Philip J. Cowen mainly focuses on Internal medicine, Endocrinology, Serotonin, Antidepressant and Psychiatry. His research integrates issues of Tyrosine, Phenylalanine, Major depressive disorder, Depression and Positron emission tomography in his study of Internal medicine. His studies deal with areas such as Agonist and Receptor as well as Endocrinology.
Philip J. Cowen interconnects Tryptophan, Neuroscience and Crossover study in the investigation of issues within Serotonin. His biological study spans a wide range of topics, including Clinical psychology and Mood. The concepts of his Prolactin study are interwoven with issues in Appetite, Anorectic and Pharmacology.
Internal medicine, Endocrinology, Depression, Pharmacology and Antidepressant are his primary areas of study. Internal medicine is often connected to Placebo in his work. His work carried out in the field of Endocrinology brings together such families of science as Tryptophan and Agonist, Receptor.
Depression is the subject of his research, which falls under Psychiatry. His research in Pharmacology intersects with topics in Antagonist and Pindolol. Philip J. Cowen has included themes like Randomized controlled trial and Clinical psychology, Mood in his Antidepressant study.
His primary scientific interests are in Depression, Antidepressant, Psychiatry, Internal medicine and Neuroscience. His Depression research includes themes of Inflammation, Glutamate receptor, Emotional processing and Serotonin. His Antidepressant research is multidisciplinary, relying on both Receptor, Placebo, Cognition and Clinical psychology.
His work on Traumatic stress, Post-traumatic stress disorder, MDMA and Schizophrenia as part of general Psychiatry study is frequently linked to Service personnel, bridging the gap between disciplines. His work deals with themes such as Escitalopram and Endocrinology, which intersect with Internal medicine. His studies in Endocrinology integrate themes in fields like Glutamine and Glutamic acid.
His primary areas of investigation include Psychiatry, Internal medicine, Neuroscience, Antidepressant and Depression. Philip J. Cowen focuses mostly in the field of Psychiatry, narrowing it down to topics relating to Audiology and, in certain cases, Transcranial direct-current stimulation, Stimulation and Dorsolateral prefrontal cortex. Philip J. Cowen regularly ties together related areas like Endocrinology in his Internal medicine studies.
The Neuroscience study combines topics in areas such as Nuclear magnetic resonance spectroscopy and Inositol. He combines subjects such as Biomarker, Drug action, Randomized controlled trial and Clinical onset with his study of Antidepressant. His Depression research is multidisciplinary, incorporating elements of Hippocampus and Immune system.
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Brain serotonin1A receptor binding measured by positron emission tomography with [11C]WAY-100635: effects of depression and antidepressant treatment
Peter A. Sargent;Karen Husted Kjaer;Christopher J. Bench;Eugenii A. Rabiner.
Archives of General Psychiatry (2000)
Direct radioimmunoassay for melatonin in plasma.
S Fraser;P Cowen;M Franklin;C Franey.
Clinical Chemistry (1983)
Evidence-based guidelines for treating depressive disorders with antidepressants: A revision of the 2000 British Association for Psychopharmacology guidelines
IM Anderson;IN Ferrier;RC Baldwin;PJ Cowen.
Journal of Psychopharmacology (2008)
Increased positive versus negative affective perception and memory in healthy volunteers following selective serotonin and norepinephrine reuptake inhibition.
Catherine J. Harmer;Nicholas C. Shelley;Philip J. Cowen;Guy M. Goodwin.
American Journal of Psychiatry (2004)
Why do antidepressants take so long to work? A cognitive neuropsychological model of antidepressant drug action
Catherine J. Harmer;Guy M. Goodwin;Philip J. Cowen.
British Journal of Psychiatry (2009)
Shorter Oxford textbook of psychiatry
Philip Cowen;Paul Harrison;Tom Burns.
(2001)
Relapse of depression after rapid depletion of tryptophan.
KA Smith;CG Fairburn;PJ Cowen.
The Lancet (1997)
Antidepressant drug treatment modifies the neural processing of nonconscious threat cues.
Catherine J. Harmer;Clare E. Mackay;Catriona B. Reid;Philip J. Cowen.
Biological Psychiatry (2006)
Effect of Acute Antidepressant Administration on Negative Affective Bias in Depressed Patients
Catherine J Harmer;Ursula O'Sullivan;Elisa Favaron;Rachel Massey-Chase.
American Journal of Psychiatry (2009)
Acute SSRI administration affects the processing of social cues in healthy volunteers.
C J Harmer;Z Bhagwagar;D I Perrett;B A Völlm.
Neuropsychopharmacology (2003)
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