His main research concerns Biochemistry, RNA editing, Molecular biology, Messenger RNA and Cell biology. Nicholas O. Davidson works mostly in the field of Biochemistry, limiting it down to topics relating to Intestinal mucosa and, in certain cases, Inflammatory bowel disease, Inflammation, Intestinal epithelium and Interleukin 10, as a part of the same area of interest. His RNA editing research is multidisciplinary, relying on both Small intestine, Genetic diversity and Apolipoprotein B.
His Molecular biology study integrates concerns from other disciplines, such as Hedgehog signaling pathway, Lipid Transport and Binding site. His work deals with themes such as Steatosis, Microsomal triglyceride transfer protein, Cell growth, Immunology and Sense, which intersect with Cell biology. Nicholas O. Davidson's looking at Lipid metabolism as part of his Endocrinology and Internal medicine and Lipid metabolism study.
Nicholas O. Davidson mainly investigates Internal medicine, Endocrinology, Apolipoprotein B, Molecular biology and Cell biology. His work in Internal medicine addresses issues such as Gastroenterology, which are connected to fields such as Crohn's disease, Short bowel syndrome and Nonalcoholic fatty liver disease. The various areas that Nicholas O. Davidson examines in his Apolipoprotein B study include Very low-density lipoprotein and RNA editing.
His RNA editing study combines topics from a wide range of disciplines, such as APOBEC and Cytidine deaminase. Nicholas O. Davidson interconnects Gene expression and Messenger RNA, RNA-binding protein in the investigation of issues within Molecular biology. His study in Cell biology focuses on Lipid droplet in particular.
His primary scientific interests are in Internal medicine, Gastroenterology, Endocrinology, Steatosis and Nonalcoholic fatty liver disease. In most of his Internal medicine studies, his work intersects topics such as Microbiome. His research brings together the fields of Acinar cell and Endocrinology.
His studies in Steatosis integrate themes in fields like Fibrosis, Steatohepatitis and Triglyceride. Nicholas O. Davidson has researched Fibrosis in several fields, including Secretion, Chylomicron and Apolipoprotein B. Nicholas O. Davidson works mostly in the field of Molecular biology, limiting it down to topics relating to Genetically modified mouse and, in certain cases, RNA.
Nicholas O. Davidson mostly deals with Internal medicine, Steatosis, Nonalcoholic fatty liver disease, Endocrinology and Gastroenterology. His Internal medicine research is multidisciplinary, incorporating perspectives in Feces and Macrophage. His Steatosis research includes elements of Microsomal triglyceride transfer protein, Triglyceride, Transgene, Fibrosis and Steatohepatitis.
The Steatohepatitis study combines topics in areas such as Molecular biology, Lipogenesis and Phosphatidylinositol. His study in Endocrinology is interdisciplinary in nature, drawing from both Proinflammatory cytokine, Phenotype and Myeloid. His biological study spans a wide range of topics, including Microbiome, Gut flora, Interquartile range, Case-control study and Crohn's disease.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
C-to-U RNA editing: mechanisms leading to genetic diversity.
Valerie Blanc;Nicholas O. Davidson.
Journal of Biological Chemistry (2003)
Protective mucosal immunity mediated by epithelial CD1d and IL-10
Torsten Olszak;Joana F. Neves;C. Marie Dowds;Kristi Baker.
Trehalose inhibits solute carrier 2A (SLC2A) proteins to induce autophagy and prevent hepatic steatosis
Brian J. DeBosch;Monique R. Heitmeier;Allyson L. Mayer;Cassandra B. Higgins.
Science Signaling (2016)
Compensatory increase in hepatic lipogenesis in mice with conditional intestine-specific Mttp deficiency.
Yan Xie;Elizabeth P. Newberry;Stephen G. Young;Sylvie Robine.
Journal of Biological Chemistry (2006)
Fatty Acid Synthase Modulates Intestinal Barrier Function through Palmitoylation of Mucin 2
Xiaochao Wei;Zhen Yang;Federico E. Rey;Vanessa K. Ridaura.
Cell Host & Microbe (2012)
Ô‖Identification of GRY-RBP as an Apolipoprotein B RNA-binding Protein That Interacts with Both Apobec-1 and Apobec-1 Complementation Factor to Modulate C to U Editing
Valerie Blanc;Naveenan Navaratnam;Jeffrey O. Henderson;Shrikant Anant.
Journal of Biological Chemistry (2001)
Type 2 diabetes mellitus: the impact on colorectal adenoma risk in women.
Jill E Elwing;Feng Gao;Nicholas O Davidson;Dayna S Early.
The American Journal of Gastroenterology (2006)
Novel Role for RNA-binding Protein CUGBP2 in Mammalian RNA Editing CUGBP2 MODULATES C TO U EDITING OF APOLIPOPROTEIN B mRNA BY INTERACTING WITH APOBEC-1 AND ACF, THE APOBEC-1 COMPLEMENTATION FACTOR
Shrikant Anant;Jeffrey O. Henderson;Debnath Mukhopadhyay;Naveenan Navaratnam.
Journal of Biological Chemistry (2001)
Altered hepatic triglyceride content after partial hepatectomy without impaired liver regeneration in multiple murine genetic models.
Elizabeth P. Newberry;Susan M. Kennedy;Yan Xie;Jianyang Luo.
An AU-Rich Sequence Element (UUUN[A/U]U) Downstream of the Edited C in Apolipoprotein B mRNA Is a High-Affinity Binding Site for Apobec-1: Binding of Apobec-1 to This Motif in the 3′ Untranslated Region of c-myc Increases mRNA Stability
Shrikant Anant;Nicholas O. Davidson.
Molecular and Cellular Biology (2000)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: