What is he best known for?
The fields of study he is best known for:
Shigeru Yanagi mostly deals with Cell biology, Molecular biology, Tyrosine kinase, Tyrosine phosphorylation and Kinase.
His Cell biology research includes themes of Ubiquitin and MARCH5.
The various areas that Shigeru Yanagi examines in his Ubiquitin study include Mutant, Mitochondrial fission and Small interfering RNA.
His Molecular biology research is multidisciplinary, relying on both Triphosphatase, Ataxia, Cerebellar ataxia, Genetic enhancement and Purkinje cell.
The Tyrosine phosphorylation study combines topics in areas such as Platelet, Proto-oncogene tyrosine-protein kinase Src and Syk.
Shigeru Yanagi has researched Kinase in several fields, including Tumor suppressor gene, Thrombin, Cell type and CRMP1.
His most cited work include:
- Deletion of SHIP or SHP-1 Reveals Two Distinct Pathways for Inhibitory Signaling (467 citations)
- A novel mitochondrial ubiquitin ligase plays a critical role in mitochondrial dynamics. (279 citations)
- A novel mitochondrial ubiquitin ligase plays a critical role in mitochondrial dynamics. (279 citations)
What are the main themes of his work throughout his whole career to date?
His main research concerns Cell biology, Molecular biology, Mitochondrion, Phosphorylation and Syk.
His Cell biology study incorporates themes from Apoptosis, Programmed cell death, Ubiquitin and MARCH5.
Within one scientific family, Shigeru Yanagi focuses on topics pertaining to Mutant under Molecular biology, and may sometimes address concerns connected to Microtubule.
His Mitochondrion study combines topics from a wide range of disciplines, such as Endocrinology, Internal medicine, Alternative splicing and Mitochondrial permeability transition pore.
His Syk research incorporates elements of Oxidative stress, Cancer research, Kinase and Phospholipase C.
His work deals with themes such as Tyrosine kinase, LYN, Intracellular signal transduction and Platelet, which intersect with Tyrosine phosphorylation.
He most often published in these fields:
- Cell biology (62.39%)
- Molecular biology (24.77%)
- Mitochondrion (19.27%)
What were the highlights of his more recent work (between 2016-2021)?
- Cell biology (62.39%)
- Mitochondrion (19.27%)
- Mitochondrial fission (7.34%)
In recent papers he was focusing on the following fields of study:
Shigeru Yanagi focuses on Cell biology, Mitochondrion, Mitochondrial fission, Ubiquitin and MARCH5.
His Cell biology research includes elements of Functional analysis and Histone.
His work in Mitochondrion addresses subjects such as Programmed cell death, which are connected to disciplines such as Downregulation and upregulation and Lactic acidosis.
His research in Mitochondrial fission intersects with topics in Autophagosome and Syntaxin 17.
Shigeru Yanagi has included themes like Chromatin, Histone H3, SUV39H1 and Nitric oxide metabolism in his Ubiquitin study.
His research in MARCH5 focuses on subjects like Apoptosis, which are connected to Retrograde signaling and Inositol.
Between 2016 and 2021, his most popular works were:
- MITOL prevents ER stress‐induced apoptosis by IRE1α ubiquitylation at ER–mitochondria contact sites (36 citations)
- Psychiatric behaviors associated with cytoskeletal defects in radial neuronal migration (14 citations)
- MITOL deletion in the brain impairs mitochondrial structure and ER tethering leading to oxidative stress (11 citations)
In his most recent research, the most cited papers focused on:
Shigeru Yanagi mainly investigates Cell biology, Mitochondrial fission, Endoplasmic reticulum, Mitochondrion and Endocytic recycling.
His Cell biology research incorporates themes from Oxidative stress, Autophagosome and Apoptosis.
His Mitochondrial fission research is multidisciplinary, incorporating perspectives in Autophagy, Phosphatidylinositol, Protein subunit and Syntaxin 17.
His biological study spans a wide range of topics, including Ubiquitin, MARCH5, Programmed cell death and Cell fate determination.
His research integrates issues of Neuroinflammation and Biogenesis in his study of Mitochondrion.
His Endocytic recycling research incorporates elements of Slit-Robo, Axon guidance, Axon, Slit and Commissure.
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