D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 62 Citations 14,709 114 World Ranking 6974 National Ranking 3237

Research.com Recognitions

Awards & Achievements

2013 - Fellow of the American Association for the Advancement of Science (AAAS)

1980 - Fellow of Alfred P. Sloan Foundation

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Genetics

His primary scientific interests are in Cell biology, Skeletal muscle, Acetylcholine receptor, Agrin and Receptor tyrosine kinase. His study in Cell biology is interdisciplinary in nature, drawing from both Neuromuscular junction and Muscle atrophy. His Neuromuscular junction research is multidisciplinary, incorporating perspectives in Receptor, Internal medicine and Endocrinology.

The Skeletal muscle study which covers Biochemistry that intersects with Muscle relaxation and Myopathy. His biological study spans a wide range of topics, including Acetylcholine, Molecular biology, Neuregulin, Regulation of gene expression and Spinal cord. His Agrin research focuses on Gene expression and how it relates to Tyrosine phosphorylation.

His most cited work include:

  • FoxO3 controls autophagy in skeletal muscle in vivo. (1370 citations)
  • The Receptor Tyrosine Kinase MuSK Is Required for Neuromuscular Junction Formation In Vivo (765 citations)
  • Agrin Acts via a MuSK Receptor Complex (594 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Cell biology, Agrin, Acetylcholine receptor, Myocyte and Molecular biology. His research in Cell biology intersects with topics in Neuromuscular junction, Immunology and Biochemistry. In Neuromuscular junction, Steven J. Burden works on issues like Endocrinology, which are connected to Motor nerve.

The Agrin study combines topics in areas such as Receptor tyrosine kinase, Phosphorylation and Neuroscience. He works mostly in the field of Acetylcholine receptor, limiting it down to topics relating to Synapse and, in certain cases, Neurotransmission. Steven J. Burden focuses mostly in the field of Myocyte, narrowing it down to topics relating to Skeletal muscle and, in certain cases, Neuregulin and Transgene.

He most often published in these fields:

  • Cell biology (49.57%)
  • Agrin (40.17%)
  • Acetylcholine receptor (39.32%)

What were the highlights of his more recent work (between 2009-2020)?

  • Cell biology (49.57%)
  • Agrin (40.17%)
  • Acetylcholine receptor (39.32%)

In recent papers he was focusing on the following fields of study:

Steven J. Burden mainly investigates Cell biology, Agrin, Acetylcholine receptor, Myasthenia gravis and Synapse. His work in the fields of Cell biology, such as Signal transduction, Myocyte and Myogenesis, overlaps with other areas such as Abelson Tyrosine-Protein Kinase 2 and Myoblast proliferation. Steven J. Burden works mostly in the field of Signal transduction, limiting it down to concerns involving Skeletal muscle and, occasionally, Gene expression.

The various areas that Steven J. Burden examines in his Myocyte study include Regulation of gene expression and Transgene. His work carried out in the field of Agrin brings together such families of science as Neuromuscular junction, Endocrinology, Receptor tyrosine kinase and Kinase activity. Steven J. Burden is interested in Dok-7, which is a branch of Acetylcholine receptor.

Between 2009 and 2020, his most popular works were:

  • A dystroglycan mutation associated with limb-girdle muscular dystrophy. (198 citations)
  • MuSK IgG4 autoantibodies cause myasthenia gravis by inhibiting binding between MuSK and Lrp4 (155 citations)
  • Lrp4 is a retrograde signal for presynaptic differentiation at neuromuscular synapses (138 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Genetics

Steven J. Burden mainly focuses on Agrin, Cell biology, Receptor tyrosine kinase, Acetylcholine receptor and Kinase activity. His Agrin study integrates concerns from other disciplines, such as Molecular biology, Endocrinology and Neuromuscular junction. Steven J. Burden incorporates Cell biology and Somite in his research.

He studied Receptor tyrosine kinase and Immunology that intersect with Retrograde signaling, Muscle Denervation, SOD1, Amyotrophic lateral sclerosis and Genetically modified mouse. He works in the field of Acetylcholine receptor, namely Dok-7. The study incorporates disciplines such as Synapsin and Skeletal muscle in addition to Synapse.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

FoxO3 controls autophagy in skeletal muscle in vivo.

Cristina Mammucari;Giulia Milan;Vanina Romanello;Eva Masiero.
Cell Metabolism (2007)

1988 Citations

The Receptor Tyrosine Kinase MuSK Is Required for Neuromuscular Junction Formation In Vivo

Thomas M DeChiara;David C Bowen;David M Valenzuela;Mary V Simmons.
Cell (1996)

1060 Citations

Agrin Acts via a MuSK Receptor Complex

David J Glass;David C Bowen;Trevor N Stitt;Czeslaw Radziejewski.
Cell (1996)

834 Citations

Lrp4 Is a Receptor for Agrin and Forms a Complex with MuSK

Natalie Kim;Amy L. Stiegler;Thomas O. Cameron;Peter T. Hallock.
Cell (2008)

597 Citations

Patterning of muscle acetylcholine receptor gene expression in the absence of motor innervation

Xia Yang;Silvia Arber;Christopher William;Li Li.
Neuron (2001)

536 Citations

Receptor tyrosine kinase specific for the skeletal muscle lineage: Expression in embryonic muscle, at the neuromuscular junction, and after injury

David M. Valenzuela;Trevor N. Stitt;Peter S. DiStefano;Eduardo Rojas.
Neuron (1995)

490 Citations

Acetylcholine receptors in regenerating muscle accumulate at original synaptic sites in the absence of the nerve.

S J Burden;P B Sargent;U J McMahan.
Journal of Cell Biology (1979)

425 Citations

DNA topoisomerase IIβ and neural development

Xia Yang;Wei Li;Elizabeth D. Prescott;Steven J. Burden.
Science (2000)

419 Citations

Neuregulins are concentrated at nerve-muscle synapses and activate ACh–receptor gene expression

Sangmee Ahn Jo;Xuejun Zhu;Xuejun Zhu;Mark A. Marchionni;Steven J. Burden.
Nature (1995)

344 Citations

Increased mitochondrial mass in mitochondrial myopathy mice.

Anna Wredenberg;Rolf Wibom;Hans Wilhelmsson;Caroline Graff.
Proceedings of the National Academy of Sciences of the United States of America (2002)

290 Citations

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