What is he best known for?
The fields of study he is best known for:
His primary areas of investigation include Genetics, Chromosomal fragile site, Molecular biology, Chromosome Fragile Site and Chromosome.
His is doing research in Chromosome 17, Translocation Breakpoint, Nonsense mutation, Breakpoint and Progeroid syndromes, both of which are found in Genetics.
His biological study spans a wide range of topics, including Aphidicolin, DNA, DNA repair, DNA replication and X chromosome.
His Molecular biology study incorporates themes from Chromosome breakage, Biochemistry, Gene, cDNA library and Gene product.
His research investigates the connection between Chromosome Fragile Site and topics such as Metaphase that intersect with issues in Identification, Genome, Chromosome instability and Mendelian inheritance.
In his research on the topic of Chromosome, Chromosome 3 and Chromatid is strongly related with Chromosomal translocation.
His most cited work include:
- Mutations in the p53 gene occur in diverse human tumour types (2385 citations)
- Recurrent de novo point mutations in lamin A cause Hutchinson-Gilford progeria syndrome (1599 citations)
- Isolation of a partial candidate gene for Menkes disease by positional cloning. (583 citations)
What are the main themes of his work throughout his whole career to date?
Thomas W. Glover mainly investigates Genetics, Molecular biology, Chromosomal fragile site, Gene and Chromosome.
Genetics is represented through his Locus, Gene mapping, Breakpoint, X chromosome and Chromosome 17 research.
His Chromosome 17 study combines topics in areas such as Loss of heterozygosity and Neurofibromatosis.
His Molecular biology research is multidisciplinary, incorporating perspectives in Amplicon, Chromosome 3, DNA repair, Gene duplication and Fluorescence in situ hybridization.
Thomas W. Glover has included themes like Metaphase, Chromosome breakage, Chromosome Fragility and Aphidicolin in his Chromosomal fragile site study.
The Chromosome Fragile Site study combines topics in areas such as Tumor suppressor gene, FHIT and Chromosome instability.
He most often published in these fields:
- Genetics (61.99%)
- Molecular biology (35.67%)
- Chromosomal fragile site (28.07%)
What were the highlights of his more recent work (between 2011-2020)?
- Genetics (61.99%)
- Chromosomal fragile site (28.07%)
- Molecular biology (35.67%)
In recent papers he was focusing on the following fields of study:
Thomas W. Glover mainly focuses on Genetics, Chromosomal fragile site, Molecular biology, Copy-number variation and Cancer research.
His work in Genetics is not limited to one particular discipline; it also encompasses Bioinformatics.
A large part of his Chromosomal fragile site studies is devoted to Chromosome Fragile Site.
His work in Chromosome Fragile Site covers topics such as Locus which are related to areas like Pathology.
The concepts of his Molecular biology study are interwoven with issues in Breakpoint, Gene, Chromosomal translocation and Chromosome 3.
His Copy-number variation study combines topics from a wide range of disciplines, such as Aphidicolin, DNA replication, Gene duplication, Penetrance and Gonadoblastoma.
Between 2011 and 2020, his most popular works were:
- Large transcription units unify copy number variants and common fragile sites arising under replication stress (111 citations)
- Fragile sites in cancer: more than meets the eye. (110 citations)
- REV1 and polymerase ζ facilitate homologous recombination repair (95 citations)
In his most recent research, the most cited papers focused on:
His primary scientific interests are in Genetics, Copy-number variation, DNA replication, DNA repair and DNA repair protein XRCC4.
His studies in Chromosome Fragile Site, Chromosome instability, Chromosomal fragile site and Chromosome breakage are all subfields of Genetics research.
His Copy-number variation study is concerned with the field of Gene as a whole.
In his research, Mutagen and Breakpoint is intimately related to Genome, which falls under the overarching field of DNA replication.
His research integrates issues of DNA polymerase and Proliferating cell nuclear antigen in his study of DNA repair.
As part of his studies on Homology directed repair, he frequently links adjacent subjects like Molecular biology.
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