What is he best known for?
The fields of study he is best known for:
The scientist’s investigation covers issues in Cell biology, Molecular biology, Matrix metalloproteinase, Genetics and Biochemistry.
His Cell biology research is multidisciplinary, incorporating perspectives in Autophagy, Progerin and Proteases.
His work focuses on many connections between Autophagy and other disciplines, such as Programmed cell death, that overlap with his field of interest in Neuroscience.
His research on Molecular biology also deals with topics like
- Complementary DNA together with Peptide sequence and Signal peptide,
- Gene expression that connect with fields like Regulation of gene expression.
His Matrix metalloproteinase research is multidisciplinary, relying on both Extracellular matrix, Western blot, Collagenase and Pathology.
His biological study deals with issues like In vivo, which deal with fields such as Inflammation.
His most cited work include:
- The Hallmarks of Aging (5741 citations)
- Signatures of mutational processes in human cancer (5372 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
What are the main themes of his work throughout his whole career to date?
Carlos López-Otín mainly focuses on Cell biology, Biochemistry, Molecular biology, Matrix metalloproteinase and Genetics.
He regularly links together related areas like Autophagy in his Cell biology studies.
His Molecular biology research incorporates themes from Complementary DNA, Gene expression, Northern blot and Signal peptide.
The study incorporates disciplines such as Angiogenesis, Immunology, Pathology, Extracellular matrix and Collagenase in addition to Matrix metalloproteinase.
His Immunology research focuses on Cancer research and how it relates to Metastasis.
Gene, Progeria, Mutation, Genome and Epigenetics are subfields of Genetics in which his conducts study.
He most often published in these fields:
- Cell biology (21.79%)
- Biochemistry (19.26%)
- Molecular biology (18.48%)
What were the highlights of his more recent work (between 2013-2021)?
- Cell biology (21.79%)
- Genetics (16.54%)
- Cancer research (13.23%)
In recent papers he was focusing on the following fields of study:
Carlos López-Otín mainly investigates Cell biology, Genetics, Cancer research, Progeria and Chronic lymphocytic leukemia.
The Cell biology study combines topics in areas such as Proteases, Reprogramming and Autophagy.
His Autophagy study necessitates a more in-depth grasp of Biochemistry.
His studies examine the connections between Cancer research and genetics, as well as such issues in Immunology, with regards to Signal transduction.
His study looks at the relationship between Progeria and fields such as Neuroscience, as well as how they intersect with chemical problems.
Carlos López-Otín focuses mostly in the field of Chronic lymphocytic leukemia, narrowing it down to topics relating to Epigenetics and, in certain cases, Mitosis, Epigenomics and Bioinformatics.
Between 2013 and 2021, his most popular works were:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018. (1421 citations)
- Molecular definitions of autophagy and related processes (637 citations)
In his most recent research, the most cited papers focused on:
His main research concerns Cell biology, Genetics, Autophagy, [email protected] and Chronic lymphocytic leukemia.
His study in Cell biology is interdisciplinary in nature, drawing from both Proteases and Mitophagy.
Many of his research projects under Genetics are closely connected to Context with Context, tying the diverse disciplines of science together.
The various areas that Carlos López-Otín examines in his Gene study include Cancer and Longevity.
In his study, Chaperone-mediated autophagy is strongly linked to Programmed cell death, which falls under the umbrella field of Autophagy.
His Computational biology study incorporates themes from BECN1, Autophagosome, Autolysosome, MAP1LC3B and Sequestosome 1.
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