Roland Berger spends much of his time researching Molecular biology, Gene, Chromosomal translocation, Cancer research and Genetics. His Molecular biology research is multidisciplinary, incorporating perspectives in Complementary DNA, Fluorescence in situ hybridization, Gene rearrangement and Gene mapping. His Chromosomal translocation research integrates issues from Chromosome, Immunology and Virology.
Roland Berger studies Immunology, focusing on Myelofibrosis in particular. Roland Berger has researched Cancer research in several fields, including Tyrosine kinase, Leukemia, Retinoic acid and Acute lymphocytic leukemia. His studies deal with areas such as Oncology, Karyotype, Multivariate analysis, Pathology and Myeloid leukemia as well as Leukemia.
Roland Berger mainly focuses on Molecular biology, Chromosomal translocation, Genetics, Cytogenetics and Gene. His Molecular biology research includes elements of In situ hybridization, Locus, Gene mapping, Fluorescence in situ hybridization and Acute lymphocytic leukemia. His research integrates issues of Leukemia, Gene rearrangement, Karyotype and Cancer research in his study of Chromosomal translocation.
Leukemia is a subfield of Immunology that he studies. The Immunology study combines topics in areas such as Internal medicine and Aneuploidy. The concepts of his Cytogenetics study are interwoven with issues in Trisomy, Acute leukemia and Incidence, Pathology.
His main research concerns Molecular biology, Chromosomal translocation, Genetics, Gene and Cancer research. His biological study spans a wide range of topics, including Polymorphism, Heterochromatin, Cytogenetics, Fluorescence in situ hybridization and Leukemia. His Leukemia study is concerned with the field of Immunology as a whole.
His Chromosomal translocation study incorporates themes from Fusion gene, Gene rearrangement, Karyotype and BCL11B. His study in Gene is interdisciplinary in nature, drawing from both Acute leukemia and Acute megakaryoblastic leukemia. His Cancer research research incorporates themes from Acute myeloblastic leukemia, TEL-JAK2, Acute lymphocytic leukemia and Cell biology.
His primary areas of study are Cancer research, Genetics, Gene, Molecular biology and Chromosomal translocation. His Cancer research study combines topics in areas such as Tyrosine kinase, Gene rearrangement, Janus kinase 2 and Acute lymphocytic leukemia. His research investigates the link between Gene rearrangement and topics such as Fluorescence in situ hybridization that cross with problems in clone, Gastroenterology and Internal medicine.
The subject of his Acute lymphocytic leukemia research is within the realm of Immunology. His Immunology study typically links adjacent topics like Polycythaemia. His work carried out in the field of Chromosomal translocation brings together such families of science as Karyotype and BCL11B.
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A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera
Chloé James;Valérie Ugo;Jean-Pierre Le Couédic;Judith Staerk.
Nature (2005)
All-trans retinoic acid as a differentiation therapy for acute promyelocytic leukemia. I. Clinical results.
Sylvie Castaigne;Christine Chomienne;Marie Therese Daniel;Paola Ballerini.
Blood (1990)
All-trans retinoic acid as a differentiation therapy for acute promyelocytic leukemia. I. Clinical results.
Sylvie Castaigne;Christine Chomienne;Marie Therese Daniel;Paola Ballerini.
Blood (1990)
NB4, a maturation inducible cell line with t(15;17) marker isolated from a human acute promyelocytic leukemia (M3)
M Lanotte;V Martin-Thouvenin;S Najman;P Balerini.
Blood (1991)
A TEL-JAK2 Fusion Protein with Constitutive Kinase Activity in Human Leukemia
Virginie Lacronique;Virginie Lacronique;Anthony Boureux;Anthony Boureux;Véronique Della Valle;Véronique Della Valle;Hélène Poirel;Hélène Poirel.
Science (1997)
The t(12;21) of acute lymphoblastic leukemia results in a tel-AML1 gene fusion.
SP Romana;M Mauchauffe;M Le Coniat;I Chumakov.
Blood (1995)
High frequency of t(12;21) in childhood B-lineage acute lymphoblastic leukemia
SP Romana;H Poirel;M Leconiat;MA Flexor.
Blood (1995)
Identification of BTG2, an antiproliferative p53–dependent component of the DNA damage cellular response pathway
Jean-Pierre Rouault;Nicole Falette;Fabienne Guéhenneux;Céline Guillot.
Nature Genetics (1996)
The JAK2V617F activating mutation occurs in chronic myelomonocytic leukemia and acute myeloid leukemia, but not in acute lymphoblastic leukemia or chronic lymphocytic leukemia
Ross L. Levine;Marc Loriaux;Brian J. P. Huntly;Mignon L. Loh.
Blood (2005)
Bridging Ral GTPase to Rho pathways. RLIP76, a Ral effector with CDC42/Rac GTPase-activating protein activity.
Viviana Jullien-Flores;Olivier Dorseuil;Francisco Romero;Frank Letourneur.
Journal of Biological Chemistry (1995)
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Publications: 71
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