D-Index & Metrics Best Publications
Research.com 2022 Best Female Scientist Award Badge

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Best female scientists D-index 105 Citations 54,992 196 World Ranking 695 National Ranking 432
Medicine D-index 83 Citations 37,017 184 World Ranking 10275 National Ranking 5405
Biology and Biochemistry D-index 86 Citations 39,559 183 World Ranking 1862 National Ranking 1050

Research.com Recognitions

Awards & Achievements

2022 - Research.com Best Female Scientist Award

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Enzyme
  • DNA

Hemojuvelin, Hepcidin, Hemochromatosis, Endocrinology and Internal medicine are her primary areas of study. She has researched Hemojuvelin in several fields, including Mutation and Anemia of chronic disease, Iron-deficiency anemia. Her work is dedicated to discovering how Hepcidin, Hormone are connected with Germline mutation and other disciplines.

Her research in Hemochromatosis intersects with topics in HAMP and Ferroportin. The study incorporates disciplines such as STAT protein, STAT3 Transcription Factor and Iron deficiency in addition to Endocrinology. Her Duodenal cytochrome B research is multidisciplinary, relying on both Transferrin and Hephaestin.

Her most cited work include:

  • A rapid micropreparation technique for extraction of DNA-binding proteins from limiting numbers of mammalian cells. (2261 citations)
  • p63, a p53 homolog at 3q27-29, encodes multiple products with transactivating, death-inducing, and dominant-negative activities. (1821 citations)
  • p63, a p53 homolog at 3q27-29, encodes multiple products with transactivating, death-inducing, and dominant-negative activities. (1821 citations)

What are the main themes of her work throughout her whole career to date?

Her primary areas of investigation include Internal medicine, Endocrinology, Hepcidin, Molecular biology and Hemochromatosis. Her work in the fields of Internal medicine, such as Erythroferrone, Hormone and Small intestine, overlaps with other areas such as TMPRSS6. Her work carried out in the field of Endocrinology brings together such families of science as Iron-deficiency anemia, HAMP, Signal transduction, Iron deficiency and Serum iron.

Nancy C. Andrews focuses mostly in the field of Hepcidin, narrowing it down to topics relating to Erythropoiesis and, in certain cases, Transgene. In her work, Cell biology is strongly intertwined with Transferrin, which is a subfield of Molecular biology. She has included themes like Mutation, Biochemistry, Knockout mouse and Transferrin receptor in her Hemochromatosis study.

She most often published in these fields:

  • Internal medicine (52.63%)
  • Endocrinology (51.42%)
  • Hepcidin (46.15%)

What were the highlights of her more recent work (between 2008-2021)?

  • Internal medicine (52.63%)
  • Endocrinology (51.42%)
  • Hepcidin (46.15%)

In recent papers she was focusing on the following fields of study:

Nancy C. Andrews mainly investigates Internal medicine, Endocrinology, Hepcidin, Biochemistry and Cell biology. Nancy C. Andrews works on Endocrinology which deals in particular with Hemojuvelin. Her studies in Hemojuvelin integrate themes in fields like Mutation and Membrane protein.

Her work in the fields of Hepcidin, such as Ferroportin, intersects with other areas such as TMPRSS6. Her research in Biochemistry focuses on subjects like Intestinal epithelium, which are connected to Stem cell and Intestinal mucosa. Nancy C. Andrews works mostly in the field of Cell biology, limiting it down to topics relating to DMT1 and, in certain cases, Programmed cell death, Knockout mouse, Apoptosis and Proinflammatory cytokine, as a part of the same area of interest.

Between 2008 and 2021, her most popular works were:

  • TRP Channel Regulates EGFR Signaling in Hair Morphogenesis and Skin Barrier Formation (216 citations)
  • TRP Channel Regulates EGFR Signaling in Hair Morphogenesis and Skin Barrier Formation (216 citations)
  • Scara5 is a ferritin receptor mediating non-transferrin iron delivery. (203 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • DNA

Her main research concerns Internal medicine, Endocrinology, Cell biology, Biochemistry and Hepcidin. Her Internal medicine study focuses mostly on Iron deficiency, Transferrin, Transferrin receptor and Ferroportin. Nancy C. Andrews combines subjects such as Molecular biology and Mesenchyme with her study of Transferrin receptor.

Her Endocrinology research is multidisciplinary, incorporating perspectives in Spleen, Immunology, Receptor, Endocytosis and Proinflammatory cytokine. The various areas that Nancy C. Andrews examines in her Cell biology study include Hairless, Transgene, Neuron and Keratinocyte. As part of the same scientific family, Nancy C. Andrews usually focuses on Hepcidin, concentrating on SMAD and intersecting with Hereditary hemochromatosis, Hemochromatosis and Hemojuvelin.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

A rapid micropreparation technique for extraction of DNA-binding proteins from limiting numbers of mammalian cells.

Nancy C. Andrews;Douglas V. Faller.
Nucleic Acids Research (1991)

2789 Citations

Disorders of iron metabolism.

Nancy C. Andrews.
The New England Journal of Medicine (1999)

2766 Citations

p63, a p53 homolog at 3q27-29, encodes multiple products with transactivating, death-inducing, and dominant-negative activities.

Annie Yang;Mourad Kaghad;Yunmei Wang;Emily Gillett.
Molecular Cell (1998)

2633 Citations

Balancing Acts: Molecular Control of Mammalian Iron Metabolism

Matthias W Hentze;Martina U Muckenthaler;Nancy C Andrews.
Cell (2004)

2070 Citations

Positional cloning of zebrafish ferroportin1 identifies a conserved vertebrate iron exporter

Adriana Donovan;Alison Brownlie;Alison Brownlie;Yi Zhou;Jennifer Shepard.
Nature (2000)

1921 Citations

Microcytic anaemia mice have a mutation in Nramp2, a candidate iron transporter gene.

Mark D. Fleming;Mark D. Fleming;Cameron C. Trenor;Cameron C. Trenor;Maureen A. Su;Maureen A. Su;Dorothee Foernzler.
Nature Genetics (1997)

1334 Citations

Bone morphogenetic protein signaling by hemojuvelin regulates hepcidin expression.

Jodie L Babitt;Franklin W Huang;Diedra M Wrighting;Yin Xia.
Nature Genetics (2006)

1250 Citations

The iron exporter ferroportin/Slc40a1 is essential for iron homeostasis

Adriana Donovan;Adriana Donovan;Christine A. Lima;Christine A. Lima;Jack L. Pinkus;Geraldine S. Pinkus.
Cell Metabolism (2005)

1192 Citations

Nramp2 is mutated in the anemic Belgrade (b) rat: Evidence of a role for Nramp2 in endosomal iron transport

Mark D. Fleming;Michelle A. Romano;Maureen A. Su;Laura M. Garrick.
Proceedings of the National Academy of Sciences of the United States of America (1998)

1133 Citations

Interleukin-6 Induces Hepcidin Expression Through STAT3

Diedra M. Wrighting;Nancy C. Andrews;Nancy C. Andrews.
Blood (2006)

1059 Citations

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