D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 67 Citations 27,102 108 World Ranking 3610 National Ranking 274

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • DNA

His primary areas of study are Cell biology, Molecular biology, Transcription factor, Transcription and c-jun. The various areas that Peter Angel examines in his Cell biology study include Endocrinology, Cellular differentiation, JUNB, Osteoblast and Internal medicine. His Molecular biology research incorporates themes from Amino acid, Complementary DNA, Gene, Promoter and Kinase.

Peter Angel has included themes like Regulation of gene expression, Glucocorticoid receptor, Binding site and Cancer research in his Transcription factor study. His Regulation of gene expression research includes themes of Sequence homology, Base sequence, AP-1 Complex, Bioinformatics and Neoplastic transformation. His works in Transactivation and Enhancer RNAs are all subjects of inquiry into Transcription.

His most cited work include:

  • The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformation. (3004 citations)
  • DNA binding of the glucocorticoid receptor is not essential for survival (977 citations)
  • AP-1 subunits: quarrel and harmony among siblings (976 citations)

What are the main themes of his work throughout his whole career to date?

Cell biology, Cancer research, Molecular biology, Transcription factor and JUNB are his primary areas of study. His study in Cell biology is interdisciplinary in nature, drawing from both Endocrinology, Internal medicine, Keratinocyte and Cellular differentiation. His work deals with themes such as Carcinogenesis, Inflammation, Immunology and Podoplanin, which intersect with Cancer research.

His work focuses on many connections between Inflammation and other disciplines, such as Immune system, that overlap with his field of interest in Cancer. His work carried out in the field of Molecular biology brings together such families of science as Regulation of gene expression, Transcription, Gene expression, Gene and c-jun. His Transcription factor research integrates issues from Promoter, Activator and Binding site.

He most often published in these fields:

  • Cell biology (36.04%)
  • Cancer research (30.46%)
  • Molecular biology (28.43%)

What were the highlights of his more recent work (between 2013-2021)?

  • Cancer research (30.46%)
  • Glioma (7.61%)
  • Cell biology (36.04%)

In recent papers he was focusing on the following fields of study:

Peter Angel spends much of his time researching Cancer research, Glioma, Cell biology, In vivo and Podoplanin. He combines subjects such as Cancer, Progenitor cell, JUNB, Downregulation and upregulation and Fibrosis with his study of Cancer research. His study explores the link between Cancer and topics such as Immune system that cross with problems in Inflammation.

His study with JUNB involves better knowledge in Transcription factor. His Glioma research also works with subjects such as

  • Brain tumor which connect with PTEN and Transcriptome,
  • Tumor microenvironment, which have a strong connection to Melanoma, S100A9 and S100A8. Peter Angel studies Senescence which is a part of Cell biology.

Between 2013 and 2021, his most popular works were:

  • Human and mouse VEGFA-amplified hepatocellular carcinomas are highly sensitive to sorafenib treatment (112 citations)
  • Nrf2-Mediated Fibroblast Reprogramming Drives Cellular Senescence by Targeting the Matrisome (56 citations)
  • Homeostatic nuclear RAGE-ATM interaction is essential for efficient DNA repair. (27 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • DNA

Peter Angel mostly deals with Cancer research, Cell biology, Glioma, Carcinogenesis and Fibroblast. His Cancer research research is multidisciplinary, relying on both In vitro, Downregulation and upregulation, Metabolomics and Brain tumor, Pathology. His work in the fields of Signal transduction overlaps with other areas such as Context.

His Glioma research is multidisciplinary, incorporating perspectives in Chemotherapy, Cancer stem cell, Slice preparation, Gentamicin protection assay and Neural stem cell. The Carcinogenesis study combines topics in areas such as Calgranulin, Senescence, S100A8 and S100A9. Peter Angel works mostly in the field of Fibroblast, limiting it down to topics relating to Wound healing and, in certain cases, Extracellular matrix, Reprogramming, Transcription factor and Chromatin immunoprecipitation.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformation.

Peter Angel;Michael Karin.
Biochimica et Biophysica Acta (1991)

4542 Citations

AP-1 subunits: quarrel and harmony among siblings

Jochen Hess;Peter Angel;Marina Schorpp-Kistner.
Journal of Cell Science (2004)

1527 Citations

DNA binding of the glucocorticoid receptor is not essential for survival

Holger M Reichardt;Klaus H Kaestner;Jan Tuckermann;Oliver Kretz.
Cell (1998)

1266 Citations

12-O-tetradecanoyl-phorbol-13-acetate induction of the human collagenase gene is mediated by an inducible enhancer element located in the 5'-flanking region.

Peter Angel;I. Baumann;B. Stein;H. Delius.
Molecular and Cellular Biology (1987)

966 Citations

Prolonged activation of jun and collagenase genes by tumour necrosis factor- α

David A. Brenner;Maureen O'Hara;Peter Angel;Mario Chojkier.
Nature (1989)

916 Citations

Jun-B differs in its biological properties from, and is a negative regulator of, c-Jun.

Robert Chiu;Peter Angel;Michael Karin.
Cell (1989)

890 Citations

ATF-2 is preferentially activated by stress-activated protein kinases to mediate c-jun induction in response to genotoxic agents.

H. Van Dam;D. Wilhelm;I. Herr;A. Steffen.
The EMBO Journal (1995)

763 Citations

Control of cell cycle progression by c-Jun is p53 dependent

Martin Schreiber;Andrea Kolbus;Fabrice Piu;Fabrice Piu;Axel Szabowski.
Genes & Development (1999)

741 Citations

Altered endochondral bone development in matrix metalloproteinase 13-deficient mice

Dominique Stickens;Danielle J. Behonick;Nathalie Ortega;Babette Heyer.
Development (2004)

740 Citations

S100A8 and S100A9 in inflammation and cancer.

Christoffer Gebhardt;Julia Németh;Peter Angel;Jochen Hess.
Biochemical Pharmacology (2006)

676 Citations

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