2013 - Fellow of the American Association for the Advancement of Science (AAAS)
1980 - Fellow of Alfred P. Sloan Foundation
His primary scientific interests are in Cell biology, Skeletal muscle, Acetylcholine receptor, Agrin and Receptor tyrosine kinase. His study in Cell biology is interdisciplinary in nature, drawing from both Neuromuscular junction and Muscle atrophy. His Neuromuscular junction research is multidisciplinary, incorporating perspectives in Receptor, Internal medicine and Endocrinology.
The Skeletal muscle study which covers Biochemistry that intersects with Muscle relaxation and Myopathy. His biological study spans a wide range of topics, including Acetylcholine, Molecular biology, Neuregulin, Regulation of gene expression and Spinal cord. His Agrin research focuses on Gene expression and how it relates to Tyrosine phosphorylation.
His scientific interests lie mostly in Cell biology, Agrin, Acetylcholine receptor, Myocyte and Molecular biology. His research in Cell biology intersects with topics in Neuromuscular junction, Immunology and Biochemistry. In Neuromuscular junction, Steven J. Burden works on issues like Endocrinology, which are connected to Motor nerve.
The Agrin study combines topics in areas such as Receptor tyrosine kinase, Phosphorylation and Neuroscience. He works mostly in the field of Acetylcholine receptor, limiting it down to topics relating to Synapse and, in certain cases, Neurotransmission. Steven J. Burden focuses mostly in the field of Myocyte, narrowing it down to topics relating to Skeletal muscle and, in certain cases, Neuregulin and Transgene.
Steven J. Burden mainly investigates Cell biology, Agrin, Acetylcholine receptor, Myasthenia gravis and Synapse. His work in the fields of Cell biology, such as Signal transduction, Myocyte and Myogenesis, overlaps with other areas such as Abelson Tyrosine-Protein Kinase 2 and Myoblast proliferation. Steven J. Burden works mostly in the field of Signal transduction, limiting it down to concerns involving Skeletal muscle and, occasionally, Gene expression.
The various areas that Steven J. Burden examines in his Myocyte study include Regulation of gene expression and Transgene. His work carried out in the field of Agrin brings together such families of science as Neuromuscular junction, Endocrinology, Receptor tyrosine kinase and Kinase activity. Steven J. Burden is interested in Dok-7, which is a branch of Acetylcholine receptor.
Steven J. Burden mainly focuses on Agrin, Cell biology, Receptor tyrosine kinase, Acetylcholine receptor and Kinase activity. His Agrin study integrates concerns from other disciplines, such as Molecular biology, Endocrinology and Neuromuscular junction. Steven J. Burden incorporates Cell biology and Somite in his research.
He studied Receptor tyrosine kinase and Immunology that intersect with Retrograde signaling, Muscle Denervation, SOD1, Amyotrophic lateral sclerosis and Genetically modified mouse. He works in the field of Acetylcholine receptor, namely Dok-7. The study incorporates disciplines such as Synapsin and Skeletal muscle in addition to Synapse.
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FoxO3 controls autophagy in skeletal muscle in vivo.
Cristina Mammucari;Giulia Milan;Vanina Romanello;Eva Masiero.
Cell Metabolism (2007)
The Receptor Tyrosine Kinase MuSK Is Required for Neuromuscular Junction Formation In Vivo
Thomas M DeChiara;David C Bowen;David M Valenzuela;Mary V Simmons.
Agrin Acts via a MuSK Receptor Complex
David J Glass;David C Bowen;Trevor N Stitt;Czeslaw Radziejewski.
Lrp4 Is a Receptor for Agrin and Forms a Complex with MuSK
Natalie Kim;Amy L. Stiegler;Thomas O. Cameron;Peter T. Hallock.
Patterning of muscle acetylcholine receptor gene expression in the absence of motor innervation
Xia Yang;Silvia Arber;Christopher William;Li Li.
Receptor tyrosine kinase specific for the skeletal muscle lineage: Expression in embryonic muscle, at the neuromuscular junction, and after injury
David M. Valenzuela;Trevor N. Stitt;Peter S. DiStefano;Eduardo Rojas.
Acetylcholine receptors in regenerating muscle accumulate at original synaptic sites in the absence of the nerve.
S J Burden;P B Sargent;U J McMahan.
Journal of Cell Biology (1979)
DNA topoisomerase IIβ and neural development
Xia Yang;Wei Li;Elizabeth D. Prescott;Steven J. Burden.
Neuregulins are concentrated at nerve-muscle synapses and activate ACh–receptor gene expression
Sangmee Ahn Jo;Xuejun Zhu;Xuejun Zhu;Mark A. Marchionni;Steven J. Burden.
Increased mitochondrial mass in mitochondrial myopathy mice.
Anna Wredenberg;Rolf Wibom;Hans Wilhelmsson;Caroline Graff.
Proceedings of the National Academy of Sciences of the United States of America (2002)
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