific output includes papers published in several notable venues such as bioRxiv (Cold Spring Harbor Laboratory), Nature Communications, Nucleic Acids Research, Mutation Research/Reviews in Mutation Research, and Clinical Radiology.

Key topics covered in their work include Genetics and Neurodevelopmental Disorders, Genomic variations and chromosomal abnormalities, Genomics and Rare Diseases, Genomics and Phylogenetic Studies, DNA Repair Mechanisms, Genomics and Chromatin Dynamics, and Cancer Genomics and Diagnostics.

Frequent coauthors in their collaborations include Thomas E. Wilson, Samreen Ahmed, Amanda Winningham, and Jake Higgins.

Recent papers authored or coauthored by Thomas W. Glover are as follows:

  • Twin peaks: finding fragile sites with MiDAS-seq, 2020, published in Cell Research
  • Locus-specific transcription silencing at the FHIT gene suppresses replication stress-induced copy number variant formation and associated replication delay, 2021, published in Nucleic Acids Research
  • Diagnosis and management of acute pancreatitis, 2022, published in Clinical Radiology
  • Applications of advanced technologies for detecting genomic structural variation, 2023, published in Mutation Research/Reviews in Mutation Research
  • Replication stress induces POLQ-mediated structural variant formation throughout common fragile sites after entry into mitosis, 2024, published in Nature Communications

Thomas W. Glover was awarded the Fellow of the American Association for the Advancement of Science (AAAS) in 2018.

Best Publications

  • Recurrent de novo point mutations in lamin A cause Hutchinson-Gilford progeria syndrome

    Maria Eriksson;W. Ted Brown;Leslie B. Gordon;Leslie B. Gordon;Michael W. Glynn

  • Aberrant regulation of ras proteins in malignant tumour cells from type 1 neurofibromatosis patients.

    Tanya N. Basu;David H. Gutmann;Jonathan A. Fletcher;Thomas W. Glover

  • Isolation of a partial candidate gene for Menkes disease by positional cloning.

    Julian F. B. Mercer;Janie Livingston;Bryan Hall;Jennifer A. Paynter

  • Chromosome fragile sites.

    Sandra G. Durkin;Thomas W. Glover

  • DNA polymerase α inhibition by aphidicolin induces gaps and breaks at common fragile sites in human chromosomes

    Thomas W. Glover;Carol Berger;Jane Coyle;Barbara Echo

  • Mutations in FOXC2 (MFH-1), a forkhead family transcription factor, are responsible for the hereditary lymphedema-distichiasis syndrome.

    Jianming Fang;Susan L. Dagenais;Robert P. Erickson;Martin F. Arlt

  • ATR Regulates Fragile Site Stability

    Anne M. Casper;Paul Nghiem;Paul Nghiem;Martin F. Arlt;Thomas W. Glover

  • A de novo Alu insertion results in neurofibromatosis type 1.

    Margaret R. Wallace;Margaret R. Wallace;Lone B. Andersen;Ann M. Saulino;Paula E. Gregory

  • Somatic deletion of the neurofibromatosis type 1 gene in a neurofibrosarcoma supports a tumour suppressor gene hypothesis.

    Eric Legius;Eric Legius;Douglas A. Marchuk;Francis S. Collins;Thomas W. Glover

  • Isolation and characterization of the faciogenital dysplasia (Aarskog-Scott syndrome) gene: A putative Rho Rac guanine nucleotide exchange factor

    N. German Pasteris;Amy B. Cadle;Lindsay J. Logie;Mary E. M. Porteous

  • The Fanconi anemia pathway is required for the DNA replication stress response and for the regulation of common fragile site stability

    Niall G. Howlett;Toshiyasu Taniguchi;Sandra G. Durkin;Alan D. D'Andrea

  • FRA3B Extends Over a Broad Region and Contains a Spontaneous HPV16 Integration Site: Direct Evidence for the Coincidence of Viral Integration Sites and Fragile Sites

    Charles M. Wilke;Bryan K. Hall;Ann Hoge;William Paradee

  • Sex pheromone production and perception in European corn borer moths is determined by both autosomal and sex-linked genes.

    Wendell Roelofs;Thomas Glover;Xian-Han Tang;Isabelle Sreng

  • Incomplete processing of mutant lamin A in Hutchinson-Gilford progeria leads to nuclear abnormalities, which are reversed by farnesyltransferase inhibition

    Michael W. Glynn;Thomas W. Glover

  • Replication of a Common Fragile Site, FRA3B, Occurs Late in S Phase and is Delayed Further Upon Induction: Implications for the Mechanism of Fragile Site Induction

    Michelle M. Le Beau;Feyruz V. Rassool;Mary E. Neilly;Rafael Espinosa

  • FUdR induction of the X chromosome fragile site: evidence for the mechanism of folic acid and thymidine inhibition.

    Glover Tw

  • X-linked mental retardation: A study of 7 families

    P A Jacobs;T W Glover;M Mayer;P Fox

  • Fragile sites in cancer: more than meets the eye.

    Thomas W. Glover;Thomas E. Wilson;Martin F. Arlt

  • Chromosome breakage and recombination at fragile sites.

    T W Glover;C K Stein

  • Common fragile sites as targets for chromosome rearrangements

    Martin F. Arlt;Sandra G. Durkin;Ryan L. Ragland;Thomas W. Glover

Frequent Co-Authors

Francis S. Collins
Francis S. Collins National Institutes of Health
David G. Beer
David G. Beer University of Michigan–Ann Arbor
Robert P. Erickson
Robert P. Erickson University of Arizona
Mark B. Orringer
Mark B. Orringer University of Michigan–Ann Arbor
Samir M. Hanash
Samir M. Hanash The University of Texas MD Anderson Cancer Center
Rork Kuick
Rork Kuick University of Michigan–Ann Arbor
Harry A. Drabkin
Harry A. Drabkin Medical University of South Carolina
Frederick Hecht
Frederick Hecht University of California, San Francisco
david i smith
david i smith Mayo Clinic
Stephen T. Warren
Stephen T. Warren Emory University

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