Her primary areas of investigation include Internal medicine, Endocrinology, Cell biology, Receptor and Immunology. Her Internal medicine research includes themes of Transcription factor and Heart development. Her Endocrinology research is multidisciplinary, incorporating elements of Beta, Beta protein, Wild type, Homeotic gene and In vivo.
Her work on ITGA7 as part of general Cell biology study is frequently linked to Neuroglia, therefore connecting diverse disciplines of science. Her studies deal with areas such as Retinoid, Nuclear receptor, Retinoid X receptor, Adipocyte and Signal transduction as well as Receptor. Nadia Messaddeq usually deals with Immunology and limits it to topics linked to Integrin and Hemidesmosome, Mutation, Phenotype and Epidermolysis bullosa.
Nadia Messaddeq focuses on Cell biology, Endocrinology, Internal medicine, Receptor and Molecular biology. Her research integrates issues of Biochemistry, Mutant, Retinoic acid and Skeletal muscle in her study of Cell biology. Her Endocrinology research is multidisciplinary, relying on both Prokineticin receptor 1, Nuclear receptor, Retinoid X receptor and Signal transduction.
Her Retinoid X receptor study combines topics from a wide range of disciplines, such as Gene knockout and Retinoid. The Receptor study combines topics in areas such as Cardiomyopathy, Heart disease and Immunology. Her Molecular biology research also works with subjects such as
Her primary scientific interests are in Cell biology, Retinal degeneration, Purkinje cell, Bardet–Biedl syndrome and Myotubularin. Her work on Phagocytosis is typically connected to Context as part of general Cell biology study, connecting several disciplines of science. Her Retinal degeneration research incorporates themes from Zebrafish, Eye morphogenesis, Eye development and Coloboma.
Nadia Messaddeq interconnects Retina and Epigenetics in the investigation of issues within Purkinje cell. Her work focuses on many connections between Bardet–Biedl syndrome and other disciplines, such as Ciliopathies, that overlap with her field of interest in Alström syndrome. Her Muscle weakness study incorporates themes from Myocyte, Integrin, Laminin and Skeletal muscle.
Nadia Messaddeq spends much of her time researching Cell biology, Muscle weakness, Myotubularin, DNM2 and Transmission electron microscopy. In her work, Nadia Messaddeq performs multidisciplinary research in Cell biology and Acetyltransferase complex. The study incorporates disciplines such as Laminin, Integrin, Skeletal muscle and Focal adhesion in addition to Muscle weakness.
Nadia Messaddeq combines Myotubularin and Myocyte in her research. The concepts of her DNM2 study are interwoven with issues in Congenital myopathy, Cancer research, Myopathy and Muscle disorder. Her work deals with themes such as Electron microscope, Phagocytosis, Sertoli cell and Confocal, which intersect with Transmission electron microscopy.
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Bile acids induce energy expenditure by promoting intracellular thyroid hormone activation
Mitsuhiro Watanabe;Sander M. Houten;Chikage Mataki;Marcelo A. Christoffolete.
Absence of integrin alpha 6 leads to epidermolysis bullosa and neonatal death in mice.
E Georges-Labouesse;N Messaddeq;G Yehia;L Cadalbert.
Nature Genetics (1996)
Embryonic retinoic acid synthesis is essential for heart morphogenesis in the mouse.
Karen Niederreither;Julien Vermot;Nadia Messaddeq;Brigitte Schuhbaur.
Peroxisome proliferator-activated receptor γ is required in mature white and brown adipocytes for their survival in the mouse
Takeshi Imai;Reiko Takakuwa;Sandra Marchand;Emilie Dentz.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Skin abnormalities generated by temporally controlled RXRα mutations in mouse epidermis
Mei Li;Arup Kumar Indra;Xavier Warot;Jacques Brocard.
Essential role of α6 integrins in cortical and retinal lamination
Elisabeth Georges-Labouesse;Manuel Mark;Nadia Messaddeq;Anne Gansmüller.
Current Biology (1998)
Serotonin 2B receptor is required for heart development.
Canan G. Nebigil;Doo Sup Choi;Andrée Dierich;Pierre Hickel.
Proceedings of the National Academy of Sciences of the United States of America (2000)
Spatio-temporally controlled site-specific somatic mutagenesis in the mouse
Jacques Brocard;Xavier Warot;Olivia Wendling;Nadia Messaddeq.
Proceedings of the National Academy of Sciences of the United States of America (1997)
Oral-facial-digital type I protein is required for primary cilia formation and left-right axis specification.
Maria Immacolata Ferrante;Alessandro Zullo;Adriano Barra;Sabrina Bimonte.
Nature Genetics (2006)
Misregulated alternative splicing of BIN1 is associated with T tubule alterations and muscle weakness in myotonic dystrophy
Charlotte Fugier;Arnaud F Klein;Caroline Hammer;Stéphane Vassilopoulos.
Nature Medicine (2011)
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