D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Molecular Biology D-index 75 Citations 24,858 314 World Ranking 723 National Ranking 17

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Mutation
  • DNA

Roland Berger spends much of his time researching Molecular biology, Gene, Chromosomal translocation, Cancer research and Genetics. His Molecular biology research is multidisciplinary, incorporating perspectives in Complementary DNA, Fluorescence in situ hybridization, Gene rearrangement and Gene mapping. His Chromosomal translocation research integrates issues from Chromosome, Immunology and Virology.

Roland Berger studies Immunology, focusing on Myelofibrosis in particular. Roland Berger has researched Cancer research in several fields, including Tyrosine kinase, Leukemia, Retinoic acid and Acute lymphocytic leukemia. His studies deal with areas such as Oncology, Karyotype, Multivariate analysis, Pathology and Myeloid leukemia as well as Leukemia.

His most cited work include:

  • A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera (2928 citations)
  • All-trans retinoic acid as a differentiation therapy for acute promyelocytic leukemia. I. Clinical results. (969 citations)
  • All-trans retinoic acid as a differentiation therapy for acute promyelocytic leukemia. I. Clinical results [see comments] (919 citations)

What are the main themes of his work throughout his whole career to date?

Roland Berger mainly focuses on Molecular biology, Chromosomal translocation, Genetics, Cytogenetics and Gene. His Molecular biology research includes elements of In situ hybridization, Locus, Gene mapping, Fluorescence in situ hybridization and Acute lymphocytic leukemia. His research integrates issues of Leukemia, Gene rearrangement, Karyotype and Cancer research in his study of Chromosomal translocation.

Leukemia is a subfield of Immunology that he studies. The Immunology study combines topics in areas such as Internal medicine and Aneuploidy. The concepts of his Cytogenetics study are interwoven with issues in Trisomy, Acute leukemia and Incidence, Pathology.

He most often published in these fields:

  • Molecular biology (42.11%)
  • Chromosomal translocation (39.47%)
  • Genetics (29.82%)

What were the highlights of his more recent work (between 1999-2009)?

  • Molecular biology (42.11%)
  • Chromosomal translocation (39.47%)
  • Genetics (29.82%)

In recent papers he was focusing on the following fields of study:

His main research concerns Molecular biology, Chromosomal translocation, Genetics, Gene and Cancer research. His biological study spans a wide range of topics, including Polymorphism, Heterochromatin, Cytogenetics, Fluorescence in situ hybridization and Leukemia. His Leukemia study is concerned with the field of Immunology as a whole.

His Chromosomal translocation study incorporates themes from Fusion gene, Gene rearrangement, Karyotype and BCL11B. His study in Gene is interdisciplinary in nature, drawing from both Acute leukemia and Acute megakaryoblastic leukemia. His Cancer research research incorporates themes from Acute myeloblastic leukemia, TEL-JAK2, Acute lymphocytic leukemia and Cell biology.

Between 1999 and 2009, his most popular works were:

  • A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera (2928 citations)
  • The JAK2V617F activating mutation occurs in chronic myelomonocytic leukemia and acute myeloid leukemia, but not in acute lymphoblastic leukemia or chronic lymphocytic leukemia (338 citations)
  • A new recurrent and specific cryptic translocation, t(5;14)(q35;q32), is associated with expression of the Hox11L2 gene in T acute lymphoblastic leukemia. (216 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Mutation
  • DNA

His primary areas of study are Cancer research, Genetics, Gene, Molecular biology and Chromosomal translocation. His Cancer research study combines topics in areas such as Tyrosine kinase, Gene rearrangement, Janus kinase 2 and Acute lymphocytic leukemia. His research investigates the link between Gene rearrangement and topics such as Fluorescence in situ hybridization that cross with problems in clone, Gastroenterology and Internal medicine.

The subject of his Acute lymphocytic leukemia research is within the realm of Immunology. His Immunology study typically links adjacent topics like Polycythaemia. His work carried out in the field of Chromosomal translocation brings together such families of science as Karyotype and BCL11B.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera

Chloé James;Valérie Ugo;Jean-Pierre Le Couédic;Judith Staerk.
Nature (2005)

4251 Citations

All-trans retinoic acid as a differentiation therapy for acute promyelocytic leukemia. I. Clinical results.

Sylvie Castaigne;Christine Chomienne;Marie Therese Daniel;Paola Ballerini.
Blood (1990)

1840 Citations

All-trans retinoic acid as a differentiation therapy for acute promyelocytic leukemia. I. Clinical results.

Sylvie Castaigne;Christine Chomienne;Marie Therese Daniel;Paola Ballerini.
Blood (1990)

1518 Citations

NB4, a maturation inducible cell line with t(15;17) marker isolated from a human acute promyelocytic leukemia (M3)

M Lanotte;V Martin-Thouvenin;S Najman;P Balerini.
Blood (1991)

1263 Citations

A TEL-JAK2 Fusion Protein with Constitutive Kinase Activity in Human Leukemia

Virginie Lacronique;Virginie Lacronique;Anthony Boureux;Anthony Boureux;Véronique Della Valle;Véronique Della Valle;Hélène Poirel;Hélène Poirel.
Science (1997)

999 Citations

The t(12;21) of acute lymphoblastic leukemia results in a tel-AML1 gene fusion.

SP Romana;M Mauchauffe;M Le Coniat;I Chumakov.
Blood (1995)

669 Citations

High frequency of t(12;21) in childhood B-lineage acute lymphoblastic leukemia

SP Romana;H Poirel;M Leconiat;MA Flexor.
Blood (1995)

557 Citations

Identification of BTG2, an antiproliferative p53–dependent component of the DNA damage cellular response pathway

Jean-Pierre Rouault;Nicole Falette;Fabienne Guéhenneux;Céline Guillot.
Nature Genetics (1996)

492 Citations

The JAK2V617F activating mutation occurs in chronic myelomonocytic leukemia and acute myeloid leukemia, but not in acute lymphoblastic leukemia or chronic lymphocytic leukemia

Ross L. Levine;Marc Loriaux;Brian J. P. Huntly;Mignon L. Loh.
Blood (2005)

484 Citations

Bridging Ral GTPase to Rho pathways. RLIP76, a Ral effector with CDC42/Rac GTPase-activating protein activity.

Viviana Jullien-Flores;Olivier Dorseuil;Francisco Romero;Frank Letourneur.
Journal of Biological Chemistry (1995)

398 Citations

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